trimethyloxamine has been researched along with Inflammation in 64 studies
trimethyloxamine: used in manufacture of quaternary ammonium cpds; insect attractant; warming agent for gas; oxidant; structure
trimethylamine N-oxide : A tertiary amine oxide resulting from the oxidation of the amino group of trimethylamine.
Inflammation: A pathological process characterized by injury or destruction of tissues caused by a variety of cytologic and chemical reactions. It is usually manifested by typical signs of pain, heat, redness, swelling, and loss of function.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Endothelial dysfunction is a critical initiating factor contributing to cardiovascular diseases, involving the gut microbiome-derived metabolite trimethylamine N-oxide (TMAO)." | 8.31 | Time-dependent specific molecular signatures of inflammation and remodelling are associated with trimethylamine-N-oxide (TMAO)-induced endothelial cell dysfunction. ( Bellanger, S; Cheong, KH; Chin, YL; Devasia, AG; Leo, CH; Ong, ES; Ramasamy, A; Shanmugham, M, 2023) |
| "Evidence shows that trimethylamine (TMA)/trimethylamine-N-oxide (TMAO) is closely related to non-alcoholic fatty liver disease (NAFLD)." | 8.12 | Changes of flavin-containing monooxygenases and trimethylamine-N-oxide may be involved in the promotion of non-alcoholic fatty liver disease by intestinal microbiota metabolite trimethylamine. ( Cao, P; Chen, Q; Deng, W; Gong, Z; Guo, J; Li, X; Pei, M; Shi, C; Wang, L; Wang, Y; Zhang, L, 2022) |
| "Iron-overload leads to gut dysbiosis/inflammation and disturbance of metabolites, and deferiprone alleviates those conditions more effectively in WT than in those that are thalassemic." | 8.12 | Deferiprone has less benefits on gut microbiota and metabolites in high iron-diet induced iron overload thalassemic mice than in iron overload wild-type mice: A preclinical study. ( Buddhasiri, S; Chattipakorn, N; Chattipakorn, SC; Fucharoen, S; Kittichotirat, W; Kumfu, S; Nawara, W; Sarichai, P; Sriwichaiin, S; Thiennimitr, P; Thonusin, C, 2022) |
| "Trimethylamine-N-oxide (TMAO), a gut-microbiota-dependent metabolite after ingesting dietary choline, has been identified as a novel risk factor for atherosclerosis through inducing vascular inflammation." | 8.12 | Gut-Flora-Dependent Metabolite Trimethylamine-N-Oxide Promotes Atherosclerosis-Associated Inflammation Responses by Indirect ROS Stimulation and Signaling Involving AMPK and SIRT1. ( Hong, Y; Ji, N; Luo, X; Ma, W; Nie, Z; Shan, J; Xue, J; Zhang, T; Zhang, Y; Zhou, S; Zhu, W, 2022) |
| " We aimed to evaluate whether residual risk of recurrent stroke of TMAO and its precursor choline remain among patients who received dual-antiplatelet therapy and intensive lipid-lowering therapy and with a low inflammation level (high-sensitivity C-reactive protein <2 mg/L on admission)." | 8.12 | Residual Risk of Trimethylamine-N-Oxide and Choline for Stroke Recurrence in Patients With Intensive Secondary Therapy. ( Cheng, A; Jiang, X; Jin, A; Li, H; Li, K; Lin, J; Meng, X; Wang, Y; Xu, J; Xue, J; Zhao, M; Zheng, L, 2022) |
| "Chronic inflammation is a key factor that accelerates the progression of inflammatory vascular disease." | 5.91 | Hydrogen sulfide attenuates TMAO‑induced macrophage inflammation through increased SIRT1 sulfhydration. ( Lin, XL; Liu, MH; Xiao, LL, 2023) |
| "Atherosclerosis is a chronic inflammatory disease of the arterial wall involving inflammation, redox imbalance, and impaired cholesterol transport." | 5.72 | Chronic oral trimethylamine-N-oxide administration induces experimental incipient atherosclerosis in non-genetically modified mice. ( Ancuta, B; Cismaru, G; Decea, N; Filip, GA; Florea, CM; Moldovan, R; Rosu, R; Toma, V; Vlase, L, 2022) |
| "Endothelial dysfunction is a critical initiating factor contributing to cardiovascular diseases, involving the gut microbiome-derived metabolite trimethylamine N-oxide (TMAO)." | 4.31 | Time-dependent specific molecular signatures of inflammation and remodelling are associated with trimethylamine-N-oxide (TMAO)-induced endothelial cell dysfunction. ( Bellanger, S; Cheong, KH; Chin, YL; Devasia, AG; Leo, CH; Ong, ES; Ramasamy, A; Shanmugham, M, 2023) |
