Page last updated: 2024-08-21

trimethyltin chloride and Encephalopathy, Toxic

trimethyltin chloride has been researched along with Encephalopathy, Toxic in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chen, C; Chen, M; Deng, P; He, M; Hong, H; Liu, M; Lu, Y; Pi, H; Reiter, RJ; Tian, L; Xi, Y; Xie, J; Xu, S; Yu, Z; Zhang, L; Zhou, C; Zhou, Z1
Higuchi, K; Kawada, K; Kuramoto, N; Nagashima, R; Ogita, K; Shuto, M; Sugiyama, C; Yoneyama, M1
Bal-Price, AK; Coecke, S; Hartung, T; Hogberg, HT; Kinsner-Ovaskainen, A1

Other Studies

3 other study(ies) available for trimethyltin chloride and Encephalopathy, Toxic

ArticleYear
Inhibition of SERPINA3N-dependent neuroinflammation is essential for melatonin to ameliorate trimethyltin chloride-induced neurotoxicity.
    Journal of pineal research, 2019, Volume: 67, Issue:3

    Topics: Acute-Phase Proteins; Animals; Hippocampus; Male; Melatonin; Mice; Mice, Inbred C57BL; Neurons; Neurotoxicity Syndromes; Serpins; Trimethyltin Compounds

2019
Endogenous and exogenous glucocorticoids prevent trimethyltin from causing neuronal degeneration of the mouse brain in vivo: involvement of oxidative stress pathways.
    Journal of pharmacological sciences, 2009, Volume: 110, Issue:4

    Topics: Adrenalectomy; Animals; Brain; Calpain; Caspase 3; Dexamethasone; Glucocorticoids; Male; Mice; Mifepristone; Nerve Degeneration; Neurotoxicity Syndromes; Oxidative Stress; Rabbits; Signal Transduction; Trimethyltin Compounds

2009
mRNA expression is a relevant tool to identify developmental neurotoxicants using an in vitro approach.
    Toxicological sciences : an official journal of the Society of Toxicology, 2010, Volume: 113, Issue:1

    Topics: Animals; Animals, Newborn; Cell Differentiation; Cell Proliferation; Cell Survival; Cells, Cultured; Cerebellum; Dose-Response Relationship, Drug; Environmental Pollutants; Gene Expression Regulation; Genetic Markers; Glial Fibrillary Acidic Protein; Humans; Intermediate Filament Proteins; Lead; Methylmercury Compounds; Nerve Tissue Proteins; Nestin; Neurofilament Proteins; Neuroglia; Neurons; Neurotoxicity Syndromes; Rats; Rats, Wistar; Receptors, GABA-A; Receptors, N-Methyl-D-Aspartate; Risk Assessment; Risk Factors; RNA, Messenger; SOXE Transcription Factors; Time Factors; Toxicity Tests; Trimethyltin Compounds; Valproic Acid

2010