catechin and Keloid

catechin has been researched along with Keloid in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (66.67)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bagabir, RA; Bayat, A; Paus, R; Syed, F1
Duong, HS; French, SW; Kelly, AP; Le, AD; Messadi, DV; Tang, X; Wang, L; Zhang, Q1
Jun, EK; Kim, B; Kim, H; Moon, JH; Noh, JY; Oh, C; Oh, S; Park, G; Song, HJ; Yoon, BS; You, S1

Other Studies

3 other study(ies) available for catechin and Keloid

ArticleYear
Ex vivo evaluation of antifibrotic compounds in skin scarring: EGCG and silencing of PAI-1 independently inhibit growth and induce keloid shrinkage.
    Laboratory investigation; a journal of technical methods and pathology, 2013, Volume: 93, Issue:8

    Topics: Adolescent; Adult; Apoptosis; Catechin; Cell Proliferation; Collagen; Dexamethasone; Gene Knockdown Techniques; Gene Silencing; Humans; Keloid; Organ Culture Techniques; Plasminogen Activator Inhibitor 1; RNA, Small Interfering; Skin; Young Adult

2013
Green tea extract and (-)-epigallocatechin-3-gallate inhibit mast cell-stimulated type I collagen expression in keloid fibroblasts via blocking PI-3K/AkT signaling pathways.
    The Journal of investigative dermatology, 2006, Volume: 126, Issue:12

    Topics: Camellia sinensis; Catechin; Cells, Cultured; Coculture Techniques; Collagen Type I; Fibroblasts; Humans; Keloid; Mast Cells; Phosphatidylinositol 3-Kinases; Plant Extracts; Proto-Oncogene Proteins c-akt; Signal Transduction

2006
Green tea polyphenol epigallocatechin-3-gallate suppresses collagen production and proliferation in keloid fibroblasts via inhibition of the STAT3-signaling pathway.
    The Journal of investigative dermatology, 2008, Volume: 128, Issue:10

    Topics: Animals; Catechin; Cell Cycle; Cell Movement; Cell Proliferation; Cells, Cultured; Collagen; Fibroblasts; Flavonoids; Humans; Keloid; Mice; Mice, Nude; Mitogen-Activated Protein Kinase Kinases; Phenols; Phosphatidylinositol 3-Kinases; Polyphenols; Protein Kinase Inhibitors; RNA, Small Interfering; Signal Transduction; STAT3 Transcription Factor; Tea; Triterpenes; Wound Healing

2008