uric acid has been researched along with Seizures, Febrile in 3 studies
Uric Acid: An oxidation product, via XANTHINE OXIDASE, of oxypurines such as XANTHINE and HYPOXANTHINE. It is the final oxidation product of purine catabolism in humans and primates, whereas in most other mammals URATE OXIDASE further oxidizes it to ALLANTOIN.
uric acid : An oxopurine that is the final oxidation product of purine metabolism.
6-hydroxy-1H-purine-2,8(7H,9H)-dione : A tautomer of uric acid having oxo groups at C-2 and C-8 and a hydroxy group at C-6.
7,9-dihydro-1H-purine-2,6,8(3H)-trione : An oxopurine in which the purine ring is substituted by oxo groups at positions 2, 6, and 8.
Seizures, Febrile: Seizures that occur during a febrile episode. It is a common condition, affecting 2-5% of children aged 3 months to five years. An autosomal dominant pattern of inheritance has been identified in some families. The majority are simple febrile seizures (generally defined as generalized onset, single seizures with a duration of less than 30 minutes). Complex febrile seizures are characterized by focal onset, duration greater than 30 minutes, and/or more than one seizure in a 24 hour period. The likelihood of developing epilepsy (i.e., a nonfebrile seizure disorder) following simple febrile seizures is low. Complex febrile seizures are associated with a moderately increased incidence of epilepsy. (From Menkes, Textbook of Child Neurology, 5th ed, p784)
| Excerpt | Relevance | Reference |
|---|---|---|
| "This suggests that simple febrile seizures neither significantly disturb the metabolism of nucleotides, nucleosides or bases, nor significantly deplete neuron adenosine triphosphate ATP levels." | 1.28 | Purine metabolites and pyrimidine bases in cerebrospinal fluid of children with simple febrile seizures. ( Camiña, F; Castro-Gago, M; Lojo, S; Rodríguez-Núñez, A; Rodríguez-Segade, S, 1991) |
| "Though children with febrile convulsions only have seizures in the early stage of a febrile illness and not later, these seizures have been attributed to the fever." | 1.28 | Hyperpolarization and short-circuiting as mechanisms of seizure prevention following febrile convulsions. ( Aiyathurai, EJ; Jacob, E; Low, PS, 1989) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (33.33) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Rodríguez-Núñez, A | 2 |
| Cid, E | 1 |
| Rodríguez-García, J | 1 |
| Camiña, F | 2 |
| Rodríguez-Segade, S | 2 |
| Castro-Gago, M | 2 |
| Lojo, S | 1 |
| Aiyathurai, EJ | 1 |
| Low, PS | 1 |
| Jacob, E | 1 |
3 other studies available for uric acid and Seizures, Febrile
| Article | Year |
|---|---|
Cerebrospinal fluid purine metabolite and neuron-specific enolase concentrations after febrile seizures.
Topics: Adenosine Monophosphate; Adolescent; Child; Child, Preschool; Female; Humans; Infant; Inosine Monoph | 2000 |
Purine metabolites and pyrimidine bases in cerebrospinal fluid of children with simple febrile seizures.
Topics: Adenine; Adenosine; Adenosine Monophosphate; Child, Preschool; Cytosine; Female; Guanine; Guanosine; | 1991 |
Hyperpolarization and short-circuiting as mechanisms of seizure prevention following febrile convulsions.
Topics: Blood Glucose; Calcium; Cerebral Cortex; Child, Preschool; Chlorides; Electroencephalography; Electr | 1989 |