verapamil has been researched along with Lung Injury, Acute in 3 studies
Verapamil: A calcium channel blocker that is a class IV anti-arrhythmia agent.
verapamil : A racemate comprising equimolar amounts of dexverapamil and (S)-verapamil. An L-type calcium channel blocker of the phenylalkylamine class, it is used (particularly as the hydrochloride salt) in the treatment of hypertension, angina pectoris and cardiac arrhythmia, and as a preventive medication for migraine.
2-(3,4-dimethoxyphenyl)-5-{[2-(3,4-dimethoxyphenyl)ethyl](methyl)amino}-2-(propan-2-yl)pentanenitrile : A tertiary amino compound that is 3,4-dimethoxyphenylethylamine in which the hydrogens attached to the nitrogen are replaced by a methyl group and a 4-cyano-4-(3,4-dimethoxyphenyl)-5-methylhexyl group.
| Excerpt | Relevance | Reference |
|---|---|---|
| "To observe the effect of verapamil-procaine compound (V-P) on prevention and treatment of acute respiratory distress syndrome (ARDS) subsequent to high risk operation." | 9.11 | [Clinical study of the effect of verapamil-procaine compound in prevention and treatment of acute respiratory distress syndrome after high risk operation]. ( Hou, BJ; Li, JZ; Peng, RQ; Shi, JY; Wang, WG; Yang, XG; Zhang, AM; Zhang, HL, 2005) |
| "The objective of this study was to investigate the exact therapeutic effects of Verapamil on lipopolysaccharide (LPS)-induced acute lung injury (ALI) and the molecular mechanism involved, through using LPS-induced animal models as well as LPS-stimulated mouse primary peritoneal macrophages models." | 7.91 | The therapeutic effect of verapamil in lipopolysaccharide-induced acute lung injury. ( Han, L; Li, S; Liu, Y; Song, Z; Yuan, L; Zhang, C, 2019) |
| "Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS), severe form of ALI, are characterized by overwhelming of lung inflammation, and no treatment is currently available to treat ALI/ARDS." | 5.72 | Verapamil attenuates oxidative stress and inflammatory responses in cigarette smoke (CS)-induced murine models of acute lung injury and CSE-stimulated RAW 264.7 macrophages via inhibiting the NF-κB pathway. ( Aldahish, A; Alqahtani, AM; Alqahtani, T; Fatima, M; Hussain, L; Hussain, M; Jamil, Q; Khan, KU; Mukhtar, I; Saadullah, M; Shaukat, S; Syed, SK; Wu, X; Zeng, LH, 2022) |
| "To observe the effect of verapamil-procaine compound (V-P) on prevention and treatment of acute respiratory distress syndrome (ARDS) subsequent to high risk operation." | 5.11 | [Clinical study of the effect of verapamil-procaine compound in prevention and treatment of acute respiratory distress syndrome after high risk operation]. ( Hou, BJ; Li, JZ; Peng, RQ; Shi, JY; Wang, WG; Yang, XG; Zhang, AM; Zhang, HL, 2005) |
| "The objective of this study was to investigate the exact therapeutic effects of Verapamil on lipopolysaccharide (LPS)-induced acute lung injury (ALI) and the molecular mechanism involved, through using LPS-induced animal models as well as LPS-stimulated mouse primary peritoneal macrophages models." | 3.91 | The therapeutic effect of verapamil in lipopolysaccharide-induced acute lung injury. ( Han, L; Li, S; Liu, Y; Song, Z; Yuan, L; Zhang, C, 2019) |
| "Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS), severe form of ALI, are characterized by overwhelming of lung inflammation, and no treatment is currently available to treat ALI/ARDS." | 1.72 | Verapamil attenuates oxidative stress and inflammatory responses in cigarette smoke (CS)-induced murine models of acute lung injury and CSE-stimulated RAW 264.7 macrophages via inhibiting the NF-κB pathway. ( Aldahish, A; Alqahtani, AM; Alqahtani, T; Fatima, M; Hussain, L; Hussain, M; Jamil, Q; Khan, KU; Mukhtar, I; Saadullah, M; Shaukat, S; Syed, SK; Wu, X; Zeng, LH, 2022) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Wu, X | 1 |
| Hussain, M | 1 |
| Syed, SK | 1 |
| Saadullah, M | 1 |
| Alqahtani, AM | 1 |
| Alqahtani, T | 1 |
| Aldahish, A | 1 |
| Fatima, M | 1 |
| Shaukat, S | 1 |
| Hussain, L | 1 |
| Jamil, Q | 1 |
| Mukhtar, I | 1 |
| Khan, KU | 1 |
| Zeng, LH | 1 |
| Song, Z | 1 |
| Li, S | 1 |
| Zhang, C | 1 |
| Yuan, L | 1 |
| Han, L | 1 |
| Liu, Y | 1 |
| Shi, JY | 1 |
| Li, JZ | 1 |
| Wang, WG | 1 |
| Yang, XG | 1 |
| Hou, BJ | 1 |
| Zhang, HL | 1 |
| Peng, RQ | 1 |
| Zhang, AM | 1 |
1 trial available for verapamil and Lung Injury, Acute
| Article | Year |
|---|---|
[Clinical study of the effect of verapamil-procaine compound in prevention and treatment of acute respiratory distress syndrome after high risk operation].
Topics: Acute Lung Injury; Adolescent; Adult; Drug Combinations; Female; Humans; Male; Middle Aged; Postoper | 2005 |
2 other studies available for verapamil and Lung Injury, Acute
| Article | Year |
|---|---|
Verapamil attenuates oxidative stress and inflammatory responses in cigarette smoke (CS)-induced murine models of acute lung injury and CSE-stimulated RAW 264.7 macrophages via inhibiting the NF-κB pathway.
Topics: Acute Lung Injury; Animals; Cigarette Smoking; Disease Models, Animal; Interleukin-6; Lipopolysaccha | 2022 |
The therapeutic effect of verapamil in lipopolysaccharide-induced acute lung injury.
Topics: Acute Lung Injury; Animals; Cells, Cultured; Cytokines; Disease Models, Animal; Inflammation; Inflam | 2019 |