Page last updated: 2024-10-30

vitamin k 3 and Osteoarthritis

vitamin k 3 has been researched along with Osteoarthritis in 2 studies

Vitamin K 3: A synthetic naphthoquinone without the isoprenoid side chain and biological activity, but can be converted to active vitamin K2, menaquinone, after alkylation in vivo.

Osteoarthritis: A progressive, degenerative joint disease, the most common form of arthritis, especially in older persons. The disease is thought to result not from the aging process but from biochemical changes and biomechanical stresses affecting articular cartilage. In the foreign literature it is often called osteoarthrosis deformans.

Research Excerpts

ExcerptRelevanceReference
"Age-related osteoarthritis is a common form of arthritis that has been associated with mitochondrial dysfunction and oxidative stress."1.43Oxidative Stress Promotes Peroxiredoxin Hyperoxidation and Attenuates Pro-survival Signaling in Aging Chondrocytes. ( Carlson, CS; Chubinskaya, S; Collins, JA; Furdui, CM; Loeser, RF; Nelson, KJ; Poole, LB; Rowe, MA; Wood, ST, 2016)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (50.00)24.3611
2020's1 (50.00)2.80

Authors

AuthorsStudies
Taylor, EL1
Collins, JA2
Gopalakrishnan, P1
Chubinskaya, S2
Loeser, RF2
Wood, ST1
Nelson, KJ1
Rowe, MA1
Carlson, CS1
Poole, LB1
Furdui, CM1

Other Studies

2 other studies available for vitamin k 3 and Osteoarthritis

ArticleYear
Age and oxidative stress regulate Nrf2 homeostasis in human articular chondrocytes.
    Osteoarthritis and cartilage, 2023, Volume: 31, Issue:9

    Topics: Aged; Antioxidants; Cartilage, Articular; Cells, Cultured; Chondrocytes; Homeostasis; Humans; Insuli

2023
Oxidative Stress Promotes Peroxiredoxin Hyperoxidation and Attenuates Pro-survival Signaling in Aging Chondrocytes.
    The Journal of biological chemistry, 2016, Mar-25, Volume: 291, Issue:13

    Topics: Adult; Aging; Animals; Cartilage; Catalase; Cellular Senescence; Chondrocytes; Homeodomain Proteins;

2016