tyrphostin a23 has been researched along with Ischemic Attack, Transient in 1 studies
tyrphostin A23: inhibits EGF-stimulated thymidine incorporation as well as EGF-stimulated receptor autophosphorylation & tyrosine phosphorylation & cell proliferation; structure given in first source
Ischemic Attack, Transient: Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (100.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kim, CJ | 1 |
| Kim, KW | 1 |
| Park, JW | 1 |
| Lee, JC | 1 |
| Zhang, JH | 1 |
1 other study available for tyrphostin a23 and Ischemic Attack, Transient
| Article | Year |
|---|---|
Role of tyrosine kinase in erythrocyte lysate-induced contraction in rabbit cerebral arteries.
Topics: Animals; Basilar Artery; Catechols; Enzyme Activation; Enzyme Inhibitors; Erythrocytes; Estrogens, N | 1998 |