| "Evidence shows that trimethylamine (TMA)/trimethylamine-N-oxide (TMAO) is closely related to non-alcoholic fatty liver disease (NAFLD)." | 4.12 | Changes of flavin-containing monooxygenases and trimethylamine-N-oxide may be involved in the promotion of non-alcoholic fatty liver disease by intestinal microbiota metabolite trimethylamine. ( Cao, P; Chen, Q; Deng, W; Gong, Z; Guo, J; Li, X; Pei, M; Shi, C; Wang, L; Wang, Y; Zhang, L, 2022) |
| " Therefore, PSRC1 overexpression and reduced choline consumption may further alleviate atherosclerosis." | 4.12 | Deficiency of proline/serine-rich coiled-coil protein 1 (PSRC1) accelerates trimethylamine N-oxide-induced atherosclerosis in ApoE ( Chen, M; Chen, P; Guo, Z; Liu, D; Luo, T; Ou, C, 2022) |
| "Iron-overload leads to gut dysbiosis/inflammation and disturbance of metabolites, and deferiprone alleviates those conditions more effectively in WT than in those that are thalassemic." | 4.12 | Deferiprone has less benefits on gut microbiota and metabolites in high iron-diet induced iron overload thalassemic mice than in iron overload wild-type mice: A preclinical study. ( Buddhasiri, S; Chattipakorn, N; Chattipakorn, SC; Fucharoen, S; Kittichotirat, W; Kumfu, S; Nawara, W; Sarichai, P; Sriwichaiin, S; Thiennimitr, P; Thonusin, C, 2022) |
| "Trimethylamine-N-oxide (TMAO), a gut-microbiota-dependent metabolite after ingesting dietary choline, has been identified as a novel risk factor for atherosclerosis through inducing vascular inflammation." | 4.12 | Gut-Flora-Dependent Metabolite Trimethylamine-N-Oxide Promotes Atherosclerosis-Associated Inflammation Responses by Indirect ROS Stimulation and Signaling Involving AMPK and SIRT1. ( Hong, Y; Ji, N; Luo, X; Ma, W; Nie, Z; Shan, J; Xue, J; Zhang, T; Zhang, Y; Zhou, S; Zhu, W, 2022) |
| " We aimed to evaluate whether residual risk of recurrent stroke of TMAO and its precursor choline remain among patients who received dual-antiplatelet therapy and intensive lipid-lowering therapy and with a low inflammation level (high-sensitivity C-reactive protein <2 mg/L on admission)." | 4.12 | Residual Risk of Trimethylamine-N-Oxide and Choline for Stroke Recurrence in Patients With Intensive Secondary Therapy. ( Cheng, A; Jiang, X; Jin, A; Li, H; Li, K; Lin, J; Meng, X; Wang, Y; Xu, J; Xue, J; Zhao, M; Zheng, L, 2022) |
| " We aimed to assess the correlation between circulating TMAO concentration and the risk of all-cause and cardiovascular death in CKD patients of different dialysis statuses and different races by dose-response analyses, and the underlying mechanisms were also explored by analyzing the correlations of TMAO with glomerular filtration rate (GFR) and inflammation." | 3.01 | Gut microbiota-derived trimethylamine N-oxide is associated with the risk of all-cause and cardiovascular mortality in patients with chronic kidney disease: a systematic review and dose-response meta-analysis. ( Guo, J; Li, Y; Liu, W; Liu, Y; Lu, H; Zhang, M; Zheng, H, 2023) |
| "Hypertension is the most prevalent chronic disease and a risk factor for various diseases." | 2.72 | TMA/TMAO in Hypertension: Novel Horizons and Potential Therapies. ( Ding, YJ; Jia, QJ; Li, YY; Lv, SC; Wang, YJ; Zhang, A; Zhang, JP; Zhang, WQ; Zhang, XN; Zhu, YP, 2021) |
| "Inflammation is the key for the initiation and progression of atherosclerosis." | 2.66 | Mutual Interplay of Host Immune System and Gut Microbiota in the Immunopathology of Atherosclerosis. ( Chen, YH; Kao, HL; Liu, SF; Wu, MS; Wu, WK; Yang, KC; Yeh, CF, 2020) |
| "Dysbiosis is associated with intestinal inflammation and reduced integrity of the gut barrier, which in turn increases circulating levels of bacterial structural components and microbial metabolites that may facilitate the development of CVD." | 2.58 | The gut microbiota as a novel regulator of cardiovascular function and disease. ( Battson, ML; Gentile, CL; Lee, DM; Weir, TL, 2018) |
| "Inflammation is believed to play a key role by providing matrix-degrading metalloproteinases and also by inducing death of matrix-synthesizing smooth muscle cells." | 2.50 | Biomarkers of plaque instability. ( Shah, PK, 2014) |
