threonine has been researched along with HIV Coinfection in 8 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (12.50) | 18.2507 |
2000's | 4 (50.00) | 29.6817 |
2010's | 1 (12.50) | 24.3611 |
2020's | 2 (25.00) | 2.80 |
Authors | Studies |
---|---|
Ali, A; Banerjea, AC; Das, A; Mishra, R; Raja, R; Wang, C | 1 |
Berlioz-Torrent, C; Lasserre, A; Margottin-Goguet, F; Matkovic, R; Morel, M; Vauthier, V; Versapuech, M; Zamborlini, A | 1 |
Fordyce, J; García, AA; Harrison, ML; Rock, J; Wolfe, AS; Zuñiga, JA | 1 |
Bregenzer, T; Hergersberg, M; Hohenstein, E; Huber, AR; Itin, P; Rady, PL; Streit, M; Tyring, SK | 1 |
Del Valle, L; Griffiths, KO; Husain, M; Kumar, D; Malhotra, A; Mathieson, PW; Meggs, LG; Mikulak, J; Pina-Oviedo, S; Reiss, K; Saleem, MA; Seshan, SV; Simoes, S; Singhal, PC; Vashistha, H; Wang, JY | 1 |
Albrecht, H; Bocchino, M; Cervasi, B; Costantini, A; Galati, D; Magnani, M; Montroni, M; Paiardini, M; Piedimonte, G; Silvestri, G | 1 |
Tuma, RS | 1 |
Back, NK; Berkhout, B; Boucher, CA; Keulen, W; van Wijk, A | 1 |
8 other study(ies) available for threonine and HIV Coinfection
Article | Year |
---|---|
Host AKT-mediated phosphorylation of HIV-1 accessory protein Vif potentiates infectivity via enhanced degradation of the restriction factor APOBEC3G.
Topics: APOBEC-3G Deaminase; HIV Infections; HIV-1; Humans; Phosphorylation; Protein Stability; Proto-Oncogene Proteins c-akt; Threonine; vif Gene Products, Human Immunodeficiency Virus | 2022 |
HUSH-mediated HIV silencing is independent of TASOR phosphorylation on threonine 819.
Topics: HIV Infections; HIV-1; Humans; Monomeric GTP-Binding Proteins; Nocodazole; Nuclear Proteins; Phosphorylation; SAM Domain and HD Domain-Containing Protein 1; Threonine; Viral Regulatory and Accessory Proteins; Virus Latency | 2022 |
The additive effect of type 2 diabetes on fibrinogen, von Willebrand factor, tryptophan and threonine in people living with HIV.
Topics: Biomarkers; Cross-Sectional Studies; Diabetes Mellitus, Type 2; Female; Fibrinogen; HIV; HIV Infections; Humans; Male; Middle Aged; Risk Factors; Threonine; Tryptophan; von Willebrand Factor | 2019 |
Epidermodysplasia verruciformis in a HIV-positive patient homozygous for the c917A-->T polymorphism in the TMC8/EVER2 gene.
Topics: Adult; AIDS-Related Opportunistic Infections; Alanine; Epidermodysplasia Verruciformis; Female; HIV Infections; Homozygote; Humans; Immunocompromised Host; Membrane Proteins; Mutation; Papillomaviridae; Polymorphism, Single Nucleotide; Threonine | 2009 |
Inhibition of p66ShcA longevity gene rescues podocytes from HIV-1-induced oxidative stress and apoptosis.
Topics: Active Transport, Cell Nucleus; Apoptosis; Carrier Proteins; Cell Adhesion Molecules, Neuronal; Cell Line, Transformed; Forkhead Box Protein O3; Forkhead Transcription Factors; Green Fluorescent Proteins; HIV Infections; HIV-1; Humans; Membrane Proteins; Oxidative Stress; Phenotype; Phosphorylation; Podocytes; Reactive Oxygen Species; RNA, Small Interfering; Shc Signaling Adaptor Proteins; Src Homology 2 Domain-Containing, Transforming Protein 1; Threonine; Transfection | 2009 |
Specific changes in the posttranslational regulation of nucleolin in lymphocytes from patients infected with human immunodeficiency virus.
Topics: Apoptosis; Blotting, Western; CDC2 Protein Kinase; Cell Cycle; Concanavalin A; Cyclin B; Cyclin B1; HIV Infections; HIV-1; Humans; Interleukin-2; Lymphocyte Activation; Microscopy, Confocal; Nucleolin; Phosphoproteins; Phosphorylation; Precipitin Tests; Protein Processing, Post-Translational; RNA-Binding Proteins; T-Lymphocyte Subsets; T-Lymphocytes; Threonine | 2003 |
With targeted drugs, chronic myelogenous leukemia therapy may follow HIV's model.
Topics: Antineoplastic Agents; Antiretroviral Therapy, Highly Active; Benzamides; Dasatinib; Drug Administration Schedule; Drug Costs; Fusion Proteins, bcr-abl; HIV Infections; Humans; Imatinib Mesylate; Isoleucine; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Mutation; Piperazines; Protein Kinase Inhibitors; Pyrimidines; Thiazoles; Threonine; United States | 2007 |
Initial appearance of the 184Ile variant in lamivudine-treated patients is caused by the mutational bias of human immunodeficiency virus type 1 reverse transcriptase.
Topics: Anti-HIV Agents; Genetic Variation; Genotype; HIV Infections; HIV Reverse Transcriptase; HIV-1; Humans; Lamivudine; Mutation; Reverse Transcriptase Inhibitors; Threonine | 1997 |