triiodothyronine has been researched along with Asthma, Exercise-Induced in 1 studies
Triiodothyronine: A T3 thyroid hormone normally synthesized and secreted by the thyroid gland in much smaller quantities than thyroxine (T4). Most T3 is derived from peripheral monodeiodination of T4 at the 5' position of the outer ring of the iodothyronine nucleus. The hormone finally delivered and used by the tissues is mainly T3.
3,3',5-triiodo-L-thyronine : An iodothyronine compound having iodo substituents at the 3-, 3'- and 5-positions. Although some is produced in the thyroid, most of the 3,3',5-triiodo-L-thyronine in the body is generated by mono-deiodination of L-thyroxine in the peripheral tissues. Its metabolic activity is about 3 to 5 times that of L-thyroxine. The sodium salt is used in the treatment of hypothyroidism.
Asthma, Exercise-Induced: Asthma attacks following a period of exercise. Usually the induced attack is short-lived and regresses spontaneously. The magnitude of postexertional airway obstruction is strongly influenced by the environment in which exercise is performed (i.e. inhalation of cold air during physical exertion markedly augments the severity of the airway obstruction; conversely, warm humid air blunts or abolishes it).
Excerpt | Relevance | Reference |
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"To determine whether thyrotoxicosis has an effect on the asthmatic state in subjects with mild asthma, airway responsiveness, lung function, and exercise capacity were measured in a randomized double-blind placebo-controlled trial before and after liothyronine (triiodothyronine, T3)-induced thyrotoxicosis." | 9.07 | Effect of triiodothyronine-induced thyrotoxicosis on airway hyperresponsiveness. ( Braverman, LE; Dubois, JM; Hollingsworth, HM; Irwin, RS; Pratter, MR, 1991) |
"To determine whether thyrotoxicosis has an effect on the asthmatic state in subjects with mild asthma, airway responsiveness, lung function, and exercise capacity were measured in a randomized double-blind placebo-controlled trial before and after liothyronine (triiodothyronine, T3)-induced thyrotoxicosis." | 5.07 | Effect of triiodothyronine-induced thyrotoxicosis on airway hyperresponsiveness. ( Braverman, LE; Dubois, JM; Hollingsworth, HM; Irwin, RS; Pratter, MR, 1991) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (100.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Hollingsworth, HM | 1 |
Pratter, MR | 1 |
Dubois, JM | 1 |
Braverman, LE | 1 |
Irwin, RS | 1 |
1 trial available for triiodothyronine and Asthma, Exercise-Induced
Article | Year |
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Effect of triiodothyronine-induced thyrotoxicosis on airway hyperresponsiveness.
Topics: Adult; Asthma; Asthma, Exercise-Induced; Exercise; Female; Half-Life; Heart Rate; Humans; Male; Meth | 1991 |