| "Chronic inflammation is a key factor that accelerates the progression of inflammatory vascular disease." | 1.91 | Hydrogen sulfide attenuates TMAO‑induced macrophage inflammation through increased SIRT1 sulfhydration. ( Lin, XL; Liu, MH; Xiao, LL, 2023) |
| "Notably, endotoxemia was used as a surrogate marker of gut leakage in patients." | 1.91 | TMAO reductase, a biomarker for gut permeability defect induced inflammation, in mouse model of chronic kidney disease and dextran sulfate solution-induced mucositis. ( Boonhai, S; Bootdee, K; Leelahavanichkul, A; Saisorn, W; Sitticharoenchai, P; Takkavatakarn, K; Tiranathanagul, K; Tungsanga, S, 2023) |
| "Atherosclerosis is a hallmark of cardiovascular disease, and lifestyle strongly impacts its onset and progression." | 1.72 | TMAO Upregulates Members of the miR-17/92 Cluster and Impacts Targets Associated with Atherosclerosis. ( Blanco, R; Daimiel, L; Dávalos, A; Díez-Ricote, L; Micó, V; Ordovás, JM; Ruiz-Valderrey, P; Tomé-Carneiro, J, 2022) |
| "Periodontitis was induced by unilateral ligation of the first molar in mice, and 3,3-dimethyl-1-butanol (DMB) was used as an inhibitor to reduce TMAO circulating." | 1.72 | Gut microbiota-dependent trimethylamine n-oxide pathway contributes to the bidirectional relationship between intestinal inflammation and periodontitis. ( A, L; Jiang, C; Sun, Y; Wang, Q; Xu, W; Zhou, T, 2022) |
| "Atherosclerosis is a chronic inflammatory disease of the arterial wall involving inflammation, redox imbalance, and impaired cholesterol transport." | 1.72 | Chronic oral trimethylamine-N-oxide administration induces experimental incipient atherosclerosis in non-genetically modified mice. ( Ancuta, B; Cismaru, G; Decea, N; Filip, GA; Florea, CM; Moldovan, R; Rosu, R; Toma, V; Vlase, L, 2022) |
| "Cardiac function, plasma TMAO level, cardiac hypertrophy and fibrosis, expression of inflammatory, electrophysiological studies and signaling pathway were analyzed at the sixth week after AB surgery." | 1.56 | 3,3-Dimethyl-1-butanol attenuates cardiac remodeling in pressure-overload-induced heart failure mice. ( Fu, H; Huang, H; Jiang, X; Kong, B; Shuai, W; Wang, G, 2020) |
| "Obesity is considered an important factor that increases the risk of colorectal cancer (CRC)." | 1.56 | Gut Microbiota-Mediated Inflammation and Gut Permeability in Patients with Obesity and Colorectal Cancer. ( Gómez-Millán, J; Laborda-Illanes, A; Medina, JA; Ordóñez, R; Otero, A; Plaza-Andrade, I; Queipo-Ortuño, MI; Ramos-Molina, B; Sánchez-Alcoholado, L, 2020) |
| "Choline was not significantly altered in MetS." | 1.48 | Changes to trimethylamine-N-oxide and its precursors in nascent metabolic syndrome. ( Huet, B; Jialal, I; Lent-Schochet, D; McLaughlin, M; Silva, R, 2018) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 27 (42.19) | 24.3611 |
| 2020's | 37 (57.81) | 2.80 |
| Authors | Studies |
|---|---|
| Zhang, L | 4 |
| Xie, F | 1 |
| Tang, H | 2 |
| Zhang, X | 3 |
| Hu, J | 1 |
| Zhong, X | 1 |
| Gong, N | 1 |
| Lai, Y | 2 |
| Zhou, M | 2 |
| Tian, J | 1 |
| Zhou, Z | 2 |
| Xie, L | 1 |
| Hu, Z | 2 |
| Zhu, F | 2 |
| Jiang, J | 1 |
| Nie, J | 2 |
| Yang, G | 1 |
| Shi, C | 1 |
| Pei, M | 1 |
| Wang, Y | 6 |
| Chen, Q | 1 |
| Cao, P | 1 |
| Guo, J | 2 |
| Deng, W | 1 |
| Wang, L | 1 |
| Li, X | 1 |
| Gong, Z | 1 |
| Li, C | 4 |
| Zhu, L | 1 |
| Dai, Y | 1 |
| Zhang, Z | 1 |
| Huang, L | 1 |
| Wang, TJ | 1 |
| Fu, P | 1 |
| Li, Y | 3 |
| Wang, J | 3 |
| Jiang, C | 3 |
| Liu, H | 2 |
| Jia, K | 1 |
| Ren, Z | 1 |
| Sun, J | 1 |
| Pan, LL | 1 |
| Luo, T | 1 |
| Liu, D | 1 |
| Guo, Z | 2 |
| Chen, P | 1 |
| Ou, C | 1 |
| Chen, M | 1 |
| Sriwichaiin, S | 1 |
| Thiennimitr, P | 1 |
| Thonusin, C | 1 |
| Sarichai, P | 1 |
| Buddhasiri, S | 1 |
| Kumfu, S | 1 |
| Nawara, W | 1 |
| Kittichotirat, W | 1 |
| Fucharoen, S | 1 |
| Chattipakorn, N | 1 |
| Chattipakorn, SC | 1 |
| Jiang, WY | 1 |
| Huo, JY | 1 |
| Wang, SC | 1 |
| Cheng, YD | 1 |
| Lyu, YT | 1 |
| Jiang, ZX | 1 |
| Shan, QJ | 1 |
| Zhou, S | 1 |
| Xue, J | 2 |
| Shan, J | 1 |
| Hong, Y | 4 |
| Zhu, W | 2 |
| Nie, Z | 1 |
| Zhang, Y | 3 |
| Ji, N | 1 |
