trehalose has been researched along with Genetic Predisposition in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Biczo, G; Dawson, D; Elperin, J; French, SW; Gorelick, FS; Gretler, S; Gukovskaya, AS; Gukovsky, I; Hegyi, P; Husain, SZ; Lotshaw, E; Lugea, A; Malla, SR; Mareninova, OA; Rakonczay, Z; Ruchala, P; Shalbueva, N; Vegh, ET; Wen, L; Whitelegge, J | 1 |
| Chang, MY; Chen, YC; Cheng, YL; Chou, LF; Hsieh, CY; Hung, CC; Lin, CY; Tian, YC; Yang, CW; Yang, HY | 1 |
| Ren, J; Wang, Q | 1 |
3 other study(ies) available for trehalose and Genetic Predisposition
| Article | Year |
|---|---|
Mitochondrial Dysfunction, Through Impaired Autophagy, Leads to Endoplasmic Reticulum Stress, Deregulated Lipid Metabolism, and Pancreatitis in Animal Models.
Topics: Acute Disease; Animals; Arginine; Autophagy; Bile Acids and Salts; Calcium Signaling; Ceruletide; Choline Deficiency; Cyclophilins; Disease Models, Animal; Endoplasmic Reticulum Stress; Ethionine; Genetic Predisposition to Disease; Humans; Lipid Metabolism; Membrane Potential, Mitochondrial; Mice, Inbred C57BL; Mice, Knockout; Mitochondria; Mitochondrial Proton-Translocating ATPases; Pancreas; Pancreatitis; Peptidyl-Prolyl Isomerase F; Phenotype; Rats; Time Factors; Trehalose | 2018 |
Effect of Trehalose Supplementation on Autophagy and Cystogenesis in a Mouse Model of Polycystic Kidney Disease.
Topics: Animals; Autophagy; Blood Glucose; Genetic Predisposition to Disease; Kidney; Mice; Mice, Transgenic; MicroRNAs; Polycystic Kidney, Autosomal Dominant; Trehalose | 2018 |
mTOR-Independent autophagy inducer trehalose rescues against insulin resistance-induced myocardial contractile anomalies: Role of p38 MAPK and Foxo1.
Topics: Animals; Apoptosis; Autophagy; Calcium Signaling; Cardiotonic Agents; Diabetic Cardiomyopathies; Disease Models, Animal; Enzyme Activation; Forkhead Box Protein O1; Genetic Predisposition to Disease; Insulin Resistance; Mice, Knockout; Myocardial Contraction; Myocardium; p38 Mitogen-Activated Protein Kinases; Phenotype; Phosphorylation; Proto-Oncogene Proteins c-akt; Time Factors; TOR Serine-Threonine Kinases; Trehalose; Ventricular Function, Left | 2016 |