imipramine has been researched along with pd 98059 in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (50.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Bianchi, P; Colucci, WS; Murphy, MP; Parini, A; Pimentel, DR | 1 |
Kang, HS; Kim, JS; Kim, SJ; Lee, MY | 1 |
2 other study(ies) available for imipramine and pd 98059
Article | Year |
---|---|
A new hypertrophic mechanism of serotonin in cardiac myocytes: receptor-independent ROS generation.
Topics: Adrenergic Uptake Inhibitors; Animals; Catalase; Cells, Cultured; Enzyme Activation; Flavonoids; Gene Expression; Glutathione; Hydrogen Peroxide; Hypertrophy; Imipramine; Male; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinase Kinases; Monoamine Oxidase; Monoamine Oxidase Inhibitors; Myocytes, Cardiac; Oxidation-Reduction; Oxidative Stress; Pargyline; Phosphorylation; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Serotonin; Transfection | 2005 |
Imipramine-induced cardiac depression is responsible for the increase in intracellular magnesium and the activation of ERK 1/2 in rats.
Topics: Animals; Antidepressive Agents, Tricyclic; Flavonoids; Heart; Heart Rate; Imipramine; In Vitro Techniques; Intracellular Space; Magnesium; Male; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Myocardium; Rats; Rats, Sprague-Dawley; Ventricular Function, Left | 2010 |