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vesnarinone and Brain Neoplasms

vesnarinone has been researched along with Brain Neoplasms in 1 studies

Brain Neoplasms: Neoplasms of the intracranial components of the central nervous system, including the cerebral hemispheres, basal ganglia, hypothalamus, thalamus, brain stem, and cerebellum. Brain neoplasms are subdivided into primary (originating from brain tissue) and secondary (i.e., metastatic) forms. Primary neoplasms are subdivided into benign and malignant forms. In general, brain tumors may also be classified by age of onset, histologic type, or presenting location in the brain.

Research Excerpts

ExcerptRelevanceReference
"The growth and morphological change of human glioma transplanted subcutaneously and intracerebrally to nude mice with orally administered vesnarinone, 3,4-dihydro-6-[4-(3,4-dimethoxy-benzoyl)-1-piperazinyl]-2(1H)-quinolinon e, were examined."7.70Apoptosis-inducing activity and growth suppression by vesnarinone on human glioma transplanted in nude mice. ( Adachi, M; Hori, T; Kawai, K; Nakai, S; Tanaka, S, 1999)
"The growth and morphological change of human glioma transplanted subcutaneously and intracerebrally to nude mice with orally administered vesnarinone, 3,4-dihydro-6-[4-(3,4-dimethoxy-benzoyl)-1-piperazinyl]-2(1H)-quinolinon e, were examined."3.70Apoptosis-inducing activity and growth suppression by vesnarinone on human glioma transplanted in nude mice. ( Adachi, M; Hori, T; Kawai, K; Nakai, S; Tanaka, S, 1999)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (100.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Tanaka, S1
Kawai, K1
Nakai, S1
Adachi, M1
Hori, T1

Other Studies

1 other study available for vesnarinone and Brain Neoplasms

ArticleYear
Apoptosis-inducing activity and growth suppression by vesnarinone on human glioma transplanted in nude mice.
    Neurological research, 1999, Volume: 21, Issue:4

    Topics: Animals; Antineoplastic Agents; Apoptosis; Brain Neoplasms; Dose-Response Relationship, Drug; Drug A

1999