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thyroxine and Amyloid Neuropathies

thyroxine has been researched along with Amyloid Neuropathies in 3 studies

Thyroxine: The major hormone derived from the thyroid gland. Thyroxine is synthesized via the iodination of tyrosines (MONOIODOTYROSINE) and the coupling of iodotyrosines (DIIODOTYROSINE) in the THYROGLOBULIN. Thyroxine is released from thyroglobulin by proteolysis and secreted into the blood. Thyroxine is peripherally deiodinated to form TRIIODOTHYRONINE which exerts a broad spectrum of stimulatory effects on cell metabolism.
thyroxine : An iodothyronine compound having iodo substituents at the 3-, 3'-, 5- and 5'-positions.

Amyloid Neuropathies: Disorders of the peripheral nervous system associated with the deposition of AMYLOID in nerve tissue. Familial, primary (nonfamilial), and secondary forms have been described. Some familial subtypes demonstrate an autosomal dominant pattern of inheritance. Clinical manifestations include sensory loss, mild weakness, autonomic dysfunction, and CARPAL TUNNEL SYNDROME. (Adams et al., Principles of Neurology, 6th ed, p1349)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's2 (66.67)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Almeida, MR2
Macedo, B1
Cardoso, I1
Alves, I1
Valencia, G1
Arsequell, G1
Planas, A1
Saraiva, MJ3
Kohno, K1
Palha, JA1
Miyakawa, K1
Ito, S1
Mabuchi, T1
Blaner, WS1
Iijima, H1
Tsukahara, S1
Episkopou, V1
Gottesman, ME1
Shimada, K1
Takahashi, K1
Yamamura, K1
Maeda, S1
Alves, IL1
Terazaki, H1
Ando, Y1

Other Studies

3 other studies available for thyroxine and Amyloid Neuropathies

ArticleYear
Selective binding to transthyretin and tetramer stabilization in serum from patients with familial amyloidotic polyneuropathy by an iodinated diflunisal derivative.
    The Biochemical journal, 2004, Jul-15, Volume: 381, Issue:Pt 2

    Topics: Amyloid Neuropathies; Diclofenac; Diflunisal; Flufenamic Acid; Humans; Iodine; Iodobenzoates; Molecu

2004
Analysis of amyloid deposition in a transgenic mouse model of homozygous familial amyloidotic polyneuropathy.
    The American journal of pathology, 1997, Volume: 150, Issue:4

    Topics: Amyloid; Amyloid Neuropathies; Animals; Blood Proteins; Disease Models, Animal; Female; Homozygote;

1997
Comparative studies of two transthyretin variants with protective effects on familial amyloidotic polyneuropathy: TTR R104H and TTR T119M.
    Biochemical and biophysical research communications, 2000, Apr-21, Volume: 270, Issue:3

    Topics: Amino Acid Substitution; Amyloid Neuropathies; Disease Progression; Genetic Variation; Heterozygote;

2000