threonine has been researched along with Cardiomyopathy, Hypertrophic in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Ayati, M; Bermea, KC; Everett, AD; Foster, DB; Fu, Z; Gabrielson, K; Heravi, A; Kim, HB; Medina, A; Murphy, AM; Na, CH; Paolocci, N; Ramirez-Correa, GA; Xu, M; Yang, X; Zhang, X | 1 |
| Alvarez, J; Behlke, J; Doller, A; Erdmann, J; Haase, H; Hetzer, R; Morano, I; Petzhold, D; Regitz-Zagrosek, V; Vassort, G | 1 |
| Brink, P; Corfield, V; De Lange, W; Moolman-Smook, J | 1 |
3 other study(ies) available for threonine and Cardiomyopathy, Hypertrophic
| Article | Year |
|---|---|
Alteration in tyrosine phosphorylation of cardiac proteome and EGFR pathway contribute to hypertrophic cardiomyopathy.
Topics: Animals; Cardiomegaly; Cardiomyopathy, Hypertrophic; Mice; Phosphorylation; Proteome; Serine; Threonine; Tyrosine | 2022 |
Ahnak is critical for cardiac Ca(V)1.2 calcium channel function and its beta-adrenergic regulation.
Topics: Animals; Calcium; Calcium Channels, L-Type; Cardiomyopathy, Hypertrophic; Cyclic AMP-Dependent Protein Kinases; Dose-Response Relationship, Drug; Electrophysiology; Female; Gene Expression Regulation; Genetic Variation; Glutathione Transferase; Heterozygote; Humans; Isoleucine; Male; Membrane Proteins; Models, Biological; Models, Genetic; Mutation; Mutation, Missense; Myocardium; Myocytes, Cardiac; Neoplasm Proteins; Peptides; Phosphorylation; Polymorphism, Genetic; Polymorphism, Single-Stranded Conformational; Protein Binding; Protein Structure, Tertiary; Rats; Rats, Wistar; Receptors, Adrenergic, beta; Recombinant Proteins; Software; Temperature; Threonine; Ultracentrifugation; Up-Regulation | 2005 |
Expression of HCM causing mutations: lessons learnt from genotype-phenotype studies of the South African founder MYH7 A797T mutation.
Topics: Adolescent; Adult; Aged; Alanine; Amino Acid Substitution; Cardiomyopathy, Hypertrophic; Child; Death, Sudden, Cardiac; Female; Founder Effect; Gene Expression Regulation; Genotype; Humans; Male; Middle Aged; Mutation; Myosin Heavy Chains; Pedigree; Phenotype; Prognosis; Prospective Studies; South Africa; Threonine | 2000 |