| Luo, X | 1 |
| Zhang, T | 2 |
| Ma, W | 1 |
| Xu, J | 3 |
| Zhao, M | 1 |
| Jin, A | 1 |
| Cheng, A | 1 |
| Jiang, X | 6 |
| Li, K | 1 |
| Lin, J | 1 |
| Meng, X | 1 |
| Li, H | 2 |
| Zheng, L | 1 |
| Díez-Ricote, L | 1 |
| Ruiz-Valderrey, P | 1 |
| Micó, V | 1 |
| Blanco, R | 1 |
| Tomé-Carneiro, J | 1 |
| Dávalos, A | 1 |
| Ordovás, JM | 1 |
| Daimiel, L | 1 |
| Saaoud, F | 4 |
| Liu, L | 3 |
| Xu, K | 3 |
| Cueto, R | 3 |
| Shao, Y | 3 |
| Lu, Y | 3 |
| Sun, Y | 6 |
| Snyder, NW | 3 |
| Wu, S | 3 |
| Yang, L | 3 |
| Zhou, Y | 3 |
| Williams, DL | 3 |
| Martinez, L | 3 |
| Vazquez-Padron, RI | 3 |
| Zhao, H | 3 |
| Wang, H | 5 |
| Yang, X | 4 |
| Deng, Y | 3 |
| Zou, J | 3 |
| Peng, Q | 3 |
| Fu, X | 3 |
| Duan, R | 3 |
| Chen, J | 4 |
| Chen, X | 3 |
| Kul, S | 2 |
| Caliskan, Z | 2 |
| Guvenc, TS | 2 |
| Celik, FB | 2 |
| Sarmis, A | 2 |
| Atici, A | 2 |
| Konal, O | 2 |
| Akıl, M | 2 |
| Cumen, AS | 2 |
| Bilgic, NM | 2 |
| Yilmaz, Y | 2 |
| Caliskan, M | 2 |
| Wang, Q | 2 |
| Zhou, T | 1 |
| A, L | 1 |
| Xu, W | 1 |
| Tu, R | 1 |
| Xia, J | 1 |
| Tacconi, E | 1 |
| Palma, G | 1 |
| De Biase, D | 1 |
| Luciano, A | 1 |
| Barbieri, M | 1 |
| de Nigris, F | 1 |
| Bruzzese, F | 1 |
| Florea, CM | 2 |
| Rosu, R | 2 |
| Cismaru, G | 1 |
| Moldovan, R | 2 |
| Vlase, L | 1 |
| Toma, V | 1 |
| Decea, N | 2 |
| Ancuta, B | 1 |
| Filip, GA | 2 |
| Baldea, I | 1 |
| Liu, MH | 1 |
| Lin, XL | 1 |
| Xiao, LL | 1 |
| Lu, H | 1 |
| Zhang, M | 2 |
| Zheng, H | 1 |
| Liu, Y | 3 |
| Liu, W | 1 |
| Hemmati, M | 1 |
| Kashanipoor, S | 1 |
| Mazaheri, P | 1 |
| Alibabaei, F | 1 |
| Babaeizad, A | 1 |
| Asli, S | 1 |
| Mohammadi, S | 1 |
| Gorgin, AH | 1 |
| Ghods, K | 1 |
| Yousefi, B | 1 |
| Eslami, M | 1 |
| Shanmugham, M | 1 |
| Devasia, AG | 1 |
| Chin, YL | 1 |
| Cheong, KH | 1 |
| Ong, ES | 1 |
| Bellanger, S | 1 |
| Ramasamy, A | 1 |
| Leo, CH | 1 |
| Chan, MM | 1 |
| Fong, D | 1 |
| Zhao, L | 1 |
| Zhang, C | 2 |
| Cao, G | 1 |
| Dong, X | 1 |
| Li, D | 2 |
| Jiang, L | 1 |
| Hou, J | 1 |
| Wang, G | 1 |
| Kong, B | 1 |
| Shuai, W | 1 |
| Fu, H | 1 |
| Huang, H | 1 |
| Liu, J | 2 |
| Si, C | 1 |
| Wang, X | 2 |
| Wang, RT | 1 |
| Lv, Z | 2 |
| Dai, M | 1 |
| Sánchez-Alcoholado, L | 1 |
| Ordóñez, R | 1 |
| Otero, A | 1 |
| Plaza-Andrade, I | 1 |
| Laborda-Illanes, A | 1 |
| Medina, JA | 1 |
| Ramos-Molina, B | 1 |
| Gómez-Millán, J | 1 |
| Queipo-Ortuño, MI | 1 |
| Macpherson, ME | 1 |
| Hov, JR | 1 |
| Ueland, T | 1 |
| Dahl, TB | 1 |
| Kummen, M | 1 |
| Otterdal, K | 1 |
| Holm, K | 1 |
| Berge, RK | 2 |
| Mollnes, TE | 1 |
| Trøseid, M | 3 |
| Halvorsen, B | 1 |
| Aukrust, P | 1 |
| Fevang, B | 1 |
| Jørgensen, SF | 1 |
| Yeh, CF | 1 |
| Chen, YH | 1 |
| Liu, SF | 1 |
| Kao, HL | 1 |
| Wu, MS | 1 |
| Yang, KC | 1 |
| Wu, WK | 1 |
| Shan, X | 1 |
| Tu, Q | 1 |
| Yang, Y | 1 |
| Eshghjoo, S | 1 |
| Jayaraman, A | 1 |
| Alaniz, RC | 1 |
| Zhao, X | 1 |
| Chen, Y | 2 |
| Li, L | 2 |
| Zhai, J | 1 |
| Yu, B | 1 |
| Yang, D | 1 |
| Chang, Y | 1 |
| Li, J | 2 |
| Zhang, P | 1 |
| Zhang, H | 1 |
| Zhang, WQ | 1 |
| Wang, YJ | 1 |
| Zhang, A | 1 |
| Ding, YJ | 1 |
| Zhang, XN | 1 |
| Jia, QJ | 1 |
| Zhu, YP | 1 |
| Li, YY | 1 |
| Lv, SC | 1 |
| Zhang, JP | 1 |
| Yuzefpolskaya, M | 1 |
| Bohn, B | 1 |
| Javaid, A | 1 |
| Mondellini, GM | 1 |
| Braghieri, L | 1 |
| Pinsino, A | 1 |
| Onat, D | 1 |
| Cagliostro, B | 1 |
| Kim, A | 1 |
| Takeda, K | 1 |
| Naka, Y | 1 |
| Farr, M | 1 |
| Sayer, GT | 1 |
| Uriel, N | 1 |
| Nandakumar, R | 1 |
| Mohan, S | 1 |
| Colombo, PC | 1 |
| Demmer, RT | 1 |
| Boonhai, S | 1 |
| Bootdee, K | 1 |
| Saisorn, W | 1 |
| Takkavatakarn, K | 1 |
| Sitticharoenchai, P | 1 |
| Tungsanga, S | 1 |
| Tiranathanagul, K | 1 |
| Leelahavanichkul, A | 1 |
| Yoo, W | 1 |
| Zieba, JK | 1 |
| Foegeding, NJ | 1 |
| Torres, TP | 1 |
| Shelton, CD | 1 |
| Shealy, NG | 1 |
| Byndloss, AJ | 1 |
| Cevallos, SA | 1 |
| Gertz, E | 1 |
| Tiffany, CR | 1 |
| Thomas, JD | 1 |
| Litvak, Y | 1 |
| Nguyen, H | 1 |
| Olsan, EE | 1 |
| Bennett, BJ | 1 |
| Rathmell, JC | 1 |
| Major, AS | 1 |
| Bäumler, AJ | 1 |
| Byndloss, MX | 1 |
| Hove-Skovsgaard, M | 1 |
| Gaardbo, JC | 1 |
| Kolte, L | 1 |
| Winding, K | 1 |
