triazoles has been researched along with Left Ventricular Hypertrophy in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Fukui, K; Nangaku, M; Saito, H; Sugahara, M; Tanaka, T; Uchida, L; Wakashima, T | 1 |
| Belardinelli, L; Chi, L; Dhalla, AK; Hirakawa, R; Ling, H; Rajamani, S; Zeng, A | 1 |
| Birnbaum, Y; Keyes, KT; Lin, Y; Perez-Polo, JR; Ye, Y; Zhang, C | 1 |
3 other study(ies) available for triazoles and Left Ventricular Hypertrophy
| Article | Year |
|---|---|
Effects of a prolyl hydroxylase inhibitor on kidney and cardiovascular complications in a rat model of chronic kidney disease.
Topics: Animals; Apoptosis; Cell Line; Cytokines; Disease Models, Animal; Fibrosis; Hypertrophy, Left Ventricular; Hypoxia-Inducible Factor 1, alpha Subunit; Inflammation Mediators; Kidney; Male; Mitochondria, Heart; Myocytes, Cardiac; N-substituted Glycines; Neovascularization, Physiologic; Prolyl Hydroxylases; Prolyl-Hydroxylase Inhibitors; Pyridines; Rats, Sprague-Dawley; Renal Insufficiency, Chronic; Signal Transduction; Triazoles; Ventricular Function, Left; Ventricular Remodeling | 2020 |
Inhibition of late Na+ current, a novel target to improve diastolic function and electrical abnormalities in Dahl salt-sensitive rats.
Topics: Action Potentials; Animals; Anti-Arrhythmia Agents; Arrhythmias, Cardiac; Disease Models, Animal; Dose-Response Relationship, Drug; Heart Conduction System; Heart Failure; Heart Rate; Hypertrophy, Left Ventricular; Male; Myocardial Contraction; Myocytes, Cardiac; Oxidative Stress; Pyridines; Rats, Inbred Dahl; Sodium Channel Blockers; Sodium Channels; Sodium Chloride, Dietary; Time Factors; Triazoles; Ventricular Dysfunction, Left; Ventricular Function, Left | 2016 |
The myocardial infarct size-limiting effect of sitagliptin is PKA-dependent, whereas the protective effect of pioglitazone is partially dependent on PKA.
Topics: Animals; Blood Glucose; Blotting, Western; Body Weight; Culture Media; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Dipeptidyl-Peptidase IV Inhibitors; Eicosanoids; Glucagon-Like Peptide 1; Hypertrophy, Left Ventricular; Hypoglycemic Agents; In Vitro Techniques; Mice; Myocardial Infarction; Myocardium; Myocytes, Cardiac; Organ Size; Phospholipases A2; Pioglitazone; Prostaglandin-Endoperoxide Synthases; Protective Agents; Pyrazines; Sitagliptin Phosphate; Thiazolidinediones; Triazoles | 2010 |