tryptophan and Benign Familial Infantile Convulsions

tryptophan has been researched along with Benign Familial Infantile Convulsions in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Annunziato, L; Hernandez, CC; Miceli, F; Shapiro, MS; Soldovieri, MV; Taglialatela, M1
Hirose, S; Hiroyasu, K; Ishii, A; Jibiki, I; Kaneko, S; Nakatsu, F; Ohno, H; Okada, M; Sugiura, Y; Ugawa, Y1

Other Studies

2 other study(ies) available for tryptophan and Benign Familial Infantile Convulsions

ArticleYear
Gating consequences of charge neutralization of arginine residues in the S4 segment of K(v)7.2, an epilepsy-linked K+ channel subunit.
    Biophysical journal, 2008, Volume: 95, Issue:5

    Topics: Amino Acid Sequence; Amino Acid Substitution; Animals; Arginine; CHO Cells; Cricetinae; Cricetulus; Electrophysiology; Epilepsy, Benign Neonatal; Glutamine; Humans; Ion Channel Gating; KCNQ2 Potassium Channel; Models, Molecular; Molecular Sequence Data; Mutagenesis, Site-Directed; Mutation; Patch-Clamp Techniques; Tryptophan

2008
Lack of potassium current in W309R mutant KCNQ3 channel causing benign familial neonatal convulsions (BFNC).
    Epilepsy research, 2009, Volume: 84, Issue:1

    Topics: Animals; Arginine; Cell Line, Transformed; Electric Stimulation; Epilepsy, Benign Neonatal; Humans; KCNQ3 Potassium Channel; Membrane Potentials; Mice; Mutation; Neural Conduction; Patch-Clamp Techniques; Transfection; Tryptophan

2009