| Seljeflot, I | 1 |
| Svardal, A | 1 |
| Gerstoft, J | 1 |
| Ullum, H | 1 |
| Nielsen, SD | 1 |
| Sun, G | 1 |
| Yin, Z | 1 |
| Liu, N | 1 |
| Bian, X | 1 |
| Yu, R | 1 |
| Su, X | 1 |
| Zhang, B | 1 |
| Battson, ML | 1 |
| Lee, DM | 1 |
| Weir, TL | 1 |
| Gentile, CL | 1 |
| Li, DY | 1 |
| Tang, WHW | 1 |
| Missailidis, C | 2 |
| Neogi, U | 1 |
| Stenvinkel, P | 2 |
| Nowak, P | 1 |
| Bergman, P | 2 |
| Lent-Schochet, D | 1 |
| Silva, R | 1 |
| McLaughlin, M | 1 |
| Huet, B | 1 |
| Jialal, I | 1 |
| Rhainds, D | 1 |
| Brodeur, MR | 1 |
| Tardif, JC | 1 |
| Shan, Z | 1 |
| Clish, CB | 1 |
| Hua, S | 1 |
| Scott, JM | 1 |
| Hanna, DB | 1 |
| Burk, RD | 1 |
| Haberlen, SA | 1 |
| Shah, SJ | 1 |
| Margolick, JB | 1 |
| Sears, CL | 1 |
| Post, WS | 1 |
| Landay, AL | 1 |
| Lazar, JM | 1 |
| Hodis, HN | 1 |
| Anastos, K | 1 |
| Kaplan, RC | 1 |
| Qi, Q | 1 |
| Haghikia, A | 2 |
| Li, XS | 1 |
| Liman, TG | 1 |
| Bledau, N | 1 |
| Schmidt, D | 1 |
| Zimmermann, F | 1 |
| Kränkel, N | 1 |
| Widera, C | 1 |
| Sonnenschein, K | 1 |
| Weissenborn, K | 1 |
| Fraccarollo, D | 1 |
| Heimesaat, MM | 1 |
| Bauersachs, J | 1 |
| Wang, Z | 1 |
| Bavendiek, U | 1 |
| Hazen, SL | 1 |
| Endres, M | 1 |
| Landmesser, U | 1 |
| Li, T | 1 |
| Wu, H | 1 |
| Shi, H | 1 |
| Bai, J | 1 |
| Zhao, W | 1 |
| Jiang, D | 1 |
| Chen, H | 1 |
| Li, N | 3 |
| Tang, J | 1 |
| Meng, F | 1 |
| Song, B | 1 |
| Mantziaris, V | 1 |
| Kolios, G | 1 |
| Makhija, L | 1 |
| Krishnan, V | 1 |
| Rehman, R | 1 |
| Chakraborty, S | 1 |
| Maity, S | 1 |
| Mabalirajan, U | 1 |
| Chakraborty, K | 1 |
| Ghosh, B | 1 |
| Agrawal, A | 1 |
| Gao, X | 2 |
| Liu, X | 1 |
| Xue, C | 2 |
| Xue, Y | 2 |
| Shah, PK | 1 |
| Li, Z | 1 |
| Kaysen, GA | 1 |
| Johansen, KL | 1 |
| Chertow, GM | 1 |
| Dalrymple, LS | 1 |
| Kornak, J | 1 |
| Grimes, B | 1 |
| Dwyer, T | 1 |
| Chassy, AW | 1 |
| Fiehn, O | 1 |
| Yazdekhasti, N | 1 |
| Brandsch, C | 1 |
| Schmidt, N | 1 |
| Schloesser, A | 1 |
| Huebbe, P | 1 |
| Rimbach, G | 1 |
| Stangl, GI | 1 |
| Rohrmann, S | 1 |
| Linseisen, J | 1 |
| Allenspach, M | 1 |
| von Eckardstein, A | 1 |
| Müller, D | 1 |
| Hällqvist, J | 1 |
| Qureshi, AR | 1 |
| Barany, P | 1 |
| Heimbürger, O | 1 |
| Lindholm, B | 1 |
| Sun, X | 1 |
| Jiao, X | 1 |
| Ma, Y | 1 |
| He, Y | 1 |
| Wu, D | 1 |
| Cao, M | 1 |
| Peng, J | 1 |
| Yi, S | 1 |
| Song, L | 1 |
| Zhao, J | 1 |
| Willyard, C | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Effects of a Whole Food Based Nutritional Formulation on Trimethylamine N-oxide and Cardiometabolic Endpoints in Healthy Adults.[NCT05795946] | 45 participants (Anticipated) | Interventional | 2023-04-15 | Recruiting | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
14 reviews available for trimethyloxamine and Inflammation
| Article | Year |
|---|---|
Stroke and Vascular Cognitive Impairment: The Role of Intestinal Microbiota Metabolite TMAO.
Topics: Choline; Cognitive Dysfunction; Gastrointestinal Microbiome; Humans; Inflammation; Stroke; Thrombosi | 2024 |
Microbiota Effect on Trimethylamine N-Oxide Production: From Cancer to Fitness-A Practical Preventing Recommendation and Therapies.
Topics: Animals; Cardiovascular Diseases; Choline; Inflammation; Methylamines; Microbiota; Neoplasms | 2023 |
Gut microbiota-derived trimethylamine N-oxide is associated with the risk of all-cause and cardiovascular mortality in patients with chronic kidney disease: a systematic review and dose-response meta-analysis.
Topics: Cardiovascular Diseases; Gastrointestinal Microbiome; Humans; Inflammation; Renal Insufficiency, Chr | 2023 |
Importance of gut microbiota metabolites in the development of cardiovascular diseases (CVD).
Topics: Atherosclerosis; Cardiovascular Diseases; Dysbiosis; Gastrointestinal Microbiome; Humans; Inflammati | 2023 |
The Microbial Metabolite Trimethylamine N-Oxide Links Vascular Dysfunctions and the Autoimmune Disease Rheumatoid Arthritis.
Topics: Amyloid; Animals; Arthritis, Rheumatoid; Autoimmune Diseases; Cardiovascular Diseases; Diet; Dysbios | 2019 |
Trimethylamine N-Oxide Generated by the Gut Microbiota Is Associated with Vascular Inflammation: New Insights into Atherosclerosis.
