fentanyl has been researched along with h 89 in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (50.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Lambert, DG; Smart, D | 2 |
| Furuya, H; Hatanaka, H; Hirano, M; Nakayama, H; Ueno, S; Yoshikawa, M | 1 |
| Liang, X; Su, R; Yong, Z | 1 |
4 other study(ies) available for fentanyl and h 89
| Article | Year |
|---|---|
Mu-opioid activation of phospholipase C in SH-SY5Y cells rapidly desensitizes: mechanistic studies.
Topics: 4-Aminopyridine; Calcium; Cell Line; Cyclic AMP-Dependent Protein Kinases; Enzyme Activation; Fentanyl; Humans; Indoles; Inositol 1,4,5-Trisphosphate; Isoquinolines; Protein Kinase C; Receptors, Opioid, mu; Sulfonamides; Type C Phospholipases | 1995 |
Desensitization of the mu-opioid activation of phospholipase C in SH-SY5Y cells: the role of protein kinases C and A and Ca(2+)-activated K+ currents.
Topics: 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Analgesics, Opioid; Calcium; Calcium Channel Agonists; Calcium Channel Blockers; Cyclic AMP-Dependent Protein Kinases; Enzyme Activation; Enzyme Inhibitors; Fentanyl; Humans; Indoles; Inositol 1,4,5-Trisphosphate; Isoquinolines; Narcotics; Neuroblastoma; Nifedipine; Potassium Channels; Protein Kinase C; Receptors, Opioid, mu; Sensitivity and Specificity; Sulfonamides; Tumor Cells, Cultured; Type C Phospholipases | 1995 |
Chronic fentanyl treatments induce the up-regulation of mu opioid receptor mRNA in rat pheochromocytoma cells.
Topics: Analgesics, Opioid; Animals; Cells, Cultured; Cyclic AMP; Drug Administration Schedule; Enzyme Inhibitors; Fentanyl; Intracellular Fluid; Isoquinolines; PC12 Cells; Rats; Receptors, Opioid, mu; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Signal Transduction; Sulfonamides; Thionucleotides; Up-Regulation | 2000 |
Inhibition of protein kinase A and GIRK channel reverses fentanyl-induced respiratory depression.
Topics: Animals; Bee Venoms; Cyclic AMP-Dependent Protein Kinases; Fentanyl; G Protein-Coupled Inwardly-Rectifying Potassium Channels; Isoquinolines; Male; Potassium Channel Blockers; Protein Kinase Inhibitors; Rats, Sprague-Dawley; Respiratory Insufficiency; Sulfonamides | 2018 |