| Condition | Indicated | Relationship Strength | Studies | Trials |
|---|
Cystic Fibrosis of Pancreas
[description not available] | 0 | 5.06 | 3 | 1 |
Disease Models, Animal
Naturally-occurring or experimentally-induced animal diseases with pathological processes analogous to human diseases. | 0 | 2.77 | 3 | 0 |
Innate Inflammatory Response
[description not available] | 0 | 4.17 | 3 | 1 |
Infections, Pseudomonas
[description not available] | 0 | 2.1 | 1 | 0 |
Cystic Fibrosis
An autosomal recessive genetic disease of the EXOCRINE GLANDS. It is caused by mutations in the gene encoding the CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR expressed in several organs including the LUNG, the PANCREAS, the BILIARY SYSTEM, and the SWEAT GLANDS. Cystic fibrosis is characterized by epithelial secretory dysfunction associated with ductal obstruction resulting in AIRWAY OBSTRUCTION; chronic RESPIRATORY INFECTIONS; PANCREATIC INSUFFICIENCY; maldigestion; salt depletion; and HEAT PROSTRATION. | 1 | 7.06 | 3 | 1 |
Inflammation
A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function. | 0 | 4.17 | 3 | 1 |
Pseudomonas Infections
Infections with bacteria of the genus PSEUDOMONAS. | 0 | 2.1 | 1 | 0 |
Bacteremia
The presence of viable bacteria circulating in the blood. Fever, chills, tachycardia, and tachypnea are common acute manifestations of bacteremia. The majority of cases are seen in already hospitalized patients, most of whom have underlying diseases or procedures which render their bloodstreams susceptible to invasion. | 0 | 2.1 | 1 | 0 |
Injury, Ischemia-Reperfusion
[description not available] | 0 | 2.08 | 1 | 0 |
Heart Disease, Ischemic
[description not available] | 0 | 2.08 | 1 | 0 |
Extravasation of Contrast Media
[description not available] | 0 | 2.08 | 1 | 0 |
Reperfusion Injury
Adverse functional, metabolic, or structural changes in tissues that result from the restoration of blood flow to the tissue (REPERFUSION) following ISCHEMIA. | 0 | 2.08 | 1 | 0 |
Myocardial Ischemia
A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION). | 0 | 2.08 | 1 | 0 |
Disease Exacerbation
[description not available] | 0 | 3.48 | 1 | 1 |
Adverse Drug Event
[description not available] | 0 | 3.48 | 1 | 1 |
Drug-Related Side Effects and Adverse Reactions
Disorders that result from the intended use of PHARMACEUTICAL PREPARATIONS. Included in this heading are a broad variety of chemically-induced adverse conditions due to toxicity, DRUG INTERACTIONS, and metabolic effects of pharmaceuticals. | 0 | 3.48 | 1 | 1 |
Atherogenesis
[description not available] | 0 | 2.46 | 2 | 0 |
Atherosclerosis
A thickening and loss of elasticity of the walls of ARTERIES that occurs with formation of ATHEROSCLEROTIC PLAQUES within the ARTERIAL INTIMA. | 0 | 2.46 | 2 | 0 |
Carotid Artery Narrowing
[description not available] | 0 | 2.05 | 1 | 0 |
Hyperplasia
An increase in the number of cells in a tissue or organ without tumor formation. It differs from HYPERTROPHY, which is an increase in bulk without an increase in the number of cells. | 0 | 2.44 | 2 | 0 |
Carotid Stenosis
Narrowing or stricture of any part of the CAROTID ARTERIES, most often due to atherosclerotic plaque formation. Ulcerations may form in atherosclerotic plaques and induce THROMBUS formation. Platelet or cholesterol emboli may arise from stenotic carotid lesions and induce a TRANSIENT ISCHEMIC ATTACK; CEREBROVASCULAR ACCIDENT; or temporary blindness (AMAUROSIS FUGAX). (From Adams et al., Principles of Neurology, 6th ed, pp 822-3) | 0 | 2.05 | 1 | 0 |
Rheumatoid Arthritis
[description not available] | 0 | 4.38 | 2 | 2 |
Arthritis, Rheumatoid
A chronic systemic disease, primarily of the joints, marked by inflammatory changes in the synovial membranes and articular structures, widespread fibrinoid degeneration of the collagen fibers in mesenchymal tissues, and by atrophy and rarefaction of bony structures. Etiology is unknown, but autoimmune mechanisms have been implicated. | 1 | 6.38 | 2 | 2 |
Carotid Arteriopathies, Traumatic
[description not available] | 0 | 2.02 | 1 | 0 |
Asthma, Bronchial
[description not available] | 0 | 2.94 | 1 | 0 |
Infections, Respiratory
[description not available] | 0 | 2.94 | 1 | 0 |
Viral Diseases
[description not available] | 0 | 2.94 | 1 | 0 |
Asthma
A form of bronchial disorder with three distinct components: airway hyper-responsiveness (RESPIRATORY HYPERSENSITIVITY), airway INFLAMMATION, and intermittent AIRWAY OBSTRUCTION. It is characterized by spasmodic contraction of airway smooth muscle, WHEEZING, and dyspnea (DYSPNEA, PAROXYSMAL). | 1 | 4.94 | 1 | 0 |
Respiratory Tract Infections
Invasion of the host RESPIRATORY SYSTEM by microorganisms, usually leading to pathological processes or diseases. | 0 | 2.94 | 1 | 0 |
Virus Diseases
A general term for diseases caused by viruses. | 0 | 2.94 | 1 | 0 |
Bronchial Hyperreactivity
Tendency of the smooth muscle of the tracheobronchial tree to contract more intensely in response to a given stimulus than it does in the response seen in normal individuals. This condition is present in virtually all symptomatic patients with asthma. The most prominent manifestation of this smooth muscle contraction is a decrease in airway caliber that can be readily measured in the pulmonary function laboratory. | 0 | 2.94 | 1 | 0 |