Page last updated: 2024-10-30

vitamin k 3 and Fuchs' Endothelial Dystrophy

vitamin k 3 has been researched along with Fuchs' Endothelial Dystrophy in 3 studies

Vitamin K 3: A synthetic naphthoquinone without the isoprenoid side chain and biological activity, but can be converted to active vitamin K2, menaquinone, after alkylation in vivo.

Fuchs' Endothelial Dystrophy: Disorder caused by loss of endothelium of the central cornea. It is characterized by hyaline endothelial outgrowths on Descemet's membrane, epithelial blisters, reduced vision, and pain.

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Katikireddy, KR1
White, TL1
Miyajima, T1
Vasanth, S2
Raoof, D1
Chen, Y3
Price, MO2
Price, FW2
Jurkunas, UV3
Miyai, T1
Melangath, G1
Deshpande, N1
Kumar, V1
Benischke, AS2
Halilovic, A1
Schmedt, T1
Hamill, C1
Santos, JH1

Other Studies

3 other studies available for vitamin k 3 and Fuchs' Endothelial Dystrophy

ArticleYear
NQO1 downregulation potentiates menadione-induced endothelial-mesenchymal transition during rosette formation in Fuchs endothelial corneal dystrophy.
    Free radical biology & medicine, 2018, 02-20, Volume: 116

    Topics: Aged; Aged, 80 and over; Cell Differentiation; Cell Line; Cornea; DNA Damage; Endothelial Cells; Fib

2018
Activation of PINK1-Parkin-Mediated Mitophagy Degrades Mitochondrial Quality Control Proteins in Fuchs Endothelial Corneal Dystrophy.
    The American journal of pathology, 2019, Volume: 189, Issue:10

    Topics: Antifibrinolytic Agents; Endothelium, Corneal; Fuchs' Endothelial Dystrophy; Humans; Mitochondria; M

2019
Menadione-Induced DNA Damage Leads to Mitochondrial Dysfunction and Fragmentation During Rosette Formation in Fuchs Endothelial Corneal Dystrophy.
    Antioxidants & redox signaling, 2016, 06-20, Volume: 24, Issue:18

    Topics: Apoptosis; Caspase 3; Caspase 9; Cells, Cultured; Cytochromes c; DNA Damage; DNA, Mitochondrial; Fuc

2016