vanadates has been researched along with Myocardial Infarction in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (66.67) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Nerthigan, Y; Pandey, S; Sharma, AK; Swaminathan, N; Wu, HF | 1 |
Duncker, DJ; Gho, BC; Gho, CC; Kazim, S; Liem, DA; Manintveld, OC; Verdouw, PD | 1 |
Aihara, K; Fukunaga, K; Hashimoto, M; Kasahara, J; Takada, Y | 1 |
3 other study(ies) available for vanadates and Myocardial Infarction
Article | Year |
---|---|
Aggregation of cysteamine-capped gold nanoparticles in presence of ATP as an analytical tool for rapid detection of creatine kinase (CK-MM).
Topics: Adenosine Triphosphate; Biomarkers; Creatine Kinase; Cysteamine; Gold; Humans; Limit of Detection; Linear Models; Metal Nanoparticles; Myocardial Infarction; Point-of-Care Testing; Sodium; Vanadates | 2018 |
The tyrosine phosphatase inhibitor bis(maltolato)oxovanadium attenuates myocardial reperfusion injury by opening ATP-sensitive potassium channels.
Topics: Adenosine Triphosphate; Animals; Blood Pressure; Cardiotonic Agents; Disease Models, Animal; Dose-Response Relationship, Drug; Enzyme Inhibitors; Heart Rate; Male; Membrane Proteins; Myocardial Infarction; Myocardial Reperfusion Injury; Potassium Channels; Protein Tyrosine Phosphatases; Pyrones; Rats; Rats, Wistar; Time Factors; Tyrosine; Vanadates | 2004 |
Cytoprotective effect of sodium orthovanadate on ischemia/reperfusion-induced injury in the rat heart involves Akt activation and inhibition of fodrin breakdown and apoptosis.
Topics: Animals; Apoptosis; Blood Pressure; Blotting, Western; Carrier Proteins; Caspase 3; Caspases; Male; Microfilament Proteins; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Myocytes, Cardiac; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Rats; Rats, Sprague-Dawley; Signal Transduction; Vanadates; Ventricular Function, Left | 2004 |