Page last updated: 2024-08-23

transferrin and Huntington Disease

transferrin has been researched along with Huntington Disease in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19901 (25.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's1 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bush, AI; Chen, J; Duce, JA; Fox, JH; Hersch, S; Lai, B; Lam, LQ; Marks, E; Volitakis, I; Zhang, Z1
Aronin, N; Cuiffo, B; DiFiglia, M; Hayden, MR; Kegel, KB; Qin, ZH; Sapp, E; Wang, Y; Wanker, E1
Ivanova-Smolenskaia, IA; Korshunova, TS; Mzhel'skaia, TI1
Cattaneo, E; Hilditch-Maguire, P; MacDonald, ME; Persichetti, F; Rigamonti, D; Sharp, AH; Trettel, F; Wheeler, VC1

Other Studies

4 other study(ies) available for transferrin and Huntington Disease

ArticleYear
Iron accumulates in Huntington's disease neurons: protection by deferoxamine.
    PloS one, 2013, Volume: 8, Issue:10

    Topics: Animals; Corpus Striatum; Deferoxamine; Disease Models, Animal; Female; Huntington Disease; Injections, Intraventricular; Iron; Iron Regulatory Protein 1; Iron Regulatory Protein 2; Male; Mice; Neurons; Receptors, Transferrin; Transferrin

2013
Huntingtin bodies sequester vesicle-associated proteins by a polyproline-dependent interaction.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2004, Jan-07, Volume: 24, Issue:1

    Topics: Animals; Cell Line; Cerebral Cortex; Dynamins; Endopeptidases; Epitopes; Humans; Huntingtin Protein; Huntington Disease; Immunohistochemistry; Mice; Nerve Tissue Proteins; Neurons; Nuclear Proteins; Peptides; Phagosomes; Protein Structure, Tertiary; Transferrin; Vesicular Transport Proteins

2004
[Metabolic disorders in essential tremor].
    Zhurnal nevropatologii i psikhiatrii imeni S.S. Korsakova (Moscow, Russia : 1952), 1980, Volume: 80, Issue:12

    Topics: Adolescent; Adult; Age Factors; Aged; Bilirubin; Blood Glucose; Blood Proteins; Ceruloplasmin; Cholesterol; Copper; Glucose Tolerance Test; Hepatolenticular Degeneration; Humans; Huntington Disease; Iron; Middle Aged; Pantothenate Kinase-Associated Neurodegeneration; Transaminases; Transferrin; Tremor

1980
Dominant phenotypes produced by the HD mutation in STHdh(Q111) striatal cells.
    Human molecular genetics, 2000, Nov-22, Volume: 9, Issue:19

    Topics: Animals; Cell Differentiation; Cell Nucleus; Cells, Cultured; Clone Cells; Corpus Striatum; Cytoplasm; Endoplasmic Reticulum; Genes, Dominant; Golgi Apparatus; Humans; Huntingtin Protein; Huntington Disease; Iron; Mice; Mice, Transgenic; Mutation; Nerve Tissue Proteins; Nuclear Proteins; Oxygen; Receptors, Transferrin; Stem Cells; Transferrin; Tumor Suppressor Protein p53

2000