urea has been researched along with Bessel-Hagen Disease in 2 studies
pseudourea: clinical use; structure
isourea : A carboximidic acid that is the imidic acid tautomer of urea, H2NC(=NH)OH, and its hydrocarbyl derivatives.
Excerpt | Relevance | Reference |
---|---|---|
"Hereditary multiple exostoses (HME) is a pediatric disorder caused by heparan sulfate (HS) deficiency and is characterized by growth plate-associated osteochondromas." | 1.48 | Heparan sulfate antagonism alters bone morphogenetic protein signaling and receptor dynamics, suggesting a mechanism in hereditary multiple exostoses. ( Billings, PC; Mundy, C; Pacifici, M; Takano, H; Yang, E, 2018) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Mundy, C | 2 |
Yang, E | 1 |
Takano, H | 1 |
Billings, PC | 2 |
Pacifici, M | 2 |
Huegel, J | 1 |
Sgariglia, F | 1 |
Nygren, P | 1 |
Yamaguchi, Y | 1 |
Koyama, E | 1 |
2 other studies available for urea and Bessel-Hagen Disease
Article | Year |
---|---|
Heparan sulfate antagonism alters bone morphogenetic protein signaling and receptor dynamics, suggesting a mechanism in hereditary multiple exostoses.
Topics: Animals; Bone Morphogenetic Protein 2; Bone Morphogenetic Protein Receptors, Type II; Cells, Culture | 2018 |
Perichondrium phenotype and border function are regulated by Ext1 and heparan sulfate in developing long bones: a mechanism likely deranged in Hereditary Multiple Exostoses.
Topics: Animals; Bone and Bones; Bone Morphogenetic Protein 2; Cartilage; Chondrogenesis; Choristoma; Embryo | 2013 |