Page last updated: 2024-11-05

zinc chloride and Stroke

zinc chloride has been researched along with Stroke in 3 studies

zinc chloride: RN given refers to parent cpd
zinc dichloride : A compound of zinc and chloride ions in the ratio 1:2. It exists in four crystalline forms, in each of which the Zn(2+) ions are trigonal planar coordinated to four chloride ions.

Stroke: A group of pathological conditions characterized by sudden, non-convulsive loss of neurological function due to BRAIN ISCHEMIA or INTRACRANIAL HEMORRHAGES. Stroke is classified by the type of tissue NECROSIS, such as the anatomic location, vasculature involved, etiology, age of the affected individual, and hemorrhagic vs. non-hemorrhagic nature. (From Adams et al., Principles of Neurology, 6th ed, pp777-810)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (66.67)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Pan, R1
Timmins, GS1
Liu, W1
Liu, KJ1
Limbrick, DD2
Sombati, S2
DeLorenzo, RJ2
Deshpande, LS1

Other Studies

3 other studies available for zinc chloride and Stroke

ArticleYear
Autophagy Mediates Astrocyte Death During Zinc-Potentiated Ischemia--Reperfusion Injury.
    Biological trace element research, 2015, Volume: 166, Issue:1

    Topics: Animals; Astrocytes; Autophagy; Blotting, Western; Cell Culture Techniques; Cell Hypoxia; Cell Line;

2015
Calcium influx constitutes the ionic basis for the maintenance of glutamate-induced extended neuronal depolarization associated with hippocampal neuronal death.
    Cell calcium, 2003, Volume: 33, Issue:2

    Topics: Animals; Animals, Newborn; Brain Injuries; Calcium; Calcium Channel Blockers; Calcium Signaling; Cel

2003
Activation of a novel injury-induced calcium-permeable channel that plays a key role in causing extended neuronal depolarization and initiating neuronal death in excitotoxic neuronal injury.
    The Journal of pharmacology and experimental therapeutics, 2007, Volume: 322, Issue:2

    Topics: 6-Cyano-7-nitroquinoxaline-2,3-dione; Animals; Animals, Newborn; Apoptosis; Calcium; Calcium Channel

2007