vanadates has been researched along with Carcinoma, Anaplastic in 7 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (14.29) | 18.2507 |
| 2000's | 4 (57.14) | 29.6817 |
| 2010's | 2 (28.57) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Johnson, KJ; Liu, C; Peck, AR; Rui, H; Schaber, JD; Sjolund, AB; Tran, TH; Utama, FE; Witkiewicz, AK | 1 |
| Gonçalves, AP; Máximo, V; Soares, P; Videira, A | 1 |
| Conus, NM; Cristiano, BE; Hannan, KM; Hemmings, BA; Pearson, RB | 1 |
| Banerjee, P; Chakraborty, T; Chatterjee, M; Hemalatha, P; Kanna, PS; Mahendrakumar, CB | 1 |
| Cao, Y; Dong, Z; Li, L; Liu, S; Liu, Y; Luo, W; Tang, M; Tao, Y; Wu, Y; Yan, G | 1 |
| Burns, DM; Kawase, T; Oguro, A; Orikasa, M | 1 |
| Lin, A; Marshall, MS; Yip-Schneider, MT | 1 |
7 other study(ies) available for vanadates and Carcinoma, Anaplastic
| Article | Year |
|---|---|
PTP1B suppresses prolactin activation of Stat5 in breast cancer cells.
Topics: Breast Neoplasms; Carcinoma; Down-Regulation; Drug Synergism; Dual Specificity Phosphatase 3; Enzyme Inhibitors; Female; Humans; Phosphorylation; Prolactin; Protein Tyrosine Phosphatase, Non-Receptor Type 1; Protein Tyrosine Phosphatase, Non-Receptor Type 11; Protein Tyrosine Phosphatase, Non-Receptor Type 2; Protein Tyrosine Phosphatase, Non-Receptor Type 6; STAT5 Transcription Factor; Tumor Cells, Cultured; Vanadates | 2010 |
Orthovanadate-induced cell death in RET/PTC1-harboring cancer cells involves the activation of caspases and altered signaling through PI3K/Akt/mTOR.
Topics: Antineoplastic Agents; Apoptosis; Carcinoma; Carcinoma, Papillary; Caspases; Cell Proliferation; Enzyme Activation; Glial Cell Line-Derived Neurotrophic Factor; Humans; Membrane Potential, Mitochondrial; Oncogene Protein v-akt; Oncogene Proteins, Fusion; Phosphatidylinositol 3-Kinases; Phosphorylation; Protein-Tyrosine Kinases; Reactive Oxygen Species; Signal Transduction; Thyroid Cancer, Papillary; Thyroid Neoplasms; TOR Serine-Threonine Kinases; Tumor Cells, Cultured; Vanadates | 2011 |
Direct identification of tyrosine 474 as a regulatory phosphorylation site for the Akt protein kinase.
Topics: Animals; Carcinoma; COS Cells; Culture Media, Serum-Free; Enzyme Inhibitors; Female; Genes, Reporter; Humans; Insulin-Like Growth Factor I; Mutagenesis, Site-Directed; Ovarian Neoplasms; Peptide Mapping; Phosphatidylinositol 3-Kinases; Phosphopeptides; Phosphorylation; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Recombinant Fusion Proteins; Tumor Cells, Cultured; Tyrosine; Vanadates | 2002 |
Effect of vanadium on colonic aberrant crypt foci induced in rats by 1,2 dimethyl hydrazine.
Topics: 1,2-Dimethylhydrazine; Adenoma; Animals; Carcinogens; Carcinoma; Colon; Colonic Neoplasms; Cytochrome P-450 Enzyme System; Glutathione Transferase; Male; Precancerous Conditions; Proliferating Cell Nuclear Antigen; Rats; Rats, Sprague-Dawley; Vanadates | 2003 |
Identification of novel phosphoproteins in signaling pathways triggered by latent membrane protein 1 using functional proteomics technology.
Topics: Blotting, Western; Carcinoma; Cell Line, Tumor; Chromatography, Affinity; Dose-Response Relationship, Drug; Electrophoresis, Gel, Two-Dimensional; Enzyme Inhibitors; Humans; Isoelectric Point; Mass Spectrometry; Molecular Weight; Nasopharyngeal Neoplasms; Oncogene Proteins, Viral; Phosphoproteins; Phosphorylation; Proteomics; Signal Transduction; Sodium Fluoride; Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization; Vanadates; Viral Matrix Proteins | 2006 |
Possible regulation of epidermal growth factor-receptor tyrosine autophosphorylation by calcium and G proteins in chemically permeabilized rat UMR106 cells.
Topics: Adenosine Triphosphate; Animals; Calcium; Calcium Signaling; Carcinoma; Cell Membrane Permeability; Culture Media; Dose-Response Relationship, Drug; Enzyme Inhibitors; Epidermal Growth Factor; ErbB Receptors; Genistein; GTP-Binding Proteins; Humans; Osteoblasts; Osteosarcoma; Phosphoproteins; Phosphorylation; Protein Tyrosine Phosphatases; Protein-Tyrosine Kinases; Rats; Signal Transduction; Time Factors; Tumor Cells, Cultured; Tyrosine; Vanadates | 1999 |
Pancreatic tumor cells with mutant K-ras suppress ERK activity by MEK-dependent induction of MAP kinase phosphatase-2.
Topics: Animals; Blotting, Western; Carcinoma; Cell Line; Down-Regulation; Dual-Specificity Phosphatases; Enzyme Inhibitors; Gene Expression Regulation, Enzymologic; Genes, ras; Humans; Kinetics; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinase Phosphatases; Mitogen-Activated Protein Kinases; Mutation; Pancreatic Neoplasms; Phosphorylation; Protein Phosphatase 2; Protein Tyrosine Phosphatases; Rats; Signal Transduction; Time Factors; Tumor Cells, Cultured; Up-Regulation; Vanadates | 2001 |