threonic acid has been researched along with Age-Related Memory Disorders in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 2 (50.00) | 2.80 |
Authors | Studies |
---|---|
Chen, JL; Ding, HL; Li, WB; Lin, ZJ; Liu, BL; Liu, XG; Su, MZ; Wei, XH; Xie, JC; Yang, F; Zhan, HL; Zhou, X; Zhou, XF | 1 |
Cheng, Y; Guo, Y; Liu, C; Qian, H | 1 |
Jacobs, SA; Tsien, JZ; Wang, D | 1 |
Abumaria, N; Chui, D; Huang, X; Li, W; Liu, G; Liu, XG; Liu, Y; Ren, C; Xiong, W; Yu, J; Zhu, Y | 1 |
1 review(s) available for threonic acid and Age-Related Memory Disorders
Article | Year |
---|---|
Targeting the NMDA receptor subunit NR2B for treating or preventing age-related memory decline.
Topics: Aging; Animals; Brain; Butyrates; Cognition; Dietary Supplements; Humans; Magnesium; Memory; Memory Disorders; Molecular Targeted Therapy; Neuronal Plasticity; Receptors, N-Methyl-D-Aspartate; Up-Regulation | 2014 |
3 other study(ies) available for threonic acid and Age-Related Memory Disorders
Article | Year |
---|---|
Normalization of magnesium deficiency attenuated mechanical allodynia, depressive-like behaviors, and memory deficits associated with cyclophosphamide-induced cystitis by inhibiting TNF-α/NF-κB signaling in female rats.
Topics: Animals; Butyrates; Cyclophosphamide; Cystitis; Disease Models, Animal; Female; Hyperalgesia; Magnesium Deficiency; Memory Disorders; NF-kappa B; Rats; Rats, Sprague-Dawley; Signal Transduction; Tumor Necrosis Factor-alpha | 2020 |
Magnesium-L-threonate alleviate colonic inflammation and memory impairment in chronic-plus-binge alcohol feeding mice.
Topics: Animals; Antioxidants; Binge Drinking; Brain-Gut Axis; Butyrates; Chronic Disease; Colitis; Colon; Computational Biology; Cytokines; Dietary Supplements; Gastrointestinal Microbiome; Male; Maze Learning; Memory Disorders; Mice; Mice, Inbred C57BL | 2021 |
Elevation of brain magnesium prevents synaptic loss and reverses cognitive deficits in Alzheimer's disease mouse model.
Topics: Aging; Alzheimer Disease; Amyloid beta-Peptides; Amyloid Precursor Protein Secretases; Analysis of Variance; Animals; Aspartic Acid Endopeptidases; Body Weight; Brain; Butyrates; Cognition Disorders; Disease Models, Animal; Down-Regulation; Female; Hippocampus; Humans; Learning; Male; Memory Disorders; Mice, Transgenic; Motor Activity; Plaque, Amyloid; Receptors, N-Methyl-D-Aspartate; Signal Transduction; Synapses; Synaptic Transmission | 2014 |