triiodothyronine has been researched along with Liver Failure, Acute in 4 studies
Triiodothyronine: A T3 thyroid hormone normally synthesized and secreted by the thyroid gland in much smaller quantities than thyroxine (T4). Most T3 is derived from peripheral monodeiodination of T4 at the 5' position of the outer ring of the iodothyronine nucleus. The hormone finally delivered and used by the tissues is mainly T3.
3,3',5-triiodo-L-thyronine : An iodothyronine compound having iodo substituents at the 3-, 3'- and 5-positions. Although some is produced in the thyroid, most of the 3,3',5-triiodo-L-thyronine in the body is generated by mono-deiodination of L-thyroxine in the peripheral tissues. Its metabolic activity is about 3 to 5 times that of L-thyroxine. The sodium salt is used in the treatment of hypothyroidism.
Liver Failure, Acute: A form of rapid-onset LIVER FAILURE, also known as fulminant hepatic failure, caused by severe liver injury or massive loss of HEPATOCYTES. It is characterized by sudden development of liver dysfunction and JAUNDICE. Acute liver failure may progress to exhibit cerebral dysfunction even HEPATIC COMA depending on the etiology that includes hepatic ISCHEMIA, drug toxicity, malignant infiltration, and viral hepatitis such as post-transfusion HEPATITIS B and HEPATITIS C.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Acute hepatic failure is a rare and potentially lethal complication of propylthiouracil (PTU) use for hyperthyroidism." | 7.72 | Successful treatment of hyperthyroidism with amiodarone in a patient with propylthiouracil-induced acute hepatic failure. ( Arteaga, E; Brusco, F; González, G; Soto, N, 2004) |
| "Acute hepatic failure is a rare and potentially lethal complication of propylthiouracil (PTU) use for hyperthyroidism." | 3.72 | Successful treatment of hyperthyroidism with amiodarone in a patient with propylthiouracil-induced acute hepatic failure. ( Arteaga, E; Brusco, F; González, G; Soto, N, 2004) |
| "However, the relationship between acute liver failure (ALF) and thyroid hormone levels has not yet been clarified." | 1.42 | Higher Thyroid-Stimulating Hormone, Triiodothyronine and Thyroxine Values Are Associated with Better Outcome in Acute Liver Failure. ( Anastasiou, O; Bechmann, LP; Canbay, A; Führer, D; Gerken, G; Gieseler, RK; Katsounas, A; Manka, P; Moeller, LC; Sowa, JP; Sydor, S; Syn, WK, 2015) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Liu, K | 1 |
| Chen, X | 1 |
| Ren, Y | 1 |
| Liu, C | 1 |
| Zhang, J | 1 |
| Wang, Z | 1 |
| Li, Y | 1 |
| Zhang, Y | 1 |
| Anastasiou, O | 1 |
| Sydor, S | 1 |
| Sowa, JP | 1 |
| Manka, P | 1 |
| Katsounas, A | 1 |
| Syn, WK | 1 |
| Führer, D | 1 |
| Gieseler, RK | 1 |
| Bechmann, LP | 1 |
| Gerken, G | 1 |
| Moeller, LC | 1 |
| Canbay, A | 1 |
| Brusco, F | 1 |
| González, G | 1 |
| Soto, N | 1 |
| Arteaga, E | 1 |
| Malik, R | 1 |
| Saich, R | 1 |
| Rahman, T | 1 |
| Hodgson, H | 1 |
4 other studies available for triiodothyronine and Liver Failure, Acute
| Article | Year |
|---|---|
3,3',5-triiodo-l-thyronine inhibits drug-induced liver injury through activation of PPARα as revealed by network pharmacology and biological experimental verification.
Topics: Acetaminophen; Chemical and Drug Induced Liver Injury; Humans; Liver; Liver Failure, Acute; Network | 2022 |
Higher Thyroid-Stimulating Hormone, Triiodothyronine and Thyroxine Values Are Associated with Better Outcome in Acute Liver Failure.
Topics: Adult; Female; Humans; Liver Failure, Acute; Liver Transplantation; Male; Retrospective Studies; Thy | 2015 |
Successful treatment of hyperthyroidism with amiodarone in a patient with propylthiouracil-induced acute hepatic failure.
Topics: Adult; Amiodarone; Female; Humans; Hyperthyroidism; Liver Failure, Acute; Propylthiouracil; Triiodot | 2004 |
During thioacetamide-induced acute liver failure, the proliferative response of hepatocytes to thyroid hormone is maintained, indicating a potential therapeutic approach to toxin-induced liver disease.
Topics: Animals; Cell Proliferation; Dose-Response Relationship, Drug; Hepatocytes; Liver Failure, Acute; Li | 2006 |