uric acid has been researched along with Infections, Respiratory Syncytial Virus in 2 studies
Uric Acid: An oxidation product, via XANTHINE OXIDASE, of oxypurines such as XANTHINE and HYPOXANTHINE. It is the final oxidation product of purine catabolism in humans and primates, whereas in most other mammals URATE OXIDASE further oxidizes it to ALLANTOIN.
uric acid : An oxopurine that is the final oxidation product of purine metabolism.
6-hydroxy-1H-purine-2,8(7H,9H)-dione : A tautomer of uric acid having oxo groups at C-2 and C-8 and a hydroxy group at C-6.
7,9-dihydro-1H-purine-2,6,8(3H)-trione : An oxopurine in which the purine ring is substituted by oxo groups at positions 2, 6, and 8.
| Excerpt | Relevance | Reference |
|---|---|---|
| "To investigate the role of uric acid (UA) and IL-1β in RSV immunopathology and asthma predisposition." | 7.96 | Inhibition of uric acid or IL-1β ameliorates respiratory syncytial virus immunopathology and development of asthma. ( Best, SKK; Fonseca, W; Lukacs, NW; Malinczak, CA; Morris, SB; Ptaschinski, C; Rasky, AJ; Schuler, CF, 2020) |
| "To investigate the role of uric acid (UA) and IL-1β in RSV immunopathology and asthma predisposition." | 3.96 | Inhibition of uric acid or IL-1β ameliorates respiratory syncytial virus immunopathology and development of asthma. ( Best, SKK; Fonseca, W; Lukacs, NW; Malinczak, CA; Morris, SB; Ptaschinski, C; Rasky, AJ; Schuler, CF, 2020) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 2 (100.00) | 2.80 |
| Authors | Studies |
|---|---|
| Fonseca, W | 2 |
| Malinczak, CA | 2 |
| Schuler, CF | 2 |
| Best, SKK | 2 |
| Rasky, AJ | 2 |
| Morris, SB | 2 |
| Cui, TX | 1 |
| Popova, AP | 1 |
| Lukacs, NW | 2 |
| Ptaschinski, C | 1 |
2 other studies available for uric acid and Infections, Respiratory Syncytial Virus
| Article | Year |
|---|---|
Uric acid pathway activation during respiratory virus infection promotes Th2 immune response via innate cytokine production and ILC2 accumulation.
Topics: Animals; Cytokines; Disease Models, Animal; Female; Humans; Immunity, Innate; Inflammation Mediators | 2020 |
Inhibition of uric acid or IL-1β ameliorates respiratory syncytial virus immunopathology and development of asthma.
Topics: Animals; Asthma; Immunity, Innate; Lung; Lymphocytes; Mice; Mice, Inbred BALB C; Respiratory Syncyti | 2020 |