emd 53998 has been researched along with propranolol in 4 studies
| Studies (emd 53998) | Trials (emd 53998) | Recent Studies (post-2010) (emd 53998) | Studies (propranolol) | Trials (propranolol) | Recent Studies (post-2010) (propranolol) |
|---|---|---|---|---|---|
| 112 | 0 | 9 | 33,310 | 3,406 | 3,218 |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (25.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chiba, S; Furukawa, Y; Hirose, M; Lakhe, M; Miyashita, Y; Nagashima, Y; Yonezawa, T | 1 |
| de Zeeuw, S; Duncker, DJ; Krams, R; Trines, SA; Verdouw, PD | 1 |
| Knollmann, BC; Potter, JD; Sirenko, SG | 1 |
| Copeland, O; Ferenczi, MA; Marston, SB; Messer, AE; Song, W; Vikhorev, PG; Wilkinson, R | 1 |
4 other study(ies) available for emd 53998 and propranolol
| Article | Year |
|---|---|
Different chronotropic and inotropic effects of EMD 57033 and EMD 53998, Ca2+ sensitizers, on isolated, blood-perfused dog heart preparations.
Topics: Animals; Cardiotonic Agents; Dogs; In Vitro Techniques; Myocardial Contraction; Norepinephrine; Propranolol; Quinolines; Thiadiazines | 1998 |
In vivo evidence that EMD 57033 restores myocardial responsiveness to intracoronary Ca(2+) in stunned myocardium.
Topics: Animals; Calcium; Cardiotonic Agents; Coronary Vessels; Dose-Response Relationship, Drug; Female; Heart; Hemodynamics; Male; Myocardial Stunning; Propranolol; Quinolines; Stroke Volume; Swine; Thiadiazines; Vasodilator Agents; Ventricular Function, Left | 2000 |
Differential effect of troponin T mutations on the inotropic responsiveness of mouse hearts--role of myofilament Ca2+ sensitivity increase.
Topics: Actin Cytoskeleton; Adrenergic beta-Antagonists; Animals; Calcium; Cardiomyopathy, Hypertrophic, Familial; Cardiotonic Agents; Exercise Tolerance; Heart; Isoproterenol; Mice; Mice, Transgenic; Mutation; Myocardial Contraction; Myocardium; Propranolol; Quinolines; Thiadiazines; Troponin T | 2006 |
The dilated cardiomyopathy-causing mutation ACTC E361G in cardiac muscle myofibrils specifically abolishes modulation of Ca(2+) regulation by phosphorylation of troponin I.
Topics: Actins; Animals; Calcium; Cardiomyopathy, Dilated; Carrier Proteins; Cyclic AMP-Dependent Protein Kinases; Female; Male; Mice; Mice, Transgenic; Muscle Contraction; Mutation; Myofibrils; Phosphorylation; Propranolol; Quinolines; Sarcomeres; Thiadiazines; Troponin I | 2014 |