Topics: Atherosclerosis; Gastrointestinal Microbiome; Humans; Inflammation; Methylamines | 2020 |
Mutual Interplay of Host Immune System and Gut Microbiota in the Immunopathology of Atherosclerosis.
Topics: Animals; Atherosclerosis; Clinical Trials as Topic; Cytokines; Disease Progression; Dysbiosis; Fatty | 2020 |
Microbiota-Mediated Immune Regulation in Atherosclerosis.
Topics: Animals; Atherosclerosis; Basic Helix-Loop-Helix Transcription Factors; Foam Cells; Gastrointestinal | 2021 |
TMA/TMAO in Hypertension: Novel Horizons and Potential Therapies.
Topics: Animals; Carnitine; Choline; Gastrointestinal Microbiome; Glucose; Humans; Hypertension; Inflammatio | 2021 |
The gut microbiota as a novel regulator of cardiovascular function and disease.
Topics: Aging; Animals; Anti-Bacterial Agents; Atherosclerosis; Bile Acids and Salts; Cardiovascular Disease | 2018 |
Contributory Role of Gut Microbiota and Their Metabolites Toward Cardiovascular Complications in Chronic Kidney Disease.
Topics: Animals; Cardiovascular Diseases; Cresols; Diet Therapy; Dietary Supplements; Dysbiosis; Enzyme Inhi | 2018 |
Lipids, Apolipoproteins, and Inflammatory Biomarkers of Cardiovascular Risk: What Have We Learned?
Topics: Animals; Apolipoproteins; Biomarkers; Cardiovascular Diseases; Dyslipidemias; Gastrointestinal Micro | 2018 |
Gut Microbiota, Atherosclerosis, and Therapeutic Targets.
Topics: Atherosclerosis; Disease Management; Gastrointestinal Microbiome; Humans; Inflammation; Methylamines | 2019 |
Biomarkers of plaque instability.
Topics: Antigens, Human Platelet; Apolipoprotein A-I; Atherosclerosis; Biomarkers; C-Reactive Protein; Coron | 2014 |
1 trial available for trimethyloxamine and Inflammation
| Article | Year |
|---|---|
Effect of DLT-SML on Chronic Stable Angina Through Ameliorating Inflammation, Correcting Dyslipidemia, and Regulating Gut Microbiota.
Topics: Adult; Aged; Angina, Stable; Anti-Inflammatory Agents; Bacteria; Biomarkers; China; Cytokines; Drug | 2021 |
49 other studies available for trimethyloxamine and Inflammation
| Article | Year |
|---|---|
Gut microbial metabolite TMAO increases peritoneal inflammation and peritonitis risk in peritoneal dialysis patients.
Topics: Adult; Animals; Cell Death; Cytokines; Epithelium; Female; Gastrointestinal Microbiome; Glucose; Hum | 2022 |
Trimethylamine-N-Oxide Aggravates Kidney Injury via Activation of p38/MAPK Signaling and Upregulation of HuR.
Topics: Animals; ELAV-Like Protein 1; Inflammation; Male; Methylamines; p38 Mitogen-Activated Protein Kinase | 2022 |
Trimethylamine N-oxide promotes hyperlipidemia acute pancreatitis via inflammatory response.
Topics: Animals; ATP Binding Cassette Transporter, Subfamily B, Member 1; Cytokines; Disease Models, Animal; | 2022 |
Changes of flavin-containing monooxygenases and trimethylamine-N-oxide may be involved in the promotion of non-alcoholic fatty liver disease by intestinal microbiota metabolite trimethylamine.
Topics: Animals; Cell Line; Endoplasmic Reticulum Chaperone BiP; Gastrointestinal Microbiome; Gene Silencing | 2022 |
Diet-Induced High Serum Levels of Trimethylamine-N-oxide Enhance the Cellular Inflammatory Response without Exacerbating Acute Intracerebral Hemorrhage Injury in Mice.
Topics: Acute Disease; Animals; Astrocytes; Brain Injuries; Cerebral Hemorrhage; Choline; Diet; Disease Mode | 2022 |
PRMT5 critically mediates TMAO-induced inflammatory response in vascular smooth muscle cells.
Topics: Animals; Inflammation; Methylamines; Mice; Muscle, Smooth, Vascular; Rats; Vascular Cell Adhesion Mo | 2022 |
Deficiency of proline/serine-rich coiled-coil protein 1 (PSRC1) accelerates trimethylamine N-oxide-induced atherosclerosis in ApoE
Topics: Animals; Atherosclerosis; Cholesterol; Cholesterol, LDL; Choline; Inflammation; Leukocytes, Mononucl | 2022 |
Deferiprone has less benefits on gut microbiota and metabolites in high iron-diet induced iron overload thalassemic mice than in iron overload wild-type mice: A preclinical study.
Topics: Animals; Cytokines; Deferiprone; Diet; Dysbiosis; Gastrointestinal Microbiome; Inflammation; Iron; I | 2022 |
Trimethylamine N-oxide facilitates the progression of atrial fibrillation in rats with type 2 diabetes by aggravating cardiac inflammation and connexin remodeling.
Topics: Animals; Atrial Fibrillation; Atrial Remodeling; Connexins; Diabetes Mellitus, Experimental; Diabete | 2022 |
Gut-Flora-Dependent Metabolite Trimethylamine-N-Oxide Promotes Atherosclerosis-Associated Inflammation Responses by Indirect ROS Stimulation and Signaling Involving AMPK and SIRT1.
Topics: AMP-Activated Protein Kinases; Animals; Atherosclerosis; Choline; Gastrointestinal Microbiome; Infla | 2022 |
Residual Risk of Trimethylamine-N-Oxide and Choline for Stroke Recurrence in Patients With Intensive Secondary Therapy.
Topics: C-Reactive Protein; Choline; Humans; Inflammation; Lipids; Methylamines; Oxides; Platelet Aggregatio | 2022 |
TMAO Upregulates Members of the miR-17/92 Cluster and Impacts Targets Associated with Atherosclerosis.
