Page last updated: 2024-09-03

y 27632 and Granulocytic Leukemia, Chronic

y 27632 has been researched along with Granulocytic Leukemia, Chronic in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Basbous, S; Bourmeyster, N; Brizard, F; Gombert, JM; Guilhot, F; Herbelin, A; Levescot, A; Piccirilli, N; Roy, L1
Adams, J; Burthem, J; Lucas, GS; Mottram, R; Rees-Unwin, K; Spooncer, E; Whetton, AD1

Other Studies

2 other study(ies) available for y 27632 and Granulocytic Leukemia, Chronic

ArticleYear
The Rho-ROCK pathway as a new pathological mechanism of innate immune subversion in chronic myeloid leukaemia.
    The Journal of pathology, 2016, Volume: 240, Issue:3

    Topics: Amides; Antigens, CD1d; Cell Differentiation; Dendritic Cells; Enzyme Inhibitors; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; Immunity, Innate; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Myeloid Cells; Natural Killer T-Cells; Protein Kinase Inhibitors; Pyridines; rho-Associated Kinases

2016
The rho-kinase inhibitors Y-27632 and fasudil act synergistically with imatinib to inhibit the expansion of ex vivo CD34(+) CML progenitor cells.
    Leukemia, 2007, Volume: 21, Issue:8

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; Amides; Antigens, CD34; Benzamides; Cell Proliferation; Drug Synergism; Enzyme Inhibitors; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; Intracellular Signaling Peptides and Proteins; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Piperazines; Protein Kinase Inhibitors; Protein Serine-Threonine Kinases; Protein-Tyrosine Kinases; Pyridines; Pyrimidines; rho-Associated Kinases; Stem Cells; Tumor Cells, Cultured

2007