Compounds > pectenotoxin-4
Page last updated: 2024-11-11

pectenotoxin-4

Description Research Excerpts Clinical Trials Roles Classes Pathways Study Profile Bioassays Related Drugs Related Conditions Protein Interactions Research Growth

Description

pectenotoxin-4: structure in first source [Medical Subject Headings (MeSH), National Library of Medicine, extracted Dec-2023]

Cross-References

ID SourceID
PubMed CID10533675
MeSH IDM0445836

Synonyms (2)

Synonym
6k4c6v4qst ,
pectenotoxin-4
[information is derived through text-mining from research data collected from National Library of Medicine (NLM), extracted Dec-2023]

Research

Studies (8)

TimeframeStudies, This Drug (%)All Drugs %
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's5 (62.50)29.6817
2010's2 (25.00)24.3611
2020's1 (12.50)2.80
[information is prepared from research data collected from National Library of Medicine (NLM), extracted Dec-2023]

Study Types

Publication TypeThis drug (%)All Drugs (%)
Trials0 (0.00%)5.53%
Reviews1 (12.50%)6.00%
Case Studies0 (0.00%)4.05%
Observational0 (0.00%)0.25%
Other7 (87.50%)84.16%
[information is prepared from research data collected from National Library of Medicine (NLM), extracted Dec-2023]
SubstanceRelationship StrengthStudiesTrialsClassesRoles
propadiene
[no description available]		2.4520allenes
pectenotoxin 2
pectenotoxin 2: isolated from the marine sponges Poecillastra and Jaspis; structure in first source		2.0310polycyclic ether;
spiroketal		marine metabolite
pectenotoxin 1
pectenotoxin 1: RN from Toxline; RN not in Chemline 4/87; structure given in first source		2.0310polycyclic ether;
spiroketal		marine metabolite
ConditionIndicatedRelationship StrengthStudiesTrials
Autoimmune Disease
[description not available]		03.5210
Innate Inflammatory Response
[description not available]		03.5210
Autoimmune Diseases
Disorders that are characterized by the production of antibodies that react with host tissues or immune effector cells that are autoreactive to endogenous peptides.		03.5210
Inflammation
A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function.		03.5210