triclosan has been researched along with Breast Neoplasms in 14 studies
Breast Neoplasms: Tumors or cancer of the human BREAST.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Background: Triclosan, bisphenol A (BPA), and brominated flame retardants are environmental estrogenic endocrine-disrupting compounds that may influence the prognosis of breast cancer." | 8.12 | Urinary Concentrations of Triclosan, Bisphenol A, and Brominated Flame Retardants and the Association of Triclosan with Demographic Characteristics and Body Fatness among Women with Newly Diagnosed Breast Cancer. ( Ambrosone, CB; Cheng, TD; Hong, CC; Ilozumba, MN; Shelver, WL, 2022) |
| " In this study, the influences of triclosan, an anti-bacterial agent recently known for an endocrine disrupting chemical (EDC), and kaempferol on breast cancer progression were examined by measuring their effects on epithelial-mesenchymal transition (EMT) and metastatic-related behaviors of MCF-7 breast cancer cells." | 7.85 | Kaempferol, a phytoestrogen, suppressed triclosan-induced epithelial-mesenchymal transition and metastatic-related behaviors of MCF-7 breast cancer cells. ( Choi, KC; Hwang, KA; Lee, GA, 2017) |
| "In the present study, we determined whether two endocrine-disrupting chemicals (EDCs), triclosan (TCS) and octylphenol (OP), are able to alter the expression of two cell cycle regulators, cyclin D1 and p21, in both in vitro and mouse breast cancer models." | 7.80 | Progression of breast cancer cells was enhanced by endocrine-disrupting chemicals, triclosan and octylphenol, via an estrogen receptor-dependent signaling pathway in cellular and mouse xenograft models. ( Choi, KC; Hwang, KA; Kim, HC; Lee, HR; Nam, KH, 2014) |
| "Triclosan inhibits the FAS from goose uropygial glands and human breast cancer SKBr3 cells." | 7.72 | [Inhibition of triclosan to fatty acid synthase from goose uropygial glands and human breast cancer cells in vitro]. ( Anderson, VE; Lai, BS; Wang, YQ, 2003) |
| "Triclosan was able to inhibit the induction of the androgen-responsive LTR-CAT reporter gene in S115 mouse mammary tumour cells by 10(-9) m testosterone and in T47D human breast cancer cells by 10(-8) m testosterone at concentrations of 10(-7) m and 10(-6) m, respectively." | 5.35 | Oestrogenic and androgenic activity of triclosan in breast cancer cells. ( Charles, A; Darbre, PD; Gee, RH; Taylor, N, 2008) |
| "If triclosan is an inhibitor of human fatty acid synthase and if inhibition of fatty acid synthase is toxic to breast cancer cell lines, triclosan could prove to be a lead compound for the treatment of breast cancer." | 5.31 | Triclosan inhibits enoyl-reductase of type I fatty acid synthase in vitro and is cytotoxic to MCF-7 and SKBr-3 breast cancer cells. ( Anderson, VE; Fillgrove, KL; Liu, B; Wang, Y, 2002) |
| "Background: Triclosan, bisphenol A (BPA), and brominated flame retardants are environmental estrogenic endocrine-disrupting compounds that may influence the prognosis of breast cancer." | 4.12 | Urinary Concentrations of Triclosan, Bisphenol A, and Brominated Flame Retardants and the Association of Triclosan with Demographic Characteristics and Body Fatness among Women with Newly Diagnosed Breast Cancer. ( Ambrosone, CB; Cheng, TD; Hong, CC; Ilozumba, MN; Shelver, WL, 2022) |
| " In this study, the influences of triclosan, an anti-bacterial agent recently known for an endocrine disrupting chemical (EDC), and kaempferol on breast cancer progression were examined by measuring their effects on epithelial-mesenchymal transition (EMT) and metastatic-related behaviors of MCF-7 breast cancer cells." | 3.85 | Kaempferol, a phytoestrogen, suppressed triclosan-induced epithelial-mesenchymal transition and metastatic-related behaviors of MCF-7 breast cancer cells. ( Choi, KC; Hwang, KA; Lee, GA, 2017) |
