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triiodothyronine and Chromosome Deletion

triiodothyronine has been researched along with Chromosome Deletion in 18 studies

Triiodothyronine: A T3 thyroid hormone normally synthesized and secreted by the thyroid gland in much smaller quantities than thyroxine (T4). Most T3 is derived from peripheral monodeiodination of T4 at the 5' position of the outer ring of the iodothyronine nucleus. The hormone finally delivered and used by the tissues is mainly T3.
3,3',5-triiodo-L-thyronine : An iodothyronine compound having iodo substituents at the 3-, 3'- and 5-positions. Although some is produced in the thyroid, most of the 3,3',5-triiodo-L-thyronine in the body is generated by mono-deiodination of L-thyroxine in the peripheral tissues. Its metabolic activity is about 3 to 5 times that of L-thyroxine. The sodium salt is used in the treatment of hypothyroidism.

Chromosome Deletion: Actual loss of portion of a chromosome.

Research

Studies (18)

TimeframeStudies, this research(%)All Research%
pre-19909 (50.00)18.7374
1990's9 (50.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Zhang, XK1
Hoffmann, B1
Tran, PB1
Graupner, G1
Pfahl, M1
Giralt, M1
Park, EA1
Gurney, AL1
Liu, JS1
Hakimi, P1
Hanson, RW1
Ono, S1
Schwartz, ID1
Mueller, OT1
Root, AW1
Usala, SJ1
Bercu, BB1
Yamamori, I1
Mori, Y1
Seo, H1
Hirooka, Y1
Imamura, S1
Miura, Y1
Matsui, N1
Oiso, Y1
Lin, KH1
Parkison, C1
McPhie, P1
Cheng, SY1
Voz, ML1
Peers, B1
Belayew, A1
Martial, JA1
O'Donnell, AL1
Koenig, RJ2
Tsika, RW1
Bahl, JJ1
Leinwand, LA1
Morkin, E1
Petty, KJ1
Desvergne, B1
Mitsuhashi, T1
Nikodem, VM1
Copp, RP2
Samuels, HH2
Gurr, JA1
Kourides, IA1
Chatterjee, VK1
Lee, JK1
Rentoumis, A1
Jameson, JL1
Carr, FE1
Burnside, J1
Chin, WW1
Dana, S1
Karin, M1
Medeiros-Neto, G1
Targovnik, H1
Knobel, M1
Propato, F1
Varela, V1
Alkmin, M1
Barbosa, S1
Wajchenberg, BL1
Flug, F1
Casanova, J1
Horowitz, ZD1
Janocko, L1
Plotnick, M1
Brent, GA1
Warne, RL1
Larsen, PR1
Moore, DD1
Wight, PA1
Crew, MD1
Spindler, SR1

Other Studies

18 other studies available for triiodothyronine and Chromosome Deletion

ArticleYear
Retinoid X receptor is an auxiliary protein for thyroid hormone and retinoic acid receptors.
    Nature, 1992, Jan-30, Volume: 355, Issue:6359

    Topics: Animals; Base Sequence; Carrier Proteins; Cell Line; Chromosome Deletion; Cloning, Molecular; DNA-Bi

1992
Identification of a thyroid hormone response element in the phosphoenolpyruvate carboxykinase (GTP) gene. Evidence for synergistic interaction between thyroid hormone and cAMP cis-regulatory elements.
    The Journal of biological chemistry, 1991, Nov-15, Volume: 266, Issue:32

    Topics: Base Sequence; Binding Sites; Carcinoma, Hepatocellular; Cell Line; Chromosome Deletion; Cyclic AMP;

1991
Homozygosity for a dominant negative thyroid hormone receptor gene responsible for generalized resistance to thyroid hormone.
    The Journal of clinical endocrinology and metabolism, 1991, Volume: 73, Issue:5

    Topics: Adolescent; Adult; Cells, Cultured; Child, Preschool; Chromosome Deletion; DNA; DNA Restriction Enzy

1991
Nucleotide deletion resulting in frameshift as a possible cause of complete thyroxine-binding globulin deficiency in six Japanese families.
    The Journal of clinical endocrinology and metabolism, 1991, Volume: 73, Issue:2

    Topics: Amino Acid Sequence; Base Sequence; Chromosome Deletion; DNA; Female; Frameshift Mutation; Humans; J

1991
An essential role of domain D in the hormone-binding activity of human beta 1 thyroid hormone nuclear receptor.
    Molecular endocrinology (Baltimore, Md.), 1991, Volume: 5, Issue:4

    Topics: Base Sequence; Binding, Competitive; Blotting, Northern; Blotting, Western; Chromosome Deletion; Dos

