simvastatin and u 0126
simvastatin has been researched along with u 0126 in 7 studies
Research
Studies (7)
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (14.29) | 29.6817 |
2010's | 6 (85.71) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors
Authors | Studies |
---|---|
Akamatsu, S; Dohi, S; Hirade, K; Kozawa, O; Matsuno, H; Takenaka, M; Tanabe, K | 1 |
Chen, H; Jin, WY; Ren, JY | 1 |
Chung, HJ; Jeong, BC; Kim, IS; Kim, OS; Kim, YJ; Koh, JT; Lee, KN; Lee, SE | 1 |
de Vries, EG; Korthuis, PM; Kuipers, F; van der Weide, K; Vellenga, E | 1 |
Huber, M; Marschall, JS; Schuh, W; Wilhelm, T | 1 |
Karanxha, L; Min, KS; Nör, JE; Park, SJ; Son, WJ | 1 |
Ahn, JS; Im, YH; Jung, HH; Kim, JY; Lee, SH; Park, YH | 1 |
Other Studies
7 other study(ies) available for simvastatin and u 0126
Article | Year |
---|---|
Simvastatin stimulates VEGF release via p44/p42 MAP kinase in vascular smooth muscle cells.
Topics: Animals; Butadienes; Cell Line; Dose-Response Relationship, Drug; Endothelial Growth Factors; Enzyme Inhibitors; Flavonoids; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Intercellular Signaling Peptides and Proteins; Lymphokines; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Nitriles; Phosphorylation; Rats; Simvastatin; Vascular Endothelial Growth Factor A; Vascular Endothelial Growth Factors | 2003 |
[Simvastatin suppress lipopolysaccharides induced upregulation of lipoprotein associated phospholipase A(2) expression in macrophages via inactivation of p38MAPK pathway].
Topics: 1-Alkyl-2-acetylglycerophosphocholine Esterase; Anthracenes; Butadienes; Cells, Cultured; Humans; Imidazoles; Lipopolysaccharides; Macrophages; Monocytes; Nitriles; p38 Mitogen-Activated Protein Kinases; Phospholipases A2; Phosphorylation; Pyridines; RNA, Messenger; Simvastatin | 2010 |
Lactone form 3-hydroxy-3-methylglutaryl-coenzyme A reductase inhibitors (statins) stimulate the osteoblastic differentiation of mouse periodontal ligament cells via the ERK pathway.
Topics: Alkaline Phosphatase; Animals; Anthraquinones; Blotting, Western; Bone Morphogenetic Protein 2; Butadienes; Calcification, Physiologic; Cell Culture Techniques; Cell Differentiation; Cell Line; Cell Survival; Coloring Agents; Enzyme Inhibitors; Extracellular Signal-Regulated MAP Kinases; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Integrin-Binding Sialoprotein; Lovastatin; Mice; Mitogen-Activated Protein Kinase 1; Nitriles; Osteoblasts; Osteocalcin; Periodontal Ligament; Promoter Regions, Genetic; Simvastatin; Transcriptional Activation | 2011 |
Heterogeneity in simvastatin-induced cytotoxicity in AML is caused by differences in Ras-isoprenylation.
Topics: Butadienes; Extracellular Signal-Regulated MAP Kinases; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Leukemia, Myeloid, Acute; Nitriles; Prenylation; ras Proteins; Simvastatin | 2012 |
MEK/Erk-based negative feedback mechanism involved in control of Steel Factor-triggered production of Krüppel-like factor 2 in mast cells.
Topics: Animals; Benzamides; Butadienes; Cells, Cultured; Diphenylamine; Gene Expression Regulation; Hypolipidemic Agents; Insulin-Like Growth Factor I; Interleukin-6; Kruppel-Like Transcription Factors; Lipopolysaccharides; MAP Kinase Kinase Kinases; MAP Kinase Signaling System; Mast Cells; Mice; Nitriles; Phosphatidylinositol 3-Kinase; Protein Kinase Inhibitors; Signal Transduction; Simvastatin; Stem Cell Factor; Thapsigargin; Tumor Necrosis Factor-alpha | 2012 |
Combined effects of simvastatin and enamel matrix derivative on odontoblastic differentiation of human dental pulp cells.
Topics: Alkaline Phosphatase; Anthraquinones; Butadienes; Calcification, Physiologic; Cell Culture Techniques; Cell Differentiation; Cell Survival; Coloring Agents; Core Binding Factor Alpha 1 Subunit; Dental Enamel Proteins; Dental Pulp; Enzyme Inhibitors; Extracellular Matrix Proteins; Extracellular Signal-Regulated MAP Kinases; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; MAP Kinase Signaling System; Nitriles; Odontoblasts; Osteoblasts; Osteonectin; Phosphoproteins; Phosphorylation; Sialoglycoproteins; Simvastatin; Sp7 Transcription Factor; Transcription Factors | 2013 |
Statins affect ETS1-overexpressing triple-negative breast cancer cells by restoring DUSP4 deficiency.
Topics: Antineoplastic Agents; Apoptosis; Butadienes; Cell Line, Tumor; Cell Survival; Chromones; Dual-Specificity Phosphatases; Female; Gene Expression; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase Phosphatases; Morpholines; Nitriles; Nuclear Proteins; Proto-Oncogene Mas; Proto-Oncogene Protein c-ets-1; RNA, Messenger; Simvastatin; Transcription Factors; Transfection; Triple Negative Breast Neoplasms; Tumor Stem Cell Assay | 2016 |