thymidine has been researched along with Hyperparathyroidism, Secondary in 2 studies
Hyperparathyroidism, Secondary: Abnormally elevated PARATHYROID HORMONE secretion as a response to HYPOCALCEMIA. It is caused by chronic KIDNEY FAILURE or other abnormalities in the controls of bone and mineral metabolism, leading to various BONE DISEASES, such as RENAL OSTEODYSTROPHY.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (50.00) | 18.2507 |
2000's | 1 (50.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Nakajima, K | 1 |
Umino, K | 1 |
Azuma, Y | 1 |
Kosaka, S | 1 |
Takano, K | 1 |
Obara, T | 1 |
Sato, K | 1 |
Alexiewicz, JM | 1 |
Klinger, M | 1 |
Pitts, TO | 1 |
Gaciong, Z | 1 |
Linker-Israeli, M | 1 |
Massry, SG | 1 |
2 other studies available for thymidine and Hyperparathyroidism, Secondary
Article | Year |
---|---|
Stimulating parathyroid cell proliferation and PTH release with phosphate in organ cultures obtained from patients with primary and secondary hyperparathyroidism for a prolonged period.
Topics: Calcium; Cell Proliferation; Gene Expression Regulation; Humans; Hyperparathyroidism, Primary; Hyper | 2009 |
Parathyroid hormone inhibits B cell proliferation: implications in chronic renal failure.
Topics: Adult; Aged; B-Lymphocytes; Cell Division; Colforsin; Cyclic AMP; Female; Humans; Hyperparathyroidis | 1990 |