tryptamine has been researched along with Hepatic Encephalopathy in 3 studies
Hepatic Encephalopathy: A syndrome characterized by central nervous system dysfunction in association with LIVER FAILURE, including portal-systemic shunts. Clinical features include lethargy and CONFUSION (frequently progressing to COMA); ASTERIXIS; NYSTAGMUS, PATHOLOGIC; brisk oculovestibular reflexes; decorticate and decerebrate posturing; MUSCLE SPASTICITY; and bilateral extensor plantar reflexes (see REFLEX, BABINSKI). ELECTROENCEPHALOGRAPHY may demonstrate triphasic waves. (From Adams et al., Principles of Neurology, 6th ed, pp1117-20; Plum & Posner, Diagnosis of Stupor and Coma, 3rd ed, p222-5)
| Excerpt | Relevance | Reference |
|---|---|---|
| "The distribution of [3H]tryptamine binding sites, in autopsied brain tissue from cirrhotic patients with hepatic encephalopathy (HE) and an equal number of age-matched control subjects free from hepatic, neurological, or psychiatric disorder, was investigated." | 7.69 | Region-selective decreases in densities of [3H]tryptamine binding sites in autopsied brain tissue from cirrhotic patients with hepatic encephalopathy. ( Butterworth, RF; Layrargues, GP; Mousseau, DD, 1994) |
| "Several combined pathogenetic factors such as hyperammonemia, different brain tryptophan metabolic disturbances and serotonin physiological/pharmacological alterations not yet defined in all details, will often give rise to the clinical neuropsychiatric condition known as hepatic encephalopathy (HE)." | 4.80 | Brain tryptophan/serotonin perturbations in metabolic encephalopathy and the hazards involved in the use of psychoactive drugs. ( Bengtsson, F, 1999) |
| "The distribution of [3H]tryptamine binding sites, in autopsied brain tissue from cirrhotic patients with hepatic encephalopathy (HE) and an equal number of age-matched control subjects free from hepatic, neurological, or psychiatric disorder, was investigated." | 3.69 | Region-selective decreases in densities of [3H]tryptamine binding sites in autopsied brain tissue from cirrhotic patients with hepatic encephalopathy. ( Butterworth, RF; Layrargues, GP; Mousseau, DD, 1994) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (33.33) | 18.7374 |
| 1990's | 2 (66.67) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Mousseau, DD | 1 |
| Layrargues, GP | 1 |
| Butterworth, RF | 1 |
| Bengtsson, F | 1 |
| Kienzl, E | 1 |
| Riederer, P | 1 |
| Brücke, T | 1 |
| Kleinberger, G | 1 |
| Jellinger, K | 1 |
2 reviews available for tryptamine and Hepatic Encephalopathy
| Article | Year |
|---|---|
Brain tryptophan/serotonin perturbations in metabolic encephalopathy and the hazards involved in the use of psychoactive drugs.
Topics: Animals; Brain; Hepatic Encephalopathy; Humans; Indoles; Psychotropic Drugs; Quinolinic Acid; Seroto | 1999 |
[Changes in and modulation of receptor activity in hepatic encephalopathy].
Topics: Ammonia; Animals; Aspartic Acid; Binding, Competitive; Biogenic Amines; Blood-Brain Barrier; Brain; | 1985 |
1 other study available for tryptamine and Hepatic Encephalopathy
| Article | Year |
|---|---|
Region-selective decreases in densities of [3H]tryptamine binding sites in autopsied brain tissue from cirrhotic patients with hepatic encephalopathy.
Topics: Autopsy; Binding Sites; Brain; Hepatic Encephalopathy; Humans; Kinetics; Liver Cirrhosis; Male; Midd | 1994 |