Page last updated: 2024-08-22

vanadates and Adult Spinal Muscular Atrophy

vanadates has been researched along with Adult Spinal Muscular Atrophy in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (66.67)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Hsieh-Li, HM; Li, H; Lin-Chao, S; Liu, HC; Ting, CH; Tsai, LK; Wen, HL1
Hsieh-Li, HM; Li, H; Lin, CW; Ting, CH; Wen, SL1
Androphy, EJ; Lorson, CL; Zhang, ML; Zhou, J1

Other Studies

3 other study(ies) available for vanadates and Adult Spinal Muscular Atrophy

ArticleYear
Sodium vanadate combined with L-ascorbic acid delays disease progression, enhances motor performance, and ameliorates muscle atrophy and weakness in mice with spinal muscular atrophy.
    BMC medicine, 2013, Feb-14, Volume: 11

    Topics: Adult; Animals; Ascorbic Acid; Cells, Cultured; Disease Progression; Drug Therapy, Combination; Feasibility Studies; Female; Humans; Mice; Mice, Knockout; Mice, Transgenic; Motor Skills; Muscle Weakness; Muscular Atrophy; Muscular Atrophy, Spinal; Vanadates

2013
Stat5 constitutive activation rescues defects in spinal muscular atrophy.
    Human molecular genetics, 2007, Mar-01, Volume: 16, Issue:5

    Topics: Aclarubicin; Animals; Axons; Cell Line; Cell Nucleus Structures; Cells, Cultured; Cyclic AMP Response Element-Binding Protein; Exons; Fibroblasts; Gene Expression Regulation; Humans; Hydroxamic Acids; Lymphocytes; Mice; Models, Biological; Motor Neurons; Muscular Atrophy, Spinal; Mutant Proteins; Nerve Tissue Proteins; Promoter Regions, Genetic; RNA-Binding Proteins; RNA, Messenger; Signal Transduction; SMN Complex Proteins; STAT5 Transcription Factor; Survival of Motor Neuron 2 Protein; Vanadates

2007
An in vivo reporter system for measuring increased inclusion of exon 7 in SMN2 mRNA: potential therapy of SMA.
    Gene therapy, 2001, Volume: 8, Issue:20

    Topics: Artificial Gene Fusion; Cell Line; Cyclic AMP Response Element-Binding Protein; Enzyme Inhibitors; Exons; Gene Expression; Genetic Therapy; Green Fluorescent Proteins; Humans; Luciferases; Luminescent Proteins; Motor Neurons; Muscular Atrophy, Spinal; Nerve Tissue Proteins; Phosphoric Monoester Hydrolases; Phosphorylation; Reverse Transcriptase Polymerase Chain Reaction; RNA Splicing; RNA-Binding Proteins; SMN Complex Proteins; Survival of Motor Neuron 1 Protein; Survival of Motor Neuron 2 Protein; Transfection; Vanadates

2001