substance p has been researched along with kn 93 in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (75.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, JG; Guan, BC; Li, ZW; Liu, CJ; Wu, ZZ; Yang, Q | 1 |
| Choi, SS; Ham, YO; Kwon, MS; Lee, HK; Lee, JY; Seo, YJ; Shim, EJ; Suh, HW | 1 |
| Hammond, DL; Zhang, L | 1 |
| Daugherty, SL; de Groat, WC; Zhang, X | 1 |
4 other study(ies) available for substance p and kn 93
| Article | Year |
|---|---|
Sustained potentiation by substance P of NMDA-activated current in rat primary sensory neurons.
Topics: Animals; Benzylamines; Calcium Signaling; Cell Membrane; Cells, Cultured; Chelating Agents; Chronic Disease; Dose-Response Relationship, Drug; Drug Synergism; Egtazic Acid; Enzyme Inhibitors; Ganglia, Spinal; Membrane Potentials; Models, Biological; N-Methylaspartate; Neurokinin-1 Receptor Antagonists; Neurons, Afferent; Pain; Patch-Clamp Techniques; Rats; Rats, Sprague-Dawley; Reaction Time; Receptors, N-Methyl-D-Aspartate; Receptors, Neurokinin-1; Substance P; Sulfonamides | 2004 |
Increase of phosphorylation of calcium/calmodulin-dependent protein kinase-II in several brain regions by substance P administered intrathecally in mice.
Topics: Analysis of Variance; Animals; Behavior, Animal; Benzylamines; Blotting, Western; Brain; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Dose-Response Relationship, Drug; Drug Administration Routes; Enzyme Inhibitors; Immunohistochemistry; Male; Mice; Mice, Inbred ICR; Pain Measurement; Phosphorylation; Spinal Cord; Substance P; Sulfonamides; Time Factors | 2005 |
Substance P enhances excitatory synaptic transmission on spinally projecting neurons in the rostral ventromedial medulla after inflammatory injury.
Topics: Animals; Benzylamines; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Enzyme Inhibitors; Excitatory Postsynaptic Potentials; Glutamic Acid; In Vitro Techniques; Indoles; Inflammation; Male; Maleimides; Medulla Oblongata; Neural Pathways; Neurons; Neurotransmitter Agents; Pain; Protein Kinase C; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Receptors, Neurokinin-1; Serotonin; Substance P; Sulfonamides; Synaptic Transmission | 2009 |
Activation of CaMKII and ERK1/2 contributes to the time-dependent potentiation of Ca2+ response elicited by repeated application of capsaicin in rat DRG neurons.
Topics: Animals; Benzylamines; Butadienes; Calcium Signaling; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Capsaicin; Cells, Cultured; Cyclic AMP-Dependent Protein Kinases; Enzyme Activation; Ganglia, Spinal; Male; Membrane Potentials; Microscopy, Fluorescence; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Neuronal Plasticity; Neurons; Nitriles; Patch-Clamp Techniques; Protein Kinase C; Protein Kinase Inhibitors; Pyrethrins; Rats; Rats, Sprague-Dawley; Recovery of Function; Sensory System Agents; Substance P; Sulfonamides; Tachyphylaxis; Time Factors; TRPV Cation Channels | 2011 |