Page last updated: 2024-11-04

vorinostat and Cognition Disorders

vorinostat has been researched along with Cognition Disorders in 2 studies

Vorinostat: A hydroxamic acid and anilide derivative that acts as a HISTONE DEACETYLASE inhibitor. It is used in the treatment of CUTANEOUS T-CELL LYMPHOMA and SEZARY SYNDROME.
vorinostat : A dicarboxylic acid diamide comprising suberic (octanedioic) acid coupled to aniline and hydroxylamine. A histone deacetylase inhibitor, it is marketed under the name Zolinza for the treatment of cutaneous T cell lymphoma (CTCL).

Cognition Disorders: Disorders characterized by disturbances in mental processes related to learning, thinking, reasoning, and judgment.

Research Excerpts

ExcerptRelevanceReference
"Insulin resistance has been reported as a strong risk factor for Alzheimer's disease."1.43Epigenetic modifications by inhibiting histone deacetylases reverse memory impairment in insulin resistance induced cognitive deficit in mice. ( Sharma, S; Taliyan, R, 2016)
"Sepsis-associated encephalopathy (SAE), which associates with neuronal apoptosis and cognitive disorders, is a common complication of systemic sepsis."1.40Epigenetic modulation of neuronal apoptosis and cognitive functions in sepsis-associated encephalopathy. ( Cai, S; Fang, J; Lian, Y; Wen, P; Xie, K, 2014)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fang, J1
Lian, Y1
Xie, K1
Cai, S1
Wen, P1
Sharma, S1
Taliyan, R1

Other Studies

2 other studies available for vorinostat and Cognition Disorders

ArticleYear
Epigenetic modulation of neuronal apoptosis and cognitive functions in sepsis-associated encephalopathy.
    Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology, 2014, Volume: 35, Issue:2

    Topics: Animals; Apoptosis; bcl-2-Associated X Protein; bcl-X Protein; Brain Diseases; Cells, Cultured; Cogn

2014
Epigenetic modifications by inhibiting histone deacetylases reverse memory impairment in insulin resistance induced cognitive deficit in mice.
    Neuropharmacology, 2016, Volume: 105

    Topics: Animals; Avoidance Learning; Behavior, Animal; Brain-Derived Neurotrophic Factor; Cognition Disorder

2016