Page last updated: 2024-10-20

trimethylamine and Hyperplasia

trimethylamine has been researched along with Hyperplasia in 1 studies

Hyperplasia: An increase in the number of cells in a tissue or organ without tumor formation. It differs from HYPERTROPHY, which is an increase in bulk without an increase in the number of cells.

Research Excerpts

ExcerptRelevanceReference
"Through hematoxylin and eosin staining and immunohistochemistry staining, we found that supplementary TMAO promoted balloon injury-induced neointimal hyperplasia, while reducing TMAO by antibiotic administration produced the opposite result."8.12Trimethylamine-N-oxide (TMAO) promotes balloon injury-induced neointimal hyperplasia via upregulating Beclin1 and impairing autophagic flux. ( Chen, D; Hong, Q; Huang, G; Que, D; Yan, J; Yang, P; Zhai, W; Zhong, C, 2022)
"Through hematoxylin and eosin staining and immunohistochemistry staining, we found that supplementary TMAO promoted balloon injury-induced neointimal hyperplasia, while reducing TMAO by antibiotic administration produced the opposite result."4.12Trimethylamine-N-oxide (TMAO) promotes balloon injury-induced neointimal hyperplasia via upregulating Beclin1 and impairing autophagic flux. ( Chen, D; Hong, Q; Huang, G; Que, D; Yan, J; Yang, P; Zhai, W; Zhong, C, 2022)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's1 (100.00)2.80

Authors

AuthorsStudies
Hong, Q1
Que, D1
Zhong, C1
Huang, G1
Zhai, W1
Chen, D1
Yan, J1
Yang, P1

Other Studies

1 other study available for trimethylamine and Hyperplasia

ArticleYear
Trimethylamine-N-oxide (TMAO) promotes balloon injury-induced neointimal hyperplasia via upregulating Beclin1 and impairing autophagic flux.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2022, Volume: 155

    Topics: Animals; Anti-Bacterial Agents; Beclin-1; Carotid Artery Injuries; Cell Proliferation; Endothelial C

2022