thiazolidines has been researched along with Pancreatic Neoplasms in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 2 (66.67) | 2.80 |
Authors | Studies |
---|---|
Elumalai, S; Karunakaran, U; Moon, JS; Won, KC | 1 |
Choi, KY; Han, G; Lee, SH; Ryu, WJ | 1 |
Blum, R; Bozem, M; Feick, P; Kuhlmann, S; Schulz, I | 1 |
3 other study(ies) available for thiazolidines and Pancreatic Neoplasms
Article | Year |
---|---|
High glucose-induced PRDX3 acetylation contributes to glucotoxicity in pancreatic β-cells: Prevention by Teneligliptin.
Topics: Acetylation; Diabetes Mellitus, Type 2; Glucose; Humans; Insulin-Secreting Cells; Oxidative Stress; Pancreatic Neoplasms; Peroxiredoxin III; Pyrazoles; Src Homology 2 Domain-Containing, Transforming Protein 1; Thiazolidines; Ubiquitin Thiolesterase | 2020 |
Suppression of Wnt/β-catenin and RAS/ERK pathways provides a therapeutic strategy for gemcitabine-resistant pancreatic cancer.
Topics: beta Catenin; Cell Line, Tumor; Cell Proliferation; Cell Transformation, Neoplastic; Deoxycytidine; Gemcitabine; Humans; MAP Kinase Signaling System; Neoplasm Metastasis; Pancreatic Neoplasms; Proteolysis; Proto-Oncogene Proteins p21(ras); ras Proteins; Thiazolidines; Wnt Signaling Pathway | 2021 |
Hormone-stimulated calcium release is inhibited by cytoskeleton-disrupting toxins in AR4-2J cells.
Topics: Aluminum Compounds; Animals; Bacterial Proteins; Bacterial Toxins; Bombesin; Bridged Bicyclo Compounds, Heterocyclic; Calcium; Cell Membrane Permeability; Cytochalasin D; Cytoskeleton; Cytosol; Fluorides; GTP-Binding Proteins; Hormones; Inositol 1,4,5-Trisphosphate; Pancreas; Pancreatic Neoplasms; Rats; Thiazoles; Thiazolidines; Tumor Cells, Cultured; Type C Phospholipases | 2000 |