thromboxanes has been researched along with ACD-MPV in 5 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (40.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (60.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Dakshinamurti, S; Fediuk, J; Nolette, N; Sikarwar, AS | 1 |
| Dakshinamurti, S; Fediuk, J; Hinton, M; Lizotte, PP; Nolette, N; Sikarwar, AS | 1 |
| Dakshinamurti, S; Fediuk, J; Gutsol, A; Nolette, N | 1 |
| Lyrene, RK; Philips, JB | 1 |
| McKenney, D | 1 |
1 review(s) available for thromboxanes and ACD-MPV
| Article | Year |
|---|---|
Prostaglandins, related compounds, and the perinatal pulmonary circulation.
Topics: Animals; Animals, Newborn; Arachidonic Acids; Blood Pressure; Epoprostenol; Female; Fetus; Humans; Hydroxyeicosatetraenoic Acids; Hypertension, Pulmonary; Infant, Newborn; Lung; Persistent Fetal Circulation Syndrome; Pregnancy; Prostaglandin Antagonists; Prostaglandin D2; Prostaglandins; Prostaglandins D; Prostaglandins E; Prostaglandins F; Pulmonary Circulation; Sheep; SRS-A; Thromboxanes; Vascular Resistance; Vasoconstriction | 1984 |
4 other study(ies) available for thromboxanes and ACD-MPV
| Article | Year |
|---|---|
Thromboxane-induced actin polymerization in hypoxic neonatal pulmonary arterial myocytes involves Cdc42 signaling.
Topics: 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid; Actins; Aminoquinolines; Animals; cdc42 GTP-Binding Protein; Cell Hypoxia; Cells, Cultured; Cytochalasin D; Muscle Contraction; Myocytes, Smooth Muscle; Nucleic Acid Synthesis Inhibitors; p21-Activated Kinases; Persistent Fetal Circulation Syndrome; Phosphatidylinositol 3-Kinase; Protein Isoforms; Proto-Oncogene Proteins c-akt; Pulmonary Artery; Pyrimidines; rhoA GTP-Binding Protein; Signal Transduction; Swine; Thromboxanes; Vasoconstrictor Agents; Wiskott-Aldrich Syndrome Protein, Neuronal | 2014 |
Hypoxia increases pulmonary arterial thromboxane receptor internalization independent of receptor sensitization.
Topics: Actins; Animals; Cell Hypoxia; Epoprostenol; Humans; Infant, Newborn; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Persistent Fetal Circulation Syndrome; Pulmonary Artery; Receptors, Thromboxane; Signal Transduction; Swine; Thromboxanes | 2015 |
Thromboxane-induced actin polymerization in hypoxic pulmonary artery is independent of Rho.
Topics: 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid; Actin Cytoskeleton; Actins; Animals; Animals, Newborn; Aorta, Thoracic; Cell Culture Techniques; Cell Hypoxia; Disease Models, Animal; Humans; Hypertension, Renovascular; Hypoxia; Infant, Newborn; Laser Scanning Cytometry; Lim Kinases; Muscle Cells; Persistent Fetal Circulation Syndrome; Phosphorylation; Pulmonary Artery; Receptors, Thromboxane; Renal Artery; rhoA GTP-Binding Protein; Swine; Thromboxanes; Vasoconstriction; Vasoconstrictor Agents | 2012 |
Pulmonary hypertensive disorders and the arachidonic acid metabolites.
Topics: Adult; Animals; Arachidonic Acid; Arachidonic Acids; Child; Epoprostenol; Humans; Hypertension, Pulmonary; Infant, Newborn; Persistent Fetal Circulation Syndrome; Respiratory Distress Syndrome; SRS-A; Thromboxanes | 1988 |