u-0126 has been researched along with Status-Epilepticus* in 2 studies
2 other study(ies) available for u-0126 and Status-Epilepticus
Article | Year |
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TRPC6-Mediated ERK1/2 Activation Increases Dentate Granule Cell Resistance to Status Epilepticus Via Regulating Lon Protease-1 Expression and Mitochondrial Dynamics.
Topics: Animals; Butadienes; Cell Death; Dentate Gyrus; Male; MAP Kinase Signaling System; Mitochondria; Mitochondrial Dynamics; Mitogen-Activated Protein Kinase 12; Mitogen-Activated Protein Kinase 3; Neurons; Nitriles; Phosphorylation; Protease La; Rats; Rats, Sprague-Dawley; Status Epilepticus; TRPC Cation Channels | 2019 |
TRPC6-mediated ERK1/2 phosphorylation prevents dentate granule cell degeneration via inhibiting mitochondrial elongation.
Topics: Animals; Butadienes; Disease Models, Animal; Dynamins; Enzyme Inhibitors; Male; Mitochondria; Mitochondrial Dynamics; Mitogen-Activated Protein Kinase 12; Muscarinic Agonists; Nerve Degeneration; Nerve Tissue Proteins; Neurons; Nitriles; Phosphorylation; Pilocarpine; Rats; Rats, Sprague-Dawley; RNA, Small Interfering; Signal Transduction; Status Epilepticus; TRPC Cation Channels | 2017 |