triethyltin has been researched along with Brain-Ischemia* in 2 studies
1 review(s) available for triethyltin and Brain-Ischemia
| Article | Year |
|---|---|
[Pathology of cerebral edema. II. Experimental models and modifying agents].
Current experimental models of brain edema are described and evaluated for their contribution to the knowledge of basic processes involved in its production as well their contribution to the understanding of different clinical forms. The participation of each main pathogenic mechanism in a given experimental model is analyzed and proves to vary with each particular model and site studied. The importance of various experimental models in the evaluation of different therapeutic procedures directed to control the genesis and evolution of brain edema is stressed. Topics: Animals; Brain Edema; Brain Injuries; Brain Ischemia; Brain Neoplasms; Cats; Disease Models, Animal; Freezing; Hypertension; Hypotonic Solutions; Ouabain; Radiation Injuries, Experimental; Rats; Triethyltin Compounds | 1981 |
1 other study(ies) available for triethyltin and Brain-Ischemia
| Article | Year |
|---|---|
Effect of hydration on experimentally induced cerebral edema.
Although fluid restriction is often used to manage cerebral edema, there have been no controlled studies which demonstrate its benefit. We evaluated the effects of dehydration and overhydration on the development of cerebral edema in rats subjected to triethyltin poisoning or anoxic ischemia. Four days after triethyltin poisoning, the brains of control rats receiving maintenance hydration had a mean percentage of water of 79.56%; dehydration (5% of body weight) and overhydration groups were not statistically different at 79.95% and 79.86%, respectively. Forty-seven hours after an anoxic-ischemic insult consisting of unilateral carotid artery ligation and subsequent exposure to a 4% oxygen atmosphere for 30 min, the percentage of water in control rats was 79.12%; dehydration (13% of body weight) and overhydration groups were 79.10% and 79.16%, respectively. Histopathologic analysis of brain sections did not differentiate the hydration groups (triethyltin model only). Thus, cerebral edema was not altered by hydration status in either poisoned or ischemic animals. Topics: Animals; Body Weight; Brain Chemistry; Brain Diseases; Brain Ischemia; Edema; Female; Fluid Therapy; Rats; Rats, Inbred Strains; Triethyltin Compounds; Water | 1985 |