vitamin-b-12 has been researched along with Cerebellar-Ataxia* in 4 studies
4 other study(ies) available for vitamin-b-12 and Cerebellar-Ataxia
| Article | Year |
|---|---|
Late-onset cobalamin C disease presenting with acute cerebellar ataxia.
Topics: Cerebellar Ataxia; Homocystinuria; Humans; Vitamin B 12; Vitamin B 12 Deficiency | 2021 |
Cerebellar ataxia and leukoencephalopathy associated with cobalamin deficiency.
We report a patient who presented progressive cerebellar ataxia associated with vitamin B(12) deficiency. Brain magnetic resonance imaging (MRI) demonstrated a diffuse leukoencephalopathy. Six months after the initiation of methylcobalamin therapy, there were clinical improvement and reduction in the MRI abnormalities. Topics: Brain Stem; Cerebellar Ataxia; Cerebellum; Cerebral Cortex; Dementia, Vascular; Female; Ferric Compounds; Humans; Magnetic Resonance Imaging; Middle Aged; Nerve Fibers, Myelinated; Recovery of Function; Treatment Outcome; Vitamin B 12; Vitamin B 12 Deficiency | 2003 |
Tissue S-adenosylmethionine levels in fruit bats (Rousettus aegyptiacus) with nitrous oxide-induced neuropathy.
The effect of cobalamin inactivation by the anaesthetic gas nitrous oxide on the concentration of S-adenosylmethionine (Ado Met) in brain and liver of fruit bats (Rousettus aegyptiacus) was examined. Test animals exposed to N2O-oxygen (50:50, v/v) developed ataxia and paralysis leading to death after an average of 9.8 weeks (n6). Animals receiving pteroylmonoglutamic acid supplements in the diet became ataxic earlier (mean 8.8 weeks) while those receiving methionine supplements survived for significantly longer periods (12.5 weeks, P less than 0.01). Plasma cobalamin levels indicated severe depletion of cobalamin stores in N2O-exposed animals. The mean concentration of Ado Met in the brain of N2O-treated bats was nearly 50% higher than that of untreated controls. Ado Met levels in treated bats receiving pteroylmonoglutamic acid or methionine supplements were respectively 18 and 25% higher than in controls. In contrast, the concentration of Ado Met in the liver of all the N2O-treated groups was slightly lower than in controls. These results suggest that the N2O-induced neuropathy in the fruit bat is not related to a depletion of Ado Met in the nervous system. Topics: Animals; Brain; Cerebellar Ataxia; Chiroptera; Diet; Folic Acid; Liver; Methionine; Nitrous Oxide; Paralysis; S-Adenosylmethionine; Vitamin B 12 | 1983 |
[Acute cerebellar ataxia in childhood].
Topics: Acute Disease; Cerebellar Ataxia; Child; Child, Preschool; Female; Gait; Humans; Male; Posture; Prednisone; Speech Disorders; Vitamin B 12 | 1967 |