| Condition | Indicated | Relationship Strength | Studies | Trials |
|---|
Disease Models, Animal
Naturally-occurring or experimentally-induced animal diseases with pathological processes analogous to human diseases. | 0 | 2.96 | 4 | 0 |
Sneezing
The sudden, forceful, involuntary expulsion of air from the NOSE and MOUTH caused by irritation to the MUCOUS MEMBRANES of the upper RESPIRATORY TRACT. | 0 | 2.04 | 1 | 0 |
Bilateral Nasal Obstruction
[description not available] | 0 | 2.04 | 1 | 0 |
Rhinitis, Allergic, Nonseasonal
[description not available] | 0 | 2.04 | 1 | 0 |
Rhinitis, Allergic, Perennial
Inflammation of the mucous membrane of the nose similar to that found in hay fever except that symptoms persist throughout the year. The causes are usually air-borne allergens, particularly dusts, feathers, molds, animal fur, etc. | 0 | 2.04 | 1 | 0 |
Nasal Obstruction
Any hindrance to the passage of air into and out of the nose. The obstruction may be unilateral or bilateral, and may involve any part of the NASAL CAVITY. | 0 | 2.04 | 1 | 0 |
Asthma, Bronchial
[description not available] | 0 | 2.45 | 2 | 0 |
Innate Inflammatory Response
[description not available] | 0 | 2.46 | 2 | 0 |
Asthma
A form of bronchial disorder with three distinct components: airway hyper-responsiveness (RESPIRATORY HYPERSENSITIVITY), airway INFLAMMATION, and intermittent AIRWAY OBSTRUCTION. It is characterized by spasmodic contraction of airway smooth muscle, WHEEZING, and dyspnea (DYSPNEA, PAROXYSMAL). | 0 | 7.45 | 2 | 0 |
Inflammation
A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function. | 0 | 7.46 | 2 | 0 |
Experimental Lung Inflammation
Inflammation of any part, segment or lobe, of the lung parenchyma. | 0 | 2.05 | 1 | 0 |
Pneumonia
Infection of the lung often accompanied by inflammation. | 0 | 2.05 | 1 | 0 |
Bronchial Hyperreactivity
Tendency of the smooth muscle of the tracheobronchial tree to contract more intensely in response to a given stimulus than it does in the response seen in normal individuals. This condition is present in virtually all symptomatic patients with asthma. The most prominent manifestation of this smooth muscle contraction is a decrease in airway caliber that can be readily measured in the pulmonary function laboratory. | 0 | 2.05 | 1 | 0 |
Alveolitis, Fibrosing
[description not available] | 0 | 2.05 | 1 | 0 |
Pulmonary Fibrosis
A process in which normal lung tissues are progressively replaced by FIBROBLASTS and COLLAGEN causing an irreversible loss of the ability to transfer oxygen into the bloodstream via PULMONARY ALVEOLI. Patients show progressive DYSPNEA finally resulting in death. | 0 | 7.05 | 1 | 0 |
Airway Obstruction
Any hindrance to the passage of air into and out of the lungs. | 0 | 7.04 | 1 | 0 |
Airway Hyper-Responsiveness
[description not available] | 0 | 2.04 | 1 | 0 |