| "In the present study, we determined whether two endocrine-disrupting chemicals (EDCs), triclosan (TCS) and octylphenol (OP), are able to alter the expression of two cell cycle regulators, cyclin D1 and p21, in both in vitro and mouse breast cancer models." | 3.80 | Progression of breast cancer cells was enhanced by endocrine-disrupting chemicals, triclosan and octylphenol, via an estrogen receptor-dependent signaling pathway in cellular and mouse xenograft models. ( Choi, KC; Hwang, KA; Kim, HC; Lee, HR; Nam, KH, 2014) |
| "Triclosan inhibits the FAS from goose uropygial glands and human breast cancer SKBr3 cells." | 3.72 | [Inhibition of triclosan to fatty acid synthase from goose uropygial glands and human breast cancer cells in vitro]. ( Anderson, VE; Lai, BS; Wang, YQ, 2003) |
| "MCF-7 BOS human breast cancer cells were used to investigate the potential for cytotoxicity, estrogenicity and anti-estrogenicity of these three compounds at environmentally relevant concentrations using the 3-(4,5-dimethylthiazol-2-yl)-5-(3-carboxymethoxyphenyl)-2-(4-sulfophenyl)-2H-tetrazolium, inner salt assay (MTS) and the E-SCREEN bioassay." | 1.39 | Comparison of in vitro cytotoxicity, estrogenicity and anti-estrogenicity of triclosan, perfluorooctane sulfonate and perfluorooctanoic acid. ( Fair, PA; Henry, ND, 2013) |
| "Triclosan was dissolved in five different solvents viz." | 1.36 | Evaluation of suitable solvents for testing the anti-proliferative activity of triclosan - a hydrophobic drug in cell culture. ( Aparna, G; Deepa, PR; Jayanthi, U; Krishnakumar, S; Vandhana, S, 2010) |
| "Triclosan was able to inhibit the induction of the androgen-responsive LTR-CAT reporter gene in S115 mouse mammary tumour cells by 10(-9) m testosterone and in T47D human breast cancer cells by 10(-8) m testosterone at concentrations of 10(-7) m and 10(-6) m, respectively." | 1.35 | Oestrogenic and androgenic activity of triclosan in breast cancer cells. ( Charles, A; Darbre, PD; Gee, RH; Taylor, N, 2008) |
| "If triclosan is an inhibitor of human fatty acid synthase and if inhibition of fatty acid synthase is toxic to breast cancer cell lines, triclosan could prove to be a lead compound for the treatment of breast cancer." | 1.31 | Triclosan inhibits enoyl-reductase of type I fatty acid synthase in vitro and is cytotoxic to MCF-7 and SKBr-3 breast cancer cells. ( Anderson, VE; Fillgrove, KL; Liu, B; Wang, Y, 2002) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (28.57) | 29.6817 |
| 2010's | 7 (50.00) | 24.3611 |
| 2020's | 3 (21.43) | 2.80 |
| Authors | Studies |
|---|---|
| Ilozumba, MN | 1 |
| Shelver, WL | 1 |
| Hong, CC | 1 |
| Ambrosone, CB | 1 |
| Cheng, TD | 1 |
| Cai, X | 1 |
| Ning, C | 1 |
| Fan, L | 1 |
| Li, Y | 1 |
| Wang, L | 1 |
| He, H | 1 |
| Dong, T | 1 |
| Cai, Y | 1 |
| Zhang, M | 1 |
| Lu, Z | 1 |
| Chen, C | 1 |
| Shi, K | 1 |
| Ye, T | 1 |
| Zhong, R | 1 |
| Tian, J | 1 |
| Li, H | 2 |
| Zhu, Y | 1 |
| Miao, X | 1 |
| Chen, J | 1 |
| Xie, P | 1 |
| Wu, P | 1 |
| Lin, Z | 1 |
| He, Y | 1 |
| Cai, Z | 1 |
| Lee, HR | 1 |
| Hwang, KA | 3 |
| Nam, KH | 1 |
| Kim, HC | 1 |
| Choi, KC | 3 |
| Kim, SH | 1 |
| Lee, GA | 1 |
| Vandhana, S | 1 |
| Deepa, PR | 1 |
| Aparna, G | 1 |
| Jayanthi, U | 1 |
| Krishnakumar, S | 1 |
| Witorsch, RJ | 1 |
| Thomas, JA | 1 |
| Henry, ND | 1 |
| Fair, PA | 1 |
| Williams, N | 1 |
| Sweetland, H | 1 |
| Goyal, S | 1 |
| Ivins, N | 1 |
| Leaper, DJ | 1 |
| Wang, YQ | 1 |
| Lai, BS | 1 |
| Anderson, VE | 2 |
| Darbre, PD | 2 |
| Gee, RH | 1 |
| Charles, A | 1 |
| Taylor, N | 1 |
| Liu, B | 1 |
| Wang, Y | 1 |
| Fillgrove, KL | 1 |
2 reviews available for triclosan and Breast Neoplasms
| Article | Year |
|---|---|
Personal care products and endocrine disruption: A critical review of the literature.