Topics: Animals; Atherosclerosis; Betaine; Cardiovascular Diseases; Carnitine; Choline; Humans; Inflammation | 2022 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways.
Topics: Animals; Aorta; Cardiovascular Diseases; Endothelial Cells; Humans; Inflammation; Intercellular Adhe | 2023 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Higher Circulating Trimethylamine N-Oxide Aggravates Cognitive Impairment Probably via Downregulating Hippocampal SIRT1 in Vascular Dementia Rats.
Topics: Animals; Cognitive Dysfunction; Dementia, Vascular; Hippocampus; Inflammation; Rats; Sirtuin 1; Vasc | 2022 |
Gut microbiota-derived metabolite trimethylamine N-oxide and biomarkers of inflammation are linked to endothelial and coronary microvascular function in patients with inflammatory bowel disease.
Topics: Biomarkers; Colitis, Ulcerative; Crohn Disease; Gastrointestinal Microbiome; Humans; Inflammation; I | 2023 |
Gut microbiota-derived metabolite trimethylamine N-oxide and biomarkers of inflammation are linked to endothelial and coronary microvascular function in patients with inflammatory bowel disease.
Topics: Biomarkers; Colitis, Ulcerative; Crohn Disease; Gastrointestinal Microbiome; Humans; Inflammation; I | 2023 |
Gut microbiota-derived metabolite trimethylamine N-oxide and biomarkers of inflammation are linked to endothelial and coronary microvascular function in patients with inflammatory bowel disease.
Topics: Biomarkers; Colitis, Ulcerative; Crohn Disease; Gastrointestinal Microbiome; Humans; Inflammation; I | 2023 |
Gut microbiota-derived metabolite trimethylamine N-oxide and biomarkers of inflammation are linked to endothelial and coronary microvascular function in patients with inflammatory bowel disease.
Topics: Biomarkers; Colitis, Ulcerative; Crohn Disease; Gastrointestinal Microbiome; Humans; Inflammation; I | 2023 |
Gut microbiota-dependent trimethylamine n-oxide pathway contributes to the bidirectional relationship between intestinal inflammation and periodontitis.
Topics: Animals; Gastrointestinal Microbiome; Inflammation; Methylamines; Mice; Periodontitis | 2022 |
Chronic oral trimethylamine-N-oxide administration induces experimental incipient atherosclerosis in non-genetically modified mice.
Topics: Animals; Atherosclerosis; C-Reactive Protein; Cholesterol; Inflammation; Male; Mice; Oxides; Rats; R | 2022 |
The Acute Effect of Trimethylamine-N-Oxide on Vascular Function, Oxidative Stress, and Inflammation in Rat Aortic Rings.
Topics: Animals; Inflammation; NF-E2-Related Factor 2; NF-kappa B; Oxidative Stress; Oxides; Rats; Superoxid | 2023 |
Hydrogen sulfide attenuates TMAO‑induced macrophage inflammation through increased SIRT1 sulfhydration.
Topics: Cystathionine gamma-Lyase; Humans; Hydrogen Sulfide; Inflammation; Interleukin-6; Macrophages; NF-ka | 2023 |
Time-dependent specific molecular signatures of inflammation and remodelling are associated with trimethylamine-N-oxide (TMAO)-induced endothelial cell dysfunction.
Topics: Endothelial Cells; Humans; Inflammation; Methylamines; Oxides; Vascular Diseases | 2023 |
Higher Circulating Trimethylamine N-oxide Sensitizes Sevoflurane-Induced Cognitive Dysfunction in Aged Rats Probably by Downregulating Hippocampal Methionine Sulfoxide Reductase A.
Topics: Animals; Cognitive Dysfunction; Down-Regulation; Fear; Hippocampus; Inflammation; Interleukin-1beta; | 2019 |
The Presence of High Levels of Circulating Trimethylamine N-Oxide Exacerbates Central and Peripheral Inflammation and Inflammatory Hyperalgesia in Rats Following Carrageenan Injection.
Topics: Animals; Carrageenan; Edema; Hyperalgesia; Inflammation; Inflammation Mediators; Methylamines; NF-ka | 2019 |
3,3-Dimethyl-1-butanol attenuates cardiac remodeling in pressure-overload-induced heart failure mice.
Topics: Animals; Cardiomegaly; Disease Models, Animal; Echocardiography; Electrocardiography; Fibroblasts; H | 2020 |
Baicalin ameliorates neuropathology in repeated cerebral ischemia-reperfusion injury model mice by remodeling the gut microbiota.
Topics: Animals; Brain; Clusterin; Cytokines; Disease Models, Animal; Flavonoids; Gastrointestinal Microbiom | 2020 |
Gut Microbiota-Mediated Inflammation and Gut Permeability in Patients with Obesity and Colorectal Cancer.
Topics: Aged; Bacteria; Biomarkers; Body Mass Index; Colorectal Neoplasms; Dysbiosis; Feces; Female; Gastroi | 2020 |
Gut Microbiota-Dependent Trimethylamine N-Oxide Associates With Inflammation in Common Variable Immunodeficiency.
Topics: Adult; Bacteria; Bacterial Proteins; Biomarkers; Carnitine; Common Variable Immunodeficiency; Diet; | 2020 |
Ginkgolide B treatment regulated intestinal flora to improve high-fat diet induced atherosclerosis in ApoE
Topics: Animals; Atherosclerosis; Bacteroides; Diet, High-Fat; Disease Models, Animal; Fibrinolytic Agents; | 2021 |
Levels of Trimethylamine N-Oxide Remain Elevated Long Term After Left Ventricular Assist Device and Heart Transplantation and Are Independent From Measures of Inflammation and Gut Dysbiosis.
Topics: Aged; Aged, 80 and over; Dysbiosis; Female; Gastrointestinal Microbiome; Heart Failure; Heart Transp | 2021 |
TMAO reductase, a biomarker for gut permeability defect induced inflammation, in mouse model of chronic kidney disease and dextran sulfate solution-induced mucositis.
