Page last updated: 2024-08-23

torpedo and Genetic Predisposition

torpedo has been researched along with Genetic Predisposition in 10 studies

Research

Studies (10)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (20.00)18.2507
2000's8 (80.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Changeux, JP1
Conti-Fine, BM; Milani, M; Okita, D; Ostlie, N; Wang, W1
Garcia, YR; Krolick, KA; Pothitakis, JC1
Aiki, Y; Anazawa, Y; Kono, Y; Kozaki, T; Tomita, T1
Christadoss, P; Goluszko, E; Higgs, S; Scott, BG; Tüzün, E1
Conti-Fine, BM; Milani, M; Ostlie, NS; Wang, W1
Atassi, MZ; Christadoss, P; Oshima, M; Shenoy, M1
Bernard, I; Cautain, B; De Baets, M; Druet, P; Guéry, JC; Hoedemaekers, A; Martinez, K; Saoudi, A1
Conti-Fine, BM; Karachunski, PI; Monfardini, C; Ostlie, NS1
Conti-Fine, BM; Karachunski, PI; Kronenberg, M; Monfardini, C; Ostlie, NS; Wang, W1

Reviews

1 review(s) available for torpedo and Genetic Predisposition

ArticleYear
Nicotinic receptors and nicotine addiction.
    Comptes rendus biologies, 2009, Volume: 332, Issue:5

    Topics: Acetylcholine; Adolescent; Adult; Allosteric Regulation; Animals; Brain Chemistry; Genetic Predisposition to Disease; Humans; Ion Channels; Lung Neoplasms; Mice; Nicotine; Protein Subunits; Receptors, Nicotinic; Reward; Smoking; Synaptic Transmission; Tobacco Use Disorder; Torpedo; Ventral Tegmental Area

2009

Other Studies

9 other study(ies) available for torpedo and Genetic Predisposition

ArticleYear
Absence of IL-4 facilitates the development of chronic autoimmune myasthenia gravis in C57BL/6 mice.
    Journal of immunology (Baltimore, Md. : 1950), 2003, Jan-01, Volume: 170, Issue:1

    Topics: Amino Acid Sequence; Animals; Autoantibodies; B7-1 Antigen; CD4-Positive T-Lymphocytes; CD40 Antigens; Chronic Disease; Epitopes, T-Lymphocyte; Genetic Predisposition to Disease; Immunization; Injections, Subcutaneous; Interferon-gamma; Interleukin-4; Lymphocyte Activation; Mice; Mice, Inbred C57BL; Mice, Knockout; Myasthenia Gravis, Autoimmune, Experimental; Peptide Fragments; Receptors, Cholinergic; Severity of Illness Index; Spleen; Torpedo

2003
Myocyte production of nitric oxide in response to AChR-reactive antibodies in two inbred rat strains may influence disease outcome in experimental myasthenia gravis.
    Clinical immunology (Orlando, Fla.), 2003, Volume: 106, Issue:2

    Topics: Animals; Antibody Specificity; Autoantibodies; Cells, Cultured; Clone Cells; Enzyme Induction; Enzyme Inhibitors; Female; Genetic Predisposition to Disease; Immunization, Passive; Lysine; Muscle Proteins; Muscle, Skeletal; Myasthenia Gravis, Autoimmune, Experimental; Nitric Oxide; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; Rats; Rats, Inbred Lew; Rats, Inbred WF; Receptors, Cholinergic; RNA, Messenger; Torpedo

2003
Sequence of a cDNA encoding acetylcholinesterase from susceptible and resistant two-spotted spider mite, Tetranychus urticae.
    Insect biochemistry and molecular biology, 2003, Volume: 33, Issue:5

    Topics: Acetylcholinesterase; Amino Acid Sequence; Animals; Base Sequence; DNA, Complementary; Enzyme Precursors; Genetic Predisposition to Disease; Insecticide Resistance; Insecticides; Mites; Molecular Sequence Data; Organophosphorus Compounds; Sequence Alignment; Sequence Homology, Amino Acid; Torpedo

2003
Genetic evidence for involvement of classical complement pathway in induction of experimental autoimmune myasthenia gravis.
    Journal of immunology (Baltimore, Md. : 1950), 2003, Oct-01, Volume: 171, Issue:7