Topics: Animals; Breast Neoplasms; Carbanilides; Carcinoma, Ductal, Breast; Cosmetics; Endocrine Disruptors; | 2010 |
Environmental oestrogens, cosmetics and breast cancer.
Topics: Aluminum Compounds; Animals; Aromatase Inhibitors; Breast Neoplasms; Cosmetics; Environmental Pollut | 2006 |
1 trial available for triclosan and Breast Neoplasms
| Article | Year |
|---|---|
Randomized trial of antimicrobial-coated sutures to prevent surgical site infection after breast cancer surgery.
Topics: Adult; Aged; Aged, 80 and over; Anti-Infective Agents, Local; Breast Neoplasms; Female; Humans; Midd | 2011 |
11 other studies available for triclosan and Breast Neoplasms
| Article | Year |
|---|---|
Urinary Concentrations of Triclosan, Bisphenol A, and Brominated Flame Retardants and the Association of Triclosan with Demographic Characteristics and Body Fatness among Women with Newly Diagnosed Breast Cancer.
Topics: Benzhydryl Compounds; Breast Neoplasms; Creatinine; Demography; Female; Flame Retardants; Humans; Ma | 2022 |
Triclosan is associated with breast cancer via oxidative stress and relative telomere length.
Topics: 8-Hydroxy-2'-Deoxyguanosine; Breast Neoplasms; Case-Control Studies; Female; Humans; Leukocytes, Mon | 2023 |
Spatial Metabolomics and Lipidomics Reveal the Mechanisms of the Enhanced Growth of Breast Cancer Cell Spheroids Exposed to Triclosan.
Topics: Breast Neoplasms; Female; Humans; Lipidomics; Metabolome; Metabolomics; Spectrometry, Mass, Matrix-A | 2023 |
Progression of breast cancer cells was enhanced by endocrine-disrupting chemicals, triclosan and octylphenol, via an estrogen receptor-dependent signaling pathway in cellular and mouse xenograft models.
Topics: Animals; Breast; Breast Neoplasms; Endocrine Disruptors; Female; Humans; MCF-7 Cells; Mice; Mice, In | 2014 |
Treatment with kaempferol suppresses breast cancer cell growth caused by estrogen and triclosan in cellular and xenograft breast cancer models.
Topics: Animals; Breast Neoplasms; Cell Proliferation; Estrogens; Female; Heterografts; Humans; Kaempferols; | 2016 |
Kaempferol, a phytoestrogen, suppressed triclosan-induced epithelial-mesenchymal transition and metastatic-related behaviors of MCF-7 breast cancer cells.
Topics: Anti-Infective Agents, Local; Antineoplastic Agents; Breast Neoplasms; Cell Movement; Epithelial-Mes | 2017 |
Evaluation of suitable solvents for testing the anti-proliferative activity of triclosan - a hydrophobic drug in cell culture.
Topics: Antineoplastic Agents; Breast Neoplasms; Cell Line, Tumor; Cell Proliferation; Drug Screening Assays | 2010 |
Comparison of in vitro cytotoxicity, estrogenicity and anti-estrogenicity of triclosan, perfluorooctane sulfonate and perfluorooctanoic acid.
Topics: Alkanesulfonic Acids; Breast Neoplasms; Caprylates; Cell Line, Tumor; Cell Proliferation; Cell Survi | 2013 |
[Inhibition of triclosan to fatty acid synthase from goose uropygial glands and human breast cancer cells in vitro].
Topics: Animals; Breast Neoplasms; Enzyme Inhibitors; Fatty Acid Synthases; Female; Geese; Humans; Triclosan | 2003 |
Oestrogenic and androgenic activity of triclosan in breast cancer cells.
Topics: Androgens; Anti-Infective Agents, Local; Binding, Competitive; Breast Neoplasms; Cell Line, Tumor; C | 2008 |
Triclosan inhibits enoyl-reductase of type I fatty acid synthase in vitro and is cytotoxic to MCF-7 and SKBr-3 breast cancer cells.
Topics: Antineoplastic Agents; Breast Neoplasms; Cell Survival; Enoyl-(Acyl-Carrier-Protein) Reductase (NADH | 2002 |