Topics: Animals; Biomarkers; Colitis; Dextran Sulfate; Endotoxemia; Inflammation; Interleukin-6; Mice; Mucos | 2023 |
High-fat diet-induced colonocyte dysfunction escalates microbiota-derived trimethylamine
Topics: Animals; Cell Hypoxia; Choline; Colon; Diet, High-Fat; Energy Metabolism; Epithelial Cells; Escheric | 2021 |
HIV-infected persons with type 2 diabetes show evidence of endothelial dysfunction and increased inflammation.
Topics: Arginine; Biomarkers; Cardiovascular Diseases; Case-Control Studies; Chromatography, High Pressure L | 2017 |
Gut microbial metabolite TMAO contributes to renal dysfunction in a mouse model of diet-induced obesity.
Topics: Animals; Diet, High-Fat; Disease Models, Animal; Gastrointestinal Microbiome; Hemodynamics; Inflamma | 2017 |
The microbial metabolite trimethylamine-N-oxide in association with inflammation and microbial dysregulation in three HIV cohorts at various disease stages.
Topics: Adult; Aged; Bacterial Translocation; Cardiovascular Diseases; Cluster Analysis; DNA, Bacterial; DNA | 2018 |
Changes to trimethylamine-N-oxide and its precursors in nascent metabolic syndrome.
Topics: Adult; Aged; Biomarkers; Carnitine; Choline; Female; Humans; Inflammation; Male; Metabolic Syndrome; | 2018 |
Gut Microbial-Related Choline Metabolite Trimethylamine-N-Oxide Is Associated With Progression of Carotid Artery Atherosclerosis in HIV Infection.
Topics: Atherosclerosis; Biomarkers; Carotid Arteries; Carotid Artery Diseases; Choline; Female; Gastrointes | 2018 |
Gut Microbiota-Dependent Trimethylamine N-Oxide Predicts Risk of Cardiovascular Events in Patients With Stroke and Is Related to Proinflammatory Monocytes.
Topics: Animals; Antigens, CD; Antigens, Differentiation, T-Lymphocyte; Brain Ischemia; Cardiovascular Disea | 2018 |
Lactobacillus rhamnosus GG strain mitigated the development of obstructive sleep apnea-induced hypertension in a high salt diet via regulating TMAO level and CD4
Topics: Animals; CD4-Positive T-Lymphocytes; Hypertension; Inflammation; Inflammation Mediators; Lacticaseib | 2019 |
Increased circulating trimethylamine N-oxide plays a contributory role in the development of endothelial dysfunction and hypertension in the RUPP rat model of preeclampsia.
Topics: Animals; Disease Models, Animal; Endothelium, Vascular; Female; Hexanols; Inflammation; Interleukin- | 2019 |
The presence of elevated circulating trimethylamine N-oxide exaggerates postoperative cognitive dysfunction in aged rats.
Topics: Age Factors; Animals; Brain; Cognitive Dysfunction; Disease Models, Animal; Gastrointestinal Microbi | 2019 |
Chemical chaperones mitigate experimental asthma by attenuating endoplasmic reticulum stress.
Topics: Airway Remodeling; Animals; Asthma; Endoplasmic Reticulum; Endoplasmic Reticulum Stress; Glycerol; I | 2014 |
Dietary trimethylamine N-oxide exacerbates impaired glucose tolerance in mice fed a high fat diet.
Topics: Adipose Tissue; Animals; Blood Glucose; Chemokine CCL2; Diet, High-Fat; Dietary Fats; Fasting; Gluco | 2014 |
Fish oil ameliorates trimethylamine N-oxide-exacerbated glucose intolerance in high-fat diet-fed mice.
Topics: Adiponectin; Adipose Tissue; Animals; Blood Glucose; Chemokine CCL2; Cholesterol, HDL; Cholesterol, | 2015 |
Associations of Trimethylamine N-Oxide With Nutritional and Inflammatory Biomarkers and Cardiovascular Outcomes in Patients New to Dialysis.
Topics: Biomarkers; C-Reactive Protein; Cardiovascular Diseases; Chromatography, Liquid; Cohort Studies; Com | 2015 |
Fish protein increases circulating levels of trimethylamine-N-oxide and accelerates aortic lesion formation in apoE null mice.
Topics: Animals; Aorta, Thoracic; Apolipoproteins E; Atherosclerosis; Disease Models, Animal; Fish Proteins; | 2016 |
Plasma Concentrations of Trimethylamine-N-oxide Are Directly Associated with Dairy Food Consumption and Low-Grade Inflammation in a German Adult Population.
Topics: Adult; Animals; Betaine; C-Reactive Protein; Choline; Dairy Products; Diet; Diet Surveys; Feeding Be | 2016 |
Serum Trimethylamine-N-Oxide Is Strongly Related to Renal Function and Predicts Outcome in Chronic Kidney Disease.
Topics: Adult; Aged; Betaine; Biomarkers; C-Reactive Protein; Cardiovascular Diseases; Choline; Female; Fibr | 2016 |
Trimethylamine N-oxide induces inflammation and endothelial dysfunction in human umbilical vein endothelial cells via activating ROS-TXNIP-NLRP3 inflammasome.
Topics: Carrier Proteins; Cells, Cultured; Dose-Response Relationship, Drug; Endothelium, Vascular; Human Um | 2016 |
The effect of trimethylamine N-oxide on Helicobacter pylori-induced changes of immunoinflammatory genes expression in gastric epithelial cells.
Topics: Cell Line; Chemokine CXCL1; Gastric Mucosa; Gene Expression Profiling; Helicobacter Infections; Heli | 2017 |
Pathology: At the heart of the problem.
Topics: Animals; Bacteria; Cardiovascular Diseases; Cholesterol, HDL; Cholesterol, LDL; Diet; Gastrointestin | 2013 |