vesnarinone has been researched along with Glioma in 1 studies
Glioma: Benign and malignant central nervous system neoplasms derived from glial cells (i.e., astrocytes, oligodendrocytes, and ependymocytes). Astrocytes may give rise to astrocytomas (ASTROCYTOMA) or glioblastoma multiforme (see GLIOBLASTOMA). Oligodendrocytes give rise to oligodendrogliomas (OLIGODENDROGLIOMA) and ependymocytes may undergo transformation to become EPENDYMOMA; CHOROID PLEXUS NEOPLASMS; or colloid cysts of the third ventricle. (From Escourolle et al., Manual of Basic Neuropathology, 2nd ed, p21)
| Excerpt | Relevance | Reference |
|---|---|---|
| "The growth and morphological change of human glioma transplanted subcutaneously and intracerebrally to nude mice with orally administered vesnarinone, 3,4-dihydro-6-[4-(3,4-dimethoxy-benzoyl)-1-piperazinyl]-2(1H)-quinolinon e, were examined." | 7.70 | Apoptosis-inducing activity and growth suppression by vesnarinone on human glioma transplanted in nude mice. ( Adachi, M; Hori, T; Kawai, K; Nakai, S; Tanaka, S, 1999) |
| "The growth and morphological change of human glioma transplanted subcutaneously and intracerebrally to nude mice with orally administered vesnarinone, 3,4-dihydro-6-[4-(3,4-dimethoxy-benzoyl)-1-piperazinyl]-2(1H)-quinolinon e, were examined." | 3.70 | Apoptosis-inducing activity and growth suppression by vesnarinone on human glioma transplanted in nude mice. ( Adachi, M; Hori, T; Kawai, K; Nakai, S; Tanaka, S, 1999) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (100.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Tanaka, S | 1 |
| Kawai, K | 1 |
| Nakai, S | 1 |
| Adachi, M | 1 |
| Hori, T | 1 |
1 other study available for vesnarinone and Glioma
| Article | Year |
|---|---|
Apoptosis-inducing activity and growth suppression by vesnarinone on human glioma transplanted in nude mice.
Topics: Animals; Antineoplastic Agents; Apoptosis; Brain Neoplasms; Dose-Response Relationship, Drug; Drug A | 1999 |