tryptophan has been researched along with Cardiac Remodeling, Ventricular in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 4 (66.67) | 24.3611 |
2020's | 2 (33.33) | 2.80 |
Authors | Studies |
---|---|
Alcaide, P; Anastasiou, M; Aronovitz, M; Carrillo-Salinas, FJ; Jetton, D; Kaur, K; Ngwenyama, N; Smolgovsky, SA; Tai, A | 1 |
Hsu, WT; Jui, HY; Kuo, CC; Lee, CM; Tseng, YH; Wu, KK | 1 |
Ablasser, K; Alesutan, I; Belyavskiy, E; Brussee, H; Catena, C; Fahrleitner-Pammer, A; Grübler, MR; Kolesnik, E; Lewinski, DV; März, W; Meinitzer, A; Pieske, B; Pieske-Kraigher, E; Pilz, S; Schwetz, V; Sechi, LA; Tomaschitz, A; Trummer, C; Verheyen, N; Voelkl, J | 1 |
Chen, YH; Chou, CH; Ho, YL; Huang, CC; Hung, CS; Kuo, CC; Lee, CM; Lin, YH; Liu, LD; Wu, KK; Yeh, JF | 1 |
Ashraf, M; Haider, HKh; Pasha, Z | 1 |
Janicki, JS; Law, BA; Levick, SP; Li, J; Meléndez, GC; Supowit, SC | 1 |
1 trial(s) available for tryptophan and Cardiac Remodeling, Ventricular
Article | Year |
---|---|
Low-grade inflammation and tryptophan-kynurenine pathway activation are associated with adverse cardiac remodeling in primary hyperparathyroidism: the EPATH trial.
Topics: Aged; Biomarkers; C-Reactive Protein; Cross-Sectional Studies; Echocardiography; Female; Humans; Hyperparathyroidism, Primary; Inflammation; Kynurenine; Male; Quinolinic Acid; Tryptophan; Ventricular Remodeling | 2017 |
5 other study(ies) available for tryptophan and Cardiac Remodeling, Ventricular
Article | Year |
---|---|
Gut dysbiosis induced by cardiac pressure overload enhances adverse cardiac remodeling in a T cell-dependent manner.
Topics: Animals; Basic Helix-Loop-Helix Transcription Factors; Disease Models, Animal; Dysbiosis; Endomyocardial Fibrosis; Fatty Acids, Volatile; Gastrointestinal Microbiome; Heart Failure; Hypertrophy, Left Ventricular; Hypertrophy, Right Ventricular; Inflammation; Lymphocyte Activation; Lymphocyte Depletion; Male; Mice; Mice, Inbred C57BL; Receptors, Aryl Hydrocarbon; T-Lymphocytes; Tryptophan; Ventricular Pressure; Ventricular Remodeling | 2020 |
5-Methoxytryptophan attenuates postinfarct cardiac injury by controlling oxidative stress and immune activation.
Topics: Animals; Apoptosis; Cell Line, Transformed; Disease Models, Animal; Female; Human Umbilical Vein Endothelial Cells; Humans; Hydrogen Peroxide; Immunity; Myocardial Infarction; Myocarditis; Myocardium; Myocytes, Cardiac; Oxidative Stress; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Signal Transduction; Treatment Outcome; Tryptophan; Ventricular Function, Left; Ventricular Remodeling | 2021 |
5-methoxytryptophan is a potential marker for post-myocardial infarction heart failure - a preliminary approach to clinical utility.
Topics: Aged; Biomarkers; Echocardiography; Female; Follow-Up Studies; Heart Failure; Humans; Male; Middle Aged; Prognosis; Prospective Studies; ROC Curve; ST Elevation Myocardial Infarction; Time Factors; Tryptophan; Ventricular Remodeling | 2016 |
Efficient non-viral reprogramming of myoblasts to stemness with a single small molecule to generate cardiac progenitor cells.
Topics: Animals; Biomarkers; Cell Differentiation; Cellular Reprogramming; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; Embryonic Stem Cells; Heart Function Tests; Indoles; Induced Pluripotent Stem Cells; Male; Mice; MicroRNAs; Myoblasts, Skeletal; Myocardial Contraction; Myocardial Infarction; Myocardium; Oligonucleotide Array Sequence Analysis; Phthalimides; Propionates; Stem Cell Transplantation; Teratoma; Tryptophan; Ventricular Remodeling; Viruses | 2011 |
Substance P induces adverse myocardial remodelling via a mechanism involving cardiac mast cells.
Topics: Animals; Apoptosis; Cell Degranulation; Collagen; Disease Models, Animal; Heart Failure; In Situ Nick-End Labeling; Male; Mast Cells; Matrix Metalloproteinases; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardium; Neurokinin A; Neurokinin-1 Receptor Antagonists; Rats; Rats, Sprague-Dawley; Receptors, Neurokinin-1; Substance P; Time Factors; Tryptophan; Tumor Necrosis Factor-alpha; Ultrasonography; Ventricular Remodeling | 2011 |