imipramine and pd 98059

imipramine has been researched along with pd 98059 in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bianchi, P; Colucci, WS; Murphy, MP; Parini, A; Pimentel, DR1
Kang, HS; Kim, JS; Kim, SJ; Lee, MY1

Other Studies

2 other study(ies) available for imipramine and pd 98059

ArticleYear
A new hypertrophic mechanism of serotonin in cardiac myocytes: receptor-independent ROS generation.
    FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2005, Volume: 19, Issue:6

    Topics: Adrenergic Uptake Inhibitors; Animals; Catalase; Cells, Cultured; Enzyme Activation; Flavonoids; Gene Expression; Glutathione; Hydrogen Peroxide; Hypertrophy; Imipramine; Male; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinase Kinases; Monoamine Oxidase; Monoamine Oxidase Inhibitors; Myocytes, Cardiac; Oxidation-Reduction; Oxidative Stress; Pargyline; Phosphorylation; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Serotonin; Transfection

2005
Imipramine-induced cardiac depression is responsible for the increase in intracellular magnesium and the activation of ERK 1/2 in rats.
    Journal of cardiovascular pharmacology and therapeutics, 2010, Volume: 15, Issue:3

    Topics: Animals; Antidepressive Agents, Tricyclic; Flavonoids; Heart; Heart Rate; Imipramine; In Vitro Techniques; Intracellular Space; Magnesium; Male; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Myocardium; Rats; Rats, Sprague-Dawley; Ventricular Function, Left

2010