1991
Characterization of an unusual thyroid response unit in the promoter of the human placental lactogen gene.
    The Journal of biological chemistry, 1991, Jul-15, Volume: 266, Issue:20

    Topics: Animals; Base Sequence; Binding Sites; Cell Line; Cell Nucleus; Chloramphenicol O-Acetyltransferase;

1991
Mutational analysis identifies a new functional domain of the thyroid hormone receptor.
    Molecular endocrinology (Baltimore, Md.), 1990, Volume: 4, Issue:5

    Topics: Amino Acid Sequence; Animals; Binding Sites; Chromosome Deletion; DNA Mutational Analysis; Gene Expr

1990
Thyroid hormone regulates expression of a transfected human alpha-myosin heavy-chain fusion gene in fetal rat heart cells.
    Proceedings of the National Academy of Sciences of the United States of America, 1990, Volume: 87, Issue:1

    Topics: Animals; Cells, Cultured; Chloramphenicol O-Acetyltransferase; Chromosome Deletion; Fetus; Gene Expr

1990
Identification of a thyroid hormone response element in the malic enzyme gene.
    The Journal of biological chemistry, 1990, May-05, Volume: 265, Issue:13

    Topics: Animals; Base Sequence; Cell Line; Chromosome Deletion; DNA Probes; Gene Expression Regulation; Gene

1990
Identification of an adenosine 3',5'-monophosphate (cAMP)-responsive region in the rat growth hormone gene: evidence for independent and synergistic effects of cAMP and thyroid hormone on gene expression.
    Molecular endocrinology (Baltimore, Md.), 1989, Volume: 3, Issue:5

    Topics: 1-Methyl-3-isobutylxanthine; Animals; Base Sequence; Cell Line; Chromosome Deletion; Colforsin; Cycl

1989
Regulation of the transfected human glycoprotein hormone alpha-subunit gene by dexamethasone and thyroid hormone.
    DNA (Mary Ann Liebert, Inc.), 1989, Volume: 8, Issue:7

    Topics: Animals; Cell Line; Chloramphenicol O-Acetyltransferase; Chromosome Deletion; Dexamethasone; Gene Ex

1989
Negative regulation of the thyroid-stimulating hormone alpha gene by thyroid hormone: receptor interaction adjacent to the TATA box.
    Proceedings of the National Academy of Sciences of the United States of America, 1989, Volume: 86, Issue:23

    Topics: Base Sequence; Cell Line; Cell Nucleus; Choriocarcinoma; Chromosome Deletion; Female; Gene Expressio

1989
Thyroid hormones regulate rat thyrotropin beta gene promoter activity expressed in GH3 cells.
    Molecular endocrinology (Baltimore, Md.), 1989, Volume: 3, Issue:4

    Topics: Animals; Cells, Cultured; Chimera; Chloramphenicol O-Acetyltransferase; Chromosome Deletion; Endonuc

1989
Induction of human growth hormone promoter activity by the adenosine 3',5'-monophosphate pathway involves a novel responsive element.
    Molecular endocrinology (Baltimore, Md.), 1989, Volume: 3, Issue:5

    Topics: Animals; Cell Line; Chromosome Deletion; Cyclic AMP; Dexamethasone; Gene Expression Regulation; Gene

1989
Qualitative and quantitative defects of thyroglobulin resulting in congenital goiter. Absence of gross gene deletion of coding sequences in the TG gene structure.
    Journal of endocrinological investigation, 1989, Volume: 12, Issue:11

    Topics: Adolescent; Adult; Blotting, Southern; Child; Child, Preschool; Chromatography, Gel; Chromosome Dele

1989
cis-acting elements of the rat growth hormone gene which mediate basal and regulated expression by thyroid hormone.
    The Journal of biological chemistry, 1987, May-05, Volume: 262, Issue:13

    Topics: Acetyltransferases; Animals; Base Sequence; Chimera; Chloramphenicol O-Acetyltransferase; Chromosome

1987
Thyroid hormone receptor binds to a site in the rat growth hormone promoter required for induction by thyroid hormone.
    Proceedings of the National Academy of Sciences of the United States of America, 1987, Volume: 84, Issue:16

    Topics: Animals; Base Sequence; Binding Sites; Chromosome Deletion; Growth Hormone; Pituitary Gland; Promote

1987
Discrete positive and negative thyroid hormone-responsive transcription regulatory elements of the rat growth hormone gene.
    The Journal of biological chemistry, 1987, Apr-25, Volume: 262, Issue:12

    Topics: Animals; Chimera; Chromosome Deletion; Enhancer Elements, Genetic; Genes; Genes, Regulator; Growth H

1987