tributyrin has been researched along with Carcinogenesis in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Andrade, FO; Farsky, SHP; Fernandes, LHG; Furtado, KS; Hebeda, CB; Heidor, R; Horst, MA; Miranda, MLP; Moreno, FS; Sandri, S; Yamamoto, RC | 1 |
Beland, FA; de Conti, A; Heidor, R; Koturbash, I; Kuroiwa-Trzmielina, J; Moreno, FS; Ong, TP; Pogribny, IP; Tryndyak, V | 1 |
Beland, FA; Campos, A; Carrilho, J; de Conti, A; Furtado, KS; Fuscoe, JC; Guariento, AH; Han, T; Moreno, FS; Pogribny, IP; Purgatto, E; Ross, SA; Shinohara, EM; Tryndyak, V | 1 |
3 other study(ies) available for tributyrin and Carcinogenesis
Article | Year |
---|---|
Antiangiogenic effects of the chemopreventive agent tributyrin, a butyric acid prodrug, during the promotion phase of hepatocarcinogenesis.
Topics: Actins; Angiogenesis Inhibitors; Animals; Apoptosis; Butyric Acid; Carcinogenesis; Carcinoma, Hepatocellular; Cell Movement; Gene Expression Regulation, Neoplastic; Hepatocytes; Human Umbilical Vein Endothelial Cells; Humans; Hypoxia-Inducible Factor 1, alpha Subunit; Liver; Liver Neoplasms; Liver Neoplasms, Experimental; Polysaccharides; Prodrugs; Rats; Triglycerides; Vascular Endothelial Growth Factor A; Vascular Endothelial Growth Factor Receptor-2 | 2019 |
The chemopreventive activity of the butyric acid prodrug tributyrin in experimental rat hepatocarcinogenesis is associated with p53 acetylation and activation of the p53 apoptotic signaling pathway.
Topics: Acetylation; Animals; Apoptosis; Butyric Acid; Carcinogenesis; Hepatocytes; Histone Deacetylase Inhibitors; Histone Deacetylases; Histones; Liver Neoplasms, Experimental; Lysine; Male; Precancerous Conditions; Prodrugs; Random Allocation; Rats; Rats, Wistar; Signal Transduction; Triglycerides; Tumor Suppressor Protein p53; Up-Regulation | 2013 |
Transcriptomic responses provide a new mechanistic basis for the chemopreventive effects of folic acid and tributyrin in rat liver carcinogenesis.
Topics: Animals; Antigens, CD34; Antineoplastic Combined Chemotherapy Protocols; Carcinogenesis; Carcinoma, Hepatocellular; Diethylnitrosamine; Folic Acid; Gene Expression Regulation, Neoplastic; Glutathione S-Transferase pi; Humans; Liver Neoplasms; Male; Neoplasm Proteins; Neovascularization, Pathologic; Rats; Transcriptome; Triglycerides | 2014 |