triazoles has been researched along with Diabetic Cardiomyopathies in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 2 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Carr, CA; Castro-Guarda, M; Dennis, KMJH; Heather, LC; Massourides, E; Montes Aparicio, CN; Purnama, U; Sousa Fialho, MDL; Tyler, DJ | 1 |
| Mu, J; Tian, Y; Xie, Z; Zhang, D; Zou, MH | 1 |
| Chen, WJ; Guo, M; Wang, HX | 1 |
| Ares-Carrasco, S; Caro-Vadillo, A; Egido, J; Iborra, C; Lorenzo, O; Picatoste, B; Ramírez, E; Tuñón, J | 1 |
4 other study(ies) available for triazoles and Diabetic Cardiomyopathies
| Article | Year |
|---|---|
Activation of HIF1α Rescues the Hypoxic Response and Reverses Metabolic Dysfunction in the Diabetic Heart.
Topics: Adaptation, Physiological; Anemia, Sickle Cell; Animals; Cell Line; Diabetes Mellitus, Experimental; Diabetes Mellitus, Type 2; Diabetic Cardiomyopathies; Energy Metabolism; Gene Expression Regulation; Humans; Hypoxia-Inducible Factor 1, alpha Subunit; Insulin Resistance; Myocytes, Cardiac; Oxygen; Pluripotent Stem Cells; Pyrazoles; Rats; Triazoles | 2021 |
BRD4 inhibition by JQ1 prevents high-fat diet-induced diabetic cardiomyopathy by activating PINK1/Parkin-mediated mitophagy in vivo.
Topics: Animals; Animals, Newborn; Azepines; Diabetes Mellitus, Type 2; Diabetic Cardiomyopathies; Diet, High-Fat; Gene Deletion; Mice, Inbred C57BL; Mitochondria, Heart; Mitophagy; Nuclear Proteins; Promoter Regions, Genetic; Protein Kinases; Transcription Factors; Triazoles; Ubiquitin-Protein Ligases; Up-Regulation | 2020 |
BET-inhibition by JQ1 alleviates streptozotocin-induced diabetic cardiomyopathy.
Topics: Animals; Azepines; Caveolin 1; Cell Differentiation; Cell Movement; Cell Proliferation; Collagen; Cytoprotection; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Fibrosis; Male; Mice; Myocytes, Cardiac; Myofibroblasts; Proteins; Signal Transduction; Streptozocin; Transforming Growth Factor beta1; Triazoles; Ventricular Remodeling | 2018 |
Sitagliptin reduces cardiac apoptosis, hypertrophy and fibrosis primarily by insulin-dependent mechanisms in experimental type-II diabetes. Potential roles of GLP-1 isoforms.
Topics: Animals; Apoptosis; Cardiomegaly; Cardiotonic Agents; Cells, Cultured; Diabetes Mellitus, Type 2; Diabetic Cardiomyopathies; Disease Models, Animal; Drug Evaluation, Preclinical; Fibroblasts; Fibronectins; Fibrosis; Glucagon-Like Peptide 1; Glucose Intolerance; Hypoglycemic Agents; Insulin; Male; Metformin; Myocardium; Myocytes, Cardiac; PPAR delta; Protein Isoforms; Pyrazines; Rats; Sitagliptin Phosphate; Triazoles | 2013 |