    Topics: Animals; Apoptosis; B-Lymphocyte Subsets; Complement C3; Complement C4; Complement Membrane Attack Complex; Complement Pathway, Classical; Cytokines; Genetic Carrier Screening; Genetic Predisposition to Disease; Immunity, Innate; Lymph Nodes; Lymphocytosis; Lymphopenia; Mice; Mice, Inbred C57BL; Mice, Knockout; Myasthenia Gravis, Autoimmune, Experimental; Neuromuscular Junction; Receptors, Cholinergic; T-Lymphocyte Subsets; Torpedo

2003
The susceptibility to experimental myasthenia gravis of STAT6-/- and STAT4-/- BALB/c mice suggests a pathogenic role of Th1 cells.
    Journal of immunology (Baltimore, Md. : 1950), 2004, Jan-01, Volume: 172, Issue:1

    Topics: Animals; Autoantibodies; CD4-Positive T-Lymphocytes; Cell Division; DNA-Binding Proteins; Female; Genetic Predisposition to Disease; Immunoglobulin G; Interferon-gamma; Interleukin-4; Mice; Mice, Inbred BALB C; Mice, Knockout; Myasthenia Gravis, Autoimmune, Experimental; Peptide Fragments; Protein Subunits; Receptors, Nicotinic; Signal Transduction; STAT4 Transcription Factor; STAT6 Transcription Factor; Th1 Cells; Torpedo; Trans-Activators

2004
Suppression of experimental autoimmune myasthenia gravis by epitope-specific neonatal tolerance.
    Advances in experimental medicine and biology, 1994, Volume: 347

    Topics: Amino Acid Sequence; Animals; Animals, Newborn; Autoantibodies; Autoimmune Diseases; Disease Susceptibility; Female; Gene Conversion; Genetic Predisposition to Disease; Histocompatibility Antigens Class II; Humans; Immune Tolerance; Immunization; Immunodominant Epitopes; Immunosuppression Therapy; Lymphocyte Activation; Mice; Mice, Inbred C57BL; Molecular Sequence Data; Myasthenia Gravis; Peptide Fragments; Receptors, Nicotinic; Single-Blind Method; Species Specificity; T-Lymphocytes, Helper-Inducer; Torpedo

1994
Experimental autoimmune myasthenia gravis may occur in the context of a polarized Th1- or Th2-type immune response in rats.
    Journal of immunology (Baltimore, Md. : 1950), 1999, Jun-15, Volume: 162, Issue:12

    Topics: Animals; Antibodies, Monoclonal; Female; Genetic Predisposition to Disease; Immunophenotyping; Incidence; Injections, Intraperitoneal; Interferon-gamma; Lymphocyte Activation; Male; Myasthenia Gravis; Rats; Rats, Inbred BN; Rats, Inbred Lew; Receptors, Cholinergic; Th1 Cells; Th2 Cells; Torpedo

1999
Absence of IFN-gamma or IL-12 has different effects on experimental myasthenia gravis in C57BL/6 mice.
    Journal of immunology (Baltimore, Md. : 1950), 2000, May-15, Volume: 164, Issue:10

    Topics: Animals; Autoantibodies; Binding Sites, Antibody; CD4-Positive T-Lymphocytes; CD8-Positive T-Lymphocytes; Complement System Proteins; Cytokines; Epitopes, T-Lymphocyte; Genetic Predisposition to Disease; Immunity, Innate; Immunoglobulin G; Interferon-gamma; Interleukin-12; Lymphocyte Depletion; Mice; Mice, Inbred C57BL; Mice, Knockout; Myasthenia Gravis; Neuromuscular Junction; Receptors, Cholinergic; Spleen; Th1 Cells; Torpedo

2000
Transgenic expression of IL-10 in T cells facilitates development of experimental myasthenia gravis.
    Journal of immunology (Baltimore, Md. : 1950), 2001, Apr-15, Volume: 166, Issue:8

    Topics: Animals; Autoantibodies; CD4-Positive T-Lymphocytes; Cells, Cultured; Complement System Proteins; Cytokines; Epitopes, T-Lymphocyte; Female; Genetic Predisposition to Disease; Immunoglobulin G; Injections, Subcutaneous; Interleukin-10; Male; Mice; Mice, Inbred C3H; Mice, Inbred C57BL; Mice, Transgenic; Myasthenia Gravis; Neuromuscular Junction; Receptors, Cholinergic; T-Lymphocytes